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51. The coarse‐grained plaque is an Aβ plaque‐type in APOE Ɛ4+ Alzheimer’s disease that is associated with astrogliosis and p‐tau burden

53. Association of DNA methylation from the temporal cortex and cerebellum with AD‐related neuropathology and biochemistry

54. Two rare AKAP9 variants are associated with Alzheimer's disease in African Americans

55. APOE ε4 influences medial temporal atrophy and tau deposition in atypical Alzheimer's disease

56. Genome-wide association study of brain biochemical phenotypes reveals distinct genetic architecture of Alzheimer's Disease related proteins

57. APOE ε4 influences medial temporal atrophy and tau deposition in atypical Alzheimer's disease.

58. Hemispheric Cortical Atrophy ipsilateral to Carotid Stenosis (N5.001)

59. Investigating Heterogeneity and Neuroanatomic Correlates of Longitudinal Clinical Decline in Atypical Alzheimer Disease

61. Single Nuclei Transcriptome Reveals Perturbed Brain Vascular Molecules in Alzheimer's Disease

62. An examination of atypical primary progressive aphasia variants

63. Cancer survivors have a lower frequency of dementia in the 95+ oldest‐old

65. Plasma transcript profiling identifies significant differentially expressed genes and expression of quantitative trait loci in African Americans

66. Deletion of Abi3/Gngt2 influences age-progressive amyloid β and tau pathologies in distinctive ways

67. Assessment of the genetic variance of late-onset Alzheimer's disease

68. Transcriptional landscape of human microglia reveals robust gene expression signatures that implicates age, sex and APOE-related immunometabolic pathway perturbations

70. A nonsynonymous mutation in PLCG2 reduces the risk of Alzheimer's disease, dementia with Lewy bodies and frontotemporal dementia, and increases the likelihood of longevity (vol 138, pg 237, 2019)

71. Utility of Plasma Neurofilament Light in the 1Florida Alzheimer’s Disease Research Center (ADRC)

72. Leveraging selective hippocampal vulnerability among Alzheimer’s disease subtypes reveals a novel tau binding partner SERPINA5

73. Investigating gene expression changes underlying selective hippocampal vulnerability in Alzheimer’s disease using a translational neuropathology approach

74. A consensus proteomic analysis of Alzheimer’s disease brain and cerebrospinal fluid reveals early changes in energy metabolism associated with microglia and astrocyte activation

76. Variants in the ATP-Binding Cassette Transporter (ABCA7), Apolipoprotein E ε4,and the Risk of Late-Onset Alzheimer Disease in African Americans

77. Atlas of Transcription Factor Binding Sites from ENCODE DNase Hypersensitivity Data across 27 Tissue Types

78. MRI and flortaucipir relationships in Alzheimer's phenotypes are heterogeneous

81. Longitudinal Amyloid-β PET in Atypical Alzheimer’s Disease and Frontotemporal Lobar Degeneration

82. Selective Vulnerability of the Nucleus Basalis of Meynert Among Neuropathologic Subtypes of Alzheimer Disease

83. Meta-analysis Confirms CR1, CLU, and PICALM as Alzheimer Disease Risk Loci and Reveals Interactions With APOE Genotypes

84. Individual bioenergetic capacity as a potential source of resilience to Alzheimer's disease.

86. A nonsynonymous mutation in PLCG2 reduces the risk of Alzheimer's disease, dementia with Lewy bodies and frontotemporal dementia, and increases the likelihood of longevity

88. A Consensus Proteomic Analysis of Alzheimer’s Disease Brain and Cerebrospinal Fluid Reveals Early Changes in Energy Metabolism Associated with Microglia and Astrocyte Activation

90. P2-294: RETROSPECTIVE REVIEW OF CLINICAL FEATURES OF ABCA7 LOSS-OF-FUNCTION MUTATION CARRIERS IN LATE-ONSET ALZHEIMER'S DISEASE

92. O4-10-01: CHARACTERIZING CELL TYPE-SPECIFIC EXPRESSION SIGNATURES IN BRAIN USING SINGLE CELL RNA SEQUENCING

93. P4-330: LONGITUDINAL BEHAVIOR OF BETA-AMYLOID IN NEURODEGENERATIVE DEMENTIAS

94. P2-427: THE RELATIONSHIP BETWEEN HISPANIC ETHNICITY, AMYLOID LOAD AND APOE4 CARRIER STATUS

95. O5‐02‐04: INTEGRATIVE GENOMIC PROFILING TO IDENTIFY GENES AND PATHWAYS ASSOCIATED WITH VASCULAR RISK IN AD

97. Molecular estimation of neurodegeneration pseudotime in older brains

98. Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

99. Systematic analysis of dark and camouflaged genes: disease-relevant genes hiding in plain sight

100. Development of monoclonal antibodies and quantitative ELISAs targeting insulin-degrading enzyme

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