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316 results on '"Huang, Yurong"'

51. Data from Pten Regulates Aurora-A and Cooperates with Fbxw7 in Modulating Radiation-Induced Tumor Development

Author

Kwon, Yong-Won, primary, Kim, Il-Jin, primary, Wu, Di, primary, Lu, Jing, primary, Stock, William A., primary, Liu, Yueyong, primary, Huang, Yurong, primary, Kang, Hio Chung, primary, DelRosario, Reyno, primary, Jen, Kuang-Yu, primary, Perez-Losada, Jesus, primary, Wei, Guangwei, primary, Balmain, Allan, primary, and Mao, Jian-Hua, primary

Published
2023
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52. Data from CUL4A Induces Epithelial–Mesenchymal Transition and Promotes Cancer Metastasis by Regulating ZEB1 Expression

Author

Wang, Yunshan, primary, Wen, Mingxin, primary, Kwon, Yongwon, primary, Xu, Yangyang, primary, Liu, Yueyong, primary, Zhang, Pengju, primary, He, Xiuquan, primary, Wang, Qin, primary, Huang, Yurong, primary, Jen, Kuang-Yu, primary, LaBarge, Mark A., primary, You, Liang, primary, Kogan, Scott C., primary, Gray, Joe W., primary, Mao, Jian-Hua, primary, and Wei, Guangwei, primary

Published
2023
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53. Supplementary Figure 1 from CUL4A Induces Epithelial–Mesenchymal Transition and Promotes Cancer Metastasis by Regulating ZEB1 Expression

Author

Wang, Yunshan, primary, Wen, Mingxin, primary, Kwon, Yongwon, primary, Xu, Yangyang, primary, Liu, Yueyong, primary, Zhang, Pengju, primary, He, Xiuquan, primary, Wang, Qin, primary, Huang, Yurong, primary, Jen, Kuang-Yu, primary, LaBarge, Mark A., primary, You, Liang, primary, Kogan, Scott C., primary, Gray, Joe W., primary, Mao, Jian-Hua, primary, and Wei, Guangwei, primary

Published
2023
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54. Supplementary Figure 5 from CUL4A Induces Epithelial–Mesenchymal Transition and Promotes Cancer Metastasis by Regulating ZEB1 Expression

Author

Wang, Yunshan, primary, Wen, Mingxin, primary, Kwon, Yongwon, primary, Xu, Yangyang, primary, Liu, Yueyong, primary, Zhang, Pengju, primary, He, Xiuquan, primary, Wang, Qin, primary, Huang, Yurong, primary, Jen, Kuang-Yu, primary, LaBarge, Mark A., primary, You, Liang, primary, Kogan, Scott C., primary, Gray, Joe W., primary, Mao, Jian-Hua, primary, and Wei, Guangwei, primary

Published
2023
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55. Supplementary Figure 2 from CUL4A Induces Epithelial–Mesenchymal Transition and Promotes Cancer Metastasis by Regulating ZEB1 Expression

Author

Wang, Yunshan, primary, Wen, Mingxin, primary, Kwon, Yongwon, primary, Xu, Yangyang, primary, Liu, Yueyong, primary, Zhang, Pengju, primary, He, Xiuquan, primary, Wang, Qin, primary, Huang, Yurong, primary, Jen, Kuang-Yu, primary, LaBarge, Mark A., primary, You, Liang, primary, Kogan, Scott C., primary, Gray, Joe W., primary, Mao, Jian-Hua, primary, and Wei, Guangwei, primary

Published
2023
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56. Supplementary Methods, Figure Legend, Table 1 from CUL4A Induces Epithelial–Mesenchymal Transition and Promotes Cancer Metastasis by Regulating ZEB1 Expression

Author

Wang, Yunshan, primary, Wen, Mingxin, primary, Kwon, Yongwon, primary, Xu, Yangyang, primary, Liu, Yueyong, primary, Zhang, Pengju, primary, He, Xiuquan, primary, Wang, Qin, primary, Huang, Yurong, primary, Jen, Kuang-Yu, primary, LaBarge, Mark A., primary, You, Liang, primary, Kogan, Scott C., primary, Gray, Joe W., primary, Mao, Jian-Hua, primary, and Wei, Guangwei, primary

Published
2023
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57. Supplementary Figure 6 from CUL4A Induces Epithelial–Mesenchymal Transition and Promotes Cancer Metastasis by Regulating ZEB1 Expression

