1,212 results on '"Hilsenbeck, Susan G."'
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52. Self-Sampling for Human Papillomavirus Testing: Acceptability in a U.S. Safety Net Health System
53. Combinatorial inhibition of PTPN12-regulated receptors leads to a broadly effective therapeutic strategy in triple-negative breast cancer
54. Diverse Signaling Pathways Modulate Nuclear Receptor Recruitment of N-CoR and SMRT Complexes
55. A Multiparameter Molecular Classifier to Predict Response to Neoadjuvant Lapatinib plus Trastuzumab without Chemotherapy in HER2+ Breast Cancer
56. Supplementary Figure S1 from Targeting the Pro-survival Protein BCL-2 to Prevent Breast Cancer
57. Supplementary Figure S3 from Intratumoral Heterogeneity in a Trp53-Null Mouse Model of Human Breast Cancer
58. Data from Intratumoral Heterogeneity in a Trp53-Null Mouse Model of Human Breast Cancer
59. Supplementary table 3 from Blockade of AP-1 Potentiates Endocrine Therapy and Overcomes Resistance
60. Data from Blockade of AP-1 Potentiates Endocrine Therapy and Overcomes Resistance
61. Related Article from Prevention of Tumorigenesis in p53-Null Mammary Epithelium by Rexinoid Bexarotene, Tyrosine Kinase Inhibitor Gefitinib, and Celecoxib
62. Supplementary Table S2 from Intratumoral Heterogeneity in a Trp53-Null Mouse Model of Human Breast Cancer
63. Supplementary Figure 1 from Blockade of AP-1 Potentiates Endocrine Therapy and Overcomes Resistance
64. Supplementary Figure 2 from Blockade of AP-1 Potentiates Endocrine Therapy and Overcomes Resistance
65. Supplementary table 4 from Blockade of AP-1 Potentiates Endocrine Therapy and Overcomes Resistance
66. Supplementary table 2 from Blockade of AP-1 Potentiates Endocrine Therapy and Overcomes Resistance
67. Supplementary table 1 from Blockade of AP-1 Potentiates Endocrine Therapy and Overcomes Resistance
68. Data from Targeting the Pro-survival Protein BCL-2 to Prevent Breast Cancer
69. Supplementary Methods, Figure Legends, Tables S1, S3 - S6 from Intratumoral Heterogeneity in a Trp53-Null Mouse Model of Human Breast Cancer
70. Legend for supplementay figures from Blockade of AP-1 Potentiates Endocrine Therapy and Overcomes Resistance
71. Stereotactic Biopsy of Segmental Breast Calcifications: Is Sampling of Anterior and Posterior Components Necessary?
72. FOXA1 overexpression mediates endocrine resistance by altering the ER transcriptome and IL-8 expression in ER-positive breast cancer
73. The Signaling Pathways Project, an integrated ‘omics knowledgebase for mammalian cellular signaling pathways
74. Supplementary Table 1 from TBCRC023: A Randomized Phase II Neoadjuvant Trial of Lapatinib Plus Trastuzumab Without Chemotherapy for 12 versus 24 Weeks in Patients with HER2-Positive Breast Cancer
75. Data from Mammary Precancerous Stem and Non-Stem Cells Evolve into Cancers of Distinct Subtypes
76. Supplementary Tables from HER2 Reactivation through Acquisition of the HER2 L755S Mutation as a Mechanism of Acquired Resistance to HER2-targeted Therapy in HER2+ Breast Cancer
77. Supplementary information from HER2 Reactivation through Acquisition of the HER2 L755S Mutation as a Mechanism of Acquired Resistance to HER2-targeted Therapy in HER2+ Breast Cancer
78. Data from TBCRC023: A Randomized Phase II Neoadjuvant Trial of Lapatinib Plus Trastuzumab Without Chemotherapy for 12 versus 24 Weeks in Patients with HER2-Positive Breast Cancer
79. Supplementary Figure 2 from Upregulation of ER Signaling as an Adaptive Mechanism of Cell Survival in HER2-Positive Breast Tumors Treated with Anti-HER2 Therapy
80. Supplementary Data from Evaluation of the Predictive Role of Tumor Immune Infiltrate in Patients with HER2-Positive Breast Cancer Treated with Neoadjuvant Anti-HER2 Therapy without Chemotherapy
81. Supplemental figures 7-8 from Tenascin-C and Integrin α9 Mediate Interactions of Prostate Cancer with the Bone Microenvironment
82. Supplemental figures 3-6 from Tenascin-C and Integrin α9 Mediate Interactions of Prostate Cancer with the Bone Microenvironment
83. Supplementary Figures from Activation of the IFN Signaling Pathway is Associated with Resistance to CDK4/6 Inhibitors and Immune Checkpoint Activation in ER-Positive Breast Cancer
84. Supplementary Figure 1 from Upregulation of ER Signaling as an Adaptive Mechanism of Cell Survival in HER2-Positive Breast Tumors Treated with Anti-HER2 Therapy
85. Supplemental tables 1-3. IHC reagents and conditions from Tenascin-C and Integrin α9 Mediate Interactions of Prostate Cancer with the Bone Microenvironment
86. Supplemental tables 4-5 from Tenascin-C and Integrin α9 Mediate Interactions of Prostate Cancer with the Bone Microenvironment
87. Supplementary Table 2 from TBCRC023: A Randomized Phase II Neoadjuvant Trial of Lapatinib Plus Trastuzumab Without Chemotherapy for 12 versus 24 Weeks in Patients with HER2-Positive Breast Cancer
88. Supplemental Experimental procedures from Tenascin-C and Integrin α9 Mediate Interactions of Prostate Cancer with the Bone Microenvironment
89. Figure S8 from Mammary Precancerous Stem and Non-Stem Cells Evolve into Cancers of Distinct Subtypes
90. Data from Comprehensive Genomic Analysis Identifies Novel Subtypes and Targets of Triple-Negative Breast Cancer
91. Data from Activation of the IFN Signaling Pathway is Associated with Resistance to CDK4/6 Inhibitors and Immune Checkpoint Activation in ER-Positive Breast Cancer
92. Data from Tenascin-C and Integrin α9 Mediate Interactions of Prostate Cancer with the Bone Microenvironment
93. Supplemental figures 9-11 from Tenascin-C and Integrin α9 Mediate Interactions of Prostate Cancer with the Bone Microenvironment
94. Supplementary Table Set S11-S18 from Comprehensive Genomic Analysis Identifies Novel Subtypes and Targets of Triple-Negative Breast Cancer
95. Supplementary Data from Progesterone Receptor Isoform-Specific Promoter Methylation: Association of PRA Promoter Methylation with Worse Outcome in Breast Cancer Patients
96. Supplemental Figures 1-2 from Tenascin-C and Integrin α9 Mediate Interactions of Prostate Cancer with the Bone Microenvironment
97. Supplementary Figures S1-S4 from High IGF-IR Activity in Triple-Negative Breast Cancer Cell Lines and Tumorgrafts Correlates with Sensitivity to Anti–IGF-IR Therapy
98. Auxiliary Supplementary File from Therapeutic Targeting of Nemo-like Kinase in Primary and Acquired Endocrine-resistant Breast Cancer
99. Data from A Wnt-Independent LGR4–EGFR Signaling Axis in Cancer Metastasis
100. Data from High IGF-IR Activity in Triple-Negative Breast Cancer Cell Lines and Tumorgrafts Correlates with Sensitivity to Anti–IGF-IR Therapy
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