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53 results on '"Hyang Yeon, Kim"'

51. Laboratory and field evaluations of entomopathogenic Lecanicillium attenuatum CNU-23 for control of green peach aphid (Myzus persicae)

Author

Hyang Yeon, Kim, Hyang Burm, Lee, Young Cheol, Kim, and In Seon, Kim

Subjects
Aphids, Hypocreales, RNA, Ribosomal, 18S, Animals, DNA, Intergenic, Capsicum, Pest Control, Biological, Phylogeny
Abstract

An entomopathogenic fungus was isolated from an infected aphid. The isolate conformed most closely to Lecanicillium attenuatum CBS 402.78 (AJ292434) based on the internal transcribed spacer (ITS) region of its 18S rDNA, and thus was designated L. attenuatum CNU-23. Laboratory and field evaluations of CNU-23 blastospores were carried out for the control of green peach aphids. The laboratory evaluations of CNU-23 revealed an aphid mortality of about 80% with an estimated LT50 of 3.72 days after the application of CNU-23 at 1x106 blastospores/ ml. Meanwhile, the field evaluations of CNU-23 performed on greenhouse pepper plants during the rainy season showed an aphid mortality ranging from 72% to 97%. Significant sporulation was observed in the aphids treated with CNU-23. Therefore, the results suggest that L. attenuatum CNU-23 can be used as a biocontrol agent for green peach aphids on greenhouse pepper plants.

Published
2009

52. Lysophospholipid profile in serum and liver by high-fat diet and tumor induction in obesity-resistant BALB/c mice.

Author

Hyang Yeon Kim, Minhee Kim, Hye Min Park, Jiyoung Kim, Eun Ji Kim, Choong Hwan Lee, and Jung Han Yoon Park

Subjects
*OBESITY, *COLON tumors, *PHOSPHOLIPID analysis, *ANIMAL experimentation, *ARTIFICIAL feeding, *BIOMARKERS, *BODY weight, *DIET, *FAT content of food, *LIVER, *EVALUATION of medical care, *MICE, *NUTRITION, *PHOSPHOLIPIDS, *TUMORS, *DIAGNOSIS, *CANCER risk factors
Abstract

Objective: Our previous study revealed that chronic consumption of a high-fat diet (HFD) stimulates colon cancer progression in obesity-resistant BALB/c mice. The aim of the present study was to investigate the significant alteration of metabolites caused by tumor progression and an HFD in the serum and liver in the same mouse model. Methods: Male BALB/c mice were fed either a control diet or a HFD for 20.5 wk. The syngeneic CT26 colon carcinoma cells were injected into the right rear flank of mice after 16 wk of feeding. Metabolites in serum and liver samples were analyzed by ultra-performance liquid chromatography-quadrupole-time-of-flight-mass spectrometry-based metabolomics. Results: HFD feeding and tumor injection induced changes in the choline-containing phospho-lipids, namely, phosphatidylcholines and lysophosphatidylcholines (lysoPCs), and lysophosphati-dylethanolamines in the serum and liver. The majority of these metabolite changes were due to HFD feeding (11 in sera and 5 in livers) rather than tumors (3 in sera and 1 in livers). Conclusion: The HFD- and tumor-related metabolite alterations of phospholipids, especially lysoPCs, in the liver and serum of obesity-resistant mice, suggesting that the lysoPCs are potential biomarkers for the chronic consumption of HFD in nonobese individuals. [ABSTRACT FROM AUTHOR]

Published
2014
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