Author

Wang, Yunshan, primary, Wen, Mingxin, primary, Kwon, Yongwon, primary, Xu, Yangyang, primary, Liu, Yueyong, primary, Zhang, Pengju, primary, He, Xiuquan, primary, Wang, Qin, primary, Huang, Yurong, primary, Jen, Kuang-Yu, primary, LaBarge, Mark A., primary, You, Liang, primary, Kogan, Scott C., primary, Gray, Joe W., primary, Mao, Jian-Hua, primary, and Wei, Guangwei, primary

Published
2023
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58. Supplementary Figure 4 from CUL4A Induces Epithelial–Mesenchymal Transition and Promotes Cancer Metastasis by Regulating ZEB1 Expression

Author

Wang, Yunshan, primary, Wen, Mingxin, primary, Kwon, Yongwon, primary, Xu, Yangyang, primary, Liu, Yueyong, primary, Zhang, Pengju, primary, He, Xiuquan, primary, Wang, Qin, primary, Huang, Yurong, primary, Jen, Kuang-Yu, primary, LaBarge, Mark A., primary, You, Liang, primary, Kogan, Scott C., primary, Gray, Joe W., primary, Mao, Jian-Hua, primary, and Wei, Guangwei, primary

Published
2023
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59. Supplementary Figure 3 from CUL4A Induces Epithelial–Mesenchymal Transition and Promotes Cancer Metastasis by Regulating ZEB1 Expression

Author

Wang, Yunshan, primary, Wen, Mingxin, primary, Kwon, Yongwon, primary, Xu, Yangyang, primary, Liu, Yueyong, primary, Zhang, Pengju, primary, He, Xiuquan, primary, Wang, Qin, primary, Huang, Yurong, primary, Jen, Kuang-Yu, primary, LaBarge, Mark A., primary, You, Liang, primary, Kogan, Scott C., primary, Gray, Joe W., primary, Mao, Jian-Hua, primary, and Wei, Guangwei, primary

Published
2023
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67. Studies on a Class of AWG-Based Node Architectures for Optical Burst-Switched Networks

Author

Huang, Yurong (Grace), Datta, Debasish, Qiu, Xue, Zhang, Jing, Park, Hyuk-Kyu, Kim, Young-Chon, Heritage, Jonathan P., Mukherjee, Biswanath, Kanade, Takeo, editor, Kittler, Josef, editor, Kleinberg, Jon M., editor, Mattern, Friedemann, editor, Mitchell, John C., editor, Naor, Moni, editor, Nierstrasz, Oscar, editor, Pandu Rangan, C., editor, Steffen, Bernhard, editor, Sudan, Madhu, editor, Terzopoulos, Demetri, editor, Tygar, Dough, editor, Vardi, Moshe Y., editor, Weikum, Gerhard, editor, Bubak, Marian, editor, van Albada, Geert Dick, editor, Sloot, Peter M. A., editor, and Dongarra, Jack, editor

Published
2004
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68. Explicit Routing for Traffic Engineering in Labeled Optical Burst-Switched WDM Networks

Author

Zhang, Jing, Lee, Hae-Joung, Wang, Shuang, Qiu, Xue, Zhu, Keyao, Huang, Yurong, Datta, Debasish, Kim, Young-Chon, Mukherjee, Biswanath, Kanade, Takeo, editor, Kittler, Josef, editor, Kleinberg, Jon M., editor, Mattern, Friedemann, editor, Mitchell, John C., editor, Naor, Moni, editor, Nierstrasz, Oscar, editor, Pandu Rangan, C., editor, Steffen, Bernhard, editor, Sudan, Madhu, editor, Terzopoulos, Demetri, editor, Tygar, Dough, editor, Vardi, Moshe Y., editor, Weikum, Gerhard, editor, Bubak, Marian, editor, van Albada, Geert Dick, editor, Sloot, Peter M. A., editor, and Dongarra, Jack, editor

Published
2004
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73. A cultural turn in urban governance: cultural practices of governance in the Guangdong-Hong Kong-Macau Greater Bay Area.

Author

Luo, Xianmei and Huang, Yurong

Subjects
*URBAN planning, *SUSTAINABLE development, *MUNICIPAL government, *CULTURAL industries
Abstract

Tracing the concept and phenomena of 'cultural governance', as it evolves along the agenda of China's 'modernization of governing capacity (of the state)', this article tries to capture the various term-shaping processes against the backdrop of a political discourse that gave birth to them. The article discusses topics such as urban planning, infrastructure upgrading, community reformation, and the so-called cultural-sustainable development strategy adopted by the Chinese authorities with selected cases from three mainland Chinese urban centers (Shenzhen, Foshan and Dongguan) of the Guangdong-Hong Kong-Macau Greater Bay Area. Pointing to both the flexibility and adaptability of the state's involvement, the research elaborates the models adopted by and problems associated with various pioneering programs initiated by municipalities as they carry out what might be called, arguably, a 'cultural turn' of urban governance. [ABSTRACT FROM AUTHOR]

Published
2023
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74. Circular RNA in autoimmune diseases: special emphasis on regulation mechanism in RA and SLE.

