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51. The SHDRA syndrome associated gene TMEM260 encodes a protein-specific O-mannosyltransferase

52. Missense variants in the voltage sensing and pore domain of KCNH5 cause neurodevelopmental phenotypes including epilepsy

53. P265 Current UK wide use of the BASMI outcome measure in axial spondyloarthritis: an AStretch survey

54. eP081: Dandy Walker malformation in three unrelated families with biallelic variants in CAPN15 expands the phenotypic spectrum of oculogastrointestinal neurodevelopmental disorder

55. A recurrent, de novo pathogenic variant in ARPC4 disrupts actin filament formation and causes microcephaly and speech delay

56. Variant-specific effects define the phenotypic spectrum of HNRNPH2-associated neurodevelopmental disorders in males

57. High Incidence of Noonan Syndrome Features Including Short Stature and Pulmonic Stenosis in Patients carrying NF1 Missense Mutations Affecting p.Arg1809: Genotype–Phenotype Correlation

58. Heterozygous variants in KCNC2 cause a broad spectrum of epilepsy phenotypes associated with characteristic functional alterations 2021.05.21.21257099

59. Correction to:De novo variants in SNAP25 cause an early-onset developmental and epileptic encephalopathy (Genetics in Medicine, (2021), 23, 4, (653-660), 10.1038/s41436-020-01020-w)

60. Expanding the Clinical Spectrum of Mitochondrial Citrate Carrier (SLC25A1) Deficiency: Facial Dysmorphism in Siblings with Epileptic Encephalopathy and Combined D,L-2-Hydroxyglutaric Aciduria

62. Heterozygous variants in KCNC2 cause a broad spectrum of epilepsy phenotypes associated with characteristic functional alterations 2021.05.21.21257099

63. Clinical and mutational spectrum of neurofibromatosis type 1-like syndrome

64. Heterozygous variants in KCNC2 cause a broad spectrum of epilepsy phenotypes associated with characteristic functional alterations

65. Correction to: De novo variants in SNAP25 cause an early-onset developmental and epileptic encephalopathy

66. Opportunities for fellowship education: the first year of the Medical Biochemical Genetics Clinical Core Seminar Series

67. De novo variants in SNAP25 cause an early-onset developmental and epileptic encephalopathy

69. Molecular and clinical analyses of Greig Cephalopolysyndactyly and Pallister-Hall syndromes: robust phenotype prediction from the type and position of GLI3 mutations

70. Novel FOXF1 Mutations in Sporadic and Familial Cases of Alveolar Capillary Dysplasia with Misaligned Pulmonary Veins Imply a Role for its DNA Binding Domain

72. Microduplication 22q11.2, an emerging syndrome: clinical, cytogenetic, and molecular analysis of thirteen patients

73. Subtelomeric deletion of chromosome 10p15.3: Clinical findings and molecular cytogenetic characterization

75. Further clinical and molecular delineation of the 15q24 microdeletion syndrome

76. Haploinsufficiency of the Notch Ligand DLL1 Causes Variable Neurodevelopmental Disorders

77. NCKAP1 Disruptive Variants Lead to a Neurodevelopmental Disorder with Core Features of Autism

80. Phenotypic spectrum and transcriptomic profile associated with germline variants in TRAF7

82. De Novo SOX6 Variants Cause a Neurodevelopmental Syndrome Associated with ADHD, Craniosynostosis, and Osteochondromas

86. Mutations in NCAPG2 Cause a Severe Neurodevelopmental Syndrome that Expands the Phenotypic Spectrum of Condensinopathies

88. CSGALNACT1‐congenital disorder of glycosylation: A mild skeletal dysplasia with advanced bone age

89. De Novo Pathogenic Variants in N-cadherin Cause a Syndromic Neurodevelopmental Disorder with Corpus Collosum, Axon, Cardiac, Ocular, and Genital Defects

96. Spectrum of Phenotypic, Genetic, and Functional Characteristics in Patients With Epilepsy With KCNC2Pathogenic Variants

98. Further evidence for the involvement of EFL1 in a Shwachman-Diamond-like syndrome and expansion of the phenotypic features

99. A Genocentric Approach to Discovery of Mendelian Disorders

100. De Novo Pathogenic Variants in N-cadherin Cause a Syndromic Neurodevelopmental Disorder with Corpus Callosum, Axon, Cardiac, Ocular, and Genital Defects

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