300 results on '"Bito, Virginie"'
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52. Acute exposure to glycated proteins reduces cardiomyocyte contractile capacity
53. Complimentary Herbal Treatments Used in Meknes-Tafilalet Region (Morocco) to Manage Cancer
54. Western diet given to healthy rats mimics the human phenotype of diabetic cardiomyopathy
55. Glycolaldehyde-Derived High-Molecular-Weight Advanced Glycation End-Products Induce Cardiac Dysfunction through Structural and Functional Remodeling of Cardiomyocytes.
56. Effect of Exercise Intervention on Cardiac Function in Type 2 Diabetes Mellitus: A Systematic Review
57. Pyridoxamine improves survival and limits cardiac dysfunction after MI
58. HMW-AGEs induces Ca2+ imbalance in rat cardiomyocytes
59. Reduced mitochondrial respiration in the ischemic as well as in the remote nonischemic region in postmyocardial infarction remodeling
60. Assessment of left ventricular twist and untwist rate in a rat model of myocardial infarction
61. From Bone Marrow to Cardiac Atrial Appendage Stem Cells for Cardiac Repair: A Review
62. Possibilities and limitations for co-transplantation of cardiac atrial appendage stem cells and mesenchymal stem cells for myocardial repair
63. On the interpretation of second harmonic generation intensity profiles of striated muscle
64. A Systematic Approach for Assessing Ca2+Handling in Cardiac Myocytes: Figure 1.
65. Measuring Ca2+ Sparks in Cardiac Myocytes
66. Assessing Ca2+-Removal Pathways in Cardiac Myocytes
67. Characterizing the Trigger for Sarcoplasmic Reticulum Ca2+Release in Cardiac Myocytes
68. Basic Methods for Monitoring Intracellular Ca2+ in Cardiac Myocytes Using Fluo-3
69. Measuring Sarcoplasmic Reticulum Ca2+Content, Fractional Release, and Ca2+Buffering in Cardiac Myocytes
70. Melusin protects from cardiac rupture and improves functional remodelling after myocardial infarction
71. Selective Modulation of Coupled Ryanodine Receptors During Microdomain Activation of Calcium/Calmodulin-Dependent Kinase II in the Dyadic Cleft
72. Ruthenium red as an effective blocker of calcium and sodium currents in guinea-pig isolated ventricular heart cells
73. Melusin protects from cardiac rupture and improves functional remodelling after myocardial infarction
74. ACE-inhibition, but not weight reduction restores cardiomyocyte response to β-adrenergic stimulation in the metabolic syndrome
75. Combined Na + /Ca 2+ Exchanger and L-Type Calcium Channel Block as a Potential Strategy to Suppress Arrhythmias and Maintain Ventricular Function
76. FKBP12.6 overexpression does not protect against remodelling after myocardial infarction
77. Phospholamban ablation in hearts expressing the high affinity SERCA2b isoform normalizes global Ca2+ homeostasis but not Ca2+-dependent hypertrophic signaling
78. Treatment of male mice with estrogen for the study of contractile dysfunction
79. Estrogen induces cardiomyocyte contractility via an MLCK/MRLC‐dependent mechanism
80. 3-D Localization of Subcellular Ca2+ Release Reveals a Cytoskeletal Dependence of RyR Activation
81. Transcriptome Characterization of Estrogen-Treated Human Myocardium Identifies Myosin Regulatory Light Chain Interacting Protein as a Sex-Specific Element Influencing Contractile Function
82. Abstract P089: Estradiol Impairs Cardiac Contractile Function in Male Rodents
83. Subcellular Heterogeneity of Ryanodine Receptor Properties in Ventricular Myocytes with Low T-Tubule Density
84. Sex‐specific regulation of contractile function by 17β‐estradiol in mouse cardiomyocytes
85. Inhibition of the calcium-activated chloride current in cardiac ventricular myocytes by N-(p-amylcinnamoyl)anthranilic acid (ACA)
86. Ultrastructural and Functional Remodeling of the Coupling Between Ca 2+ Influx and Sarcoplasmic Reticulum Ca 2+ Release in Right Atrial Myocytes From Experimental Persistent Atrial Fibrillation
87. Response to Letter Regarding Article, “Ventricular Phosphodiesterase-5 Expression Is Increased in Patients With Advanced Heart Failure and Contributes to Adverse Ventricular Remodeling After Myocardial Infarction in Mice”
88. Early Exercise Training After Myocardial Infarction Prevents Contractile, But Not Electrical Remodeling Or Hypertrophy
89. FKBP12.6 Overexpression Blunts Cardiomyocyte Remodeling After Left-ventricular Pressure-overload
90. Ventricular Phosphodiesterase-5 Expression Is Increased in Patients With Advanced Heart Failure and Contributes to Adverse Ventricular Remodeling After Myocardial Infarction in Mice
91. Abstract 1534: Structural and Functional Remodeling of the Coupling between Sarcolemmal Ca 2+ Channels and the Ryanodine Receptor in Atrial Fibrillation
92. Pharmacological Inhibition of Na/Ca Exchange Results in Increased Cellular Ca 2+ Load Attributable to the Predominance of Forward Mode Block
93. Remodeling of T-Tubules and