Author

Huang, Yurong, Xue, Qiuyun, Cheng, Chenglong, Wang, Yuting, Wang, Xiao, Chang, Jun, and Miao, Chenggui

Subjects
*CIRCULAR RNA, *AUTOIMMUNE diseases, *SYSTEMIC lupus erythematosus, *RHEUMATOID arthritis, *NON-coding RNA, *AUTOANTIBODIES
Abstract

Background: Autoimmune diseases are diseases caused by tissue damage caused by the body's immune response to autoantibodies. Circular RNAs (CircRNAs) are a kind of special endogenous non-coding RNA that play a biological role by regulating gene transcription. Methods: In this work, we searched the PubMed, Web of Science (SCIE), National Science and Technology Library (NSTL), and ScienceDirect Online (SDOL) databases to summarize the impact of circRNAs on autoimmune diseases, especially the results of circRNAs in rheumatoid arthritis (RA) and systemic lupus erythematosus (SLE). Results: The study on the function of circRNAs and autoimmune diseases further deepened our understanding of the development and pathogenesis of autoimmune diseases. CircRNAs may act as miRNA sponges to regulate biological processes and affect the occurrence and development of autoimmune diseases. CircRNAs are closely related to the pathogenesis of RA and SLE and may become potential biomarkers for the diagnosis and treatment of RA and SLE. Conclusion: CircRNAs play an important role in the pathogenesis of RA, SLE and other autoimmune diseases, and are expected to provide new biomarkers for the diagnosis and treatment of autoimmune diseases. However, the function and mechanism of circRNAs in autoimmune diseases need more comprehensive research. [ABSTRACT FROM AUTHOR]

Published
2023
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75. CircRnas in atherosclerosis, with special emphasis on the spongy effect of circRnas on miRnas.

Author

Cheng, Chenglong, Wang, Yuting, Xue, Qiuyun, Huang, Yurong, Wang, Xiao, Liao, Faxue, and Miao, Chenggui

Subjects
CIRCULAR RNA, MICRORNA, VASCULAR smooth muscle, NON-coding RNA, MUSCLE cells, ATHEROSCLEROSIS
Abstract

Atherosclerosis (AS) is a chronic inflammatory disease, which leads to atherosclerotic rupture, lumen stenosis and thrombosis, and often endangers life. Circular RNAs (circRNAs) are a special class of non-coding RNA molecules, whose abnormal expression has been proved to be closely related to human diseases, including AS. Both the abnormal regulation of circRNAs and the sponging effect on miRNAs would lead to changes in gene expression in the form of epigenetic modification, ultimately leading to the formation of AS. CircRNAs can be used as peripheral blood markers of AS, and play an important regulatory role in the proliferation, migration, inflammation and apoptosis of vascular smooth muscle cells, endothelial cells and macrophage, which are key cells for the development of AS. The in-depth understanding of circRNAs in AS not only provides a new method for the diagnosis of AS, but also provides a new idea for the treatment of AS. [ABSTRACT FROM AUTHOR]

Published
2023
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86. GSTP1 Ile105Val polymorphism might be associated with the risk of radiation pneumonitis among lung cancer patients in Chinese population: A prospective study

Author

Zhang Sujing, Niu Baolong, Xiang Huang, Liu Xiaoliang, Fang Tong, Gaoxiang Chen, Fang Liu, Nana Zhao, Huang Yurong, Baolin Qu, Lehui Du, Xiangkun Dai, Xiaohu Cong, S. Xu, Wei Yu, and Chuanbin Xie

Subjects
0301 basic medicine, Oncology, medicine.medical_specialty, XRCC1, Single-nucleotide polymorphism, Lower risk, 03 medical and health sciences, 0302 clinical medicine, Internal medicine, Statistical significance, Genotype, medicine, Prospective cohort study, Lung cancer, GSTP1, Genetic polymorphism, business.industry, Hazard ratio, Common Terminology Criteria for Adverse Events, medicine.disease, 030104 developmental biology, 030220 oncology & carcinogenesis, Radiation pneumonitis, business, Research Paper
Abstract

Background: Growing data suggest that DNA damage repair and detoxification pathways play crucial roles in radiation-induced toxicities. To determine whether common functional single-nucleotide polymorphisms (SNPs) in candidate genes from these pathways can be used as predictors of radiation pneumonitis (RP), we conducted a prospective study to evaluate the associations between functional SNPs and risk of RP. Methods: We recruited a total of 149 lung cancer patients who had received intensity modulated radiation therapy (IMRT). GSTP1 and XRCC1 were genotyped using the SurPlexTM-xTAG method in all patients. RP events were prospectively scored using the National Cancer Institute Common Terminology Criteria for Adverse Events (CTCAE), version 4.0. Kaplan-Meier analysis was used to determine the cumulative probability of RP of grade ≥ 2. Cox proportional hazard regression was performed to identify clinical variables and SNPs associated with risk of RP grade ≥ 2, using univariate and multivariate analysis, respectively. Results: With a median follow-up of 9 months, the incidence of RP of grade ≥ 2 was 38.3%. A predicting role in RP was observed for the GSTP1 SNP (adjusted hazard ratio 3.543; 95% CI 1.770-7.092; adjusted P< 0.001 for the Ile/Val and Val/Val genotypes versus Ile/Ile genotype). Whereas, we found that patients with XRCC1 399Arg/Gln and Gln/Gln genotypes had a lower risk of RP compares with those carrying Arg/Arg genotype (adjusted HR 0.653; 95% CI 0.342-1.245), but with no statistical significance observed (adjusted P = 0.195). Conclusions: Our results suggested a novel association between GSTP1 SNP 105Ile/Val and risk of RP development, which suggests the potential use of this genetic polymorphism as a predictor of RP. In addition, genetic polymorphisms of XRCC1 399Arg/Gln may also be associated with RP.

Published
2018

92. Electric Vehicle BMS Subdivision Technology Patent Distribution Analysis

Author

Hou Yuanyuan, Zhou Jingyan, Huang Yurong, and Qiao Jing

Subjects
ComputingMilieux_GENERAL, business.product_category, Distribution (number theory), business.industry, Electric vehicle, business, Topology, Geology, Subdivision
Abstract

Patents is one of the most important achievements of technology innovation. This article analyze the technology research and development of subdivision technology of the electric vehicles battery management system from the perspective of patent, analyze the research progress of every subdivision technology, the main innovation country, and the main body of innovation, in order to provide intelligence support for the enterprise technology research and development and looking for partners.

Published
2019

94. Association of DNA repair gene polymorphisms with the risk of radiation pneumonitis in lung cancer patients

Author

Yanan Han, Liu Xiaoliang, Fang Tong, Xiaohu Cong, Xiangkun Dai, Lanqing Liang, Fang Liu, Huang Yurong, Nana Zhao, Lehui Du, Ju Zhongjian, Wei Yang, Xiang Huang, Chuanbin Xie, Baolin Qu, and Wei Yu

Subjects
0301 basic medicine, Oncology, medicine.medical_specialty, XRCC1, DNA repair, 03 medical and health sciences, 0302 clinical medicine, Internal medicine, Genotype, medicine, SNP, Lung cancer, Radiation Pneumonitis, business.industry, medicine.disease, lung cancer, 030104 developmental biology, 030220 oncology & carcinogenesis, ERCC2, ERCC1, radiation pneumonitis, business, Research Paper
Abstract

A total of 149 lung cancer patients were recruited to receive intensity modulated radiation therapy (IMRT). The association of developing radiation pneumonitis (RP) with genetic polymorphism was evaluated. The risks of four polymorphic sites in three DNA repair related genes (ERCC1, rs116615:T354C and rs3212986:C1516A; ERCC2, rs13181:A2251C; XRCC1, rs25487:A1196G) for developing grade ≥ 2 RP were assessed respectively. It was observed that ERCC1 T354C SNP had a significant effect on the development of grade ≥ 2 RP (CT/TT vs. CC, adjusted HR = 0.517, 95% CI, 0.285-0.939; adjusted P = 0.030). It is the first time demonstrating that CT/TT genotype of ERCC1 354 was significantly associated with lower RP risk after radio therapy.

Published
2017

98. GSTP1 Ile105Val polymorphism might be associated with the risk of radiation pneumonitis among lung cancer patients in Chinese population: A prospective study

Author

Du, Lehui, primary, Yu, Wei, additional, Huang, Xiang, additional, Zhao, Nana, additional, Liu, Fang, additional, tong, Fang, additional, Zhang, Sujing, additional, Niu, Baolong, additional, Liu, Xiaoliang, additional, Xu, Shouping, additional, Huang, Yurong, additional, Dai, Xiangkun, additional, Xie, Chuanbin, additional, Chen, Gaoxiang, additional, Cong, Xiaohu, additional, and Qu, Baolin, additional

Published
2018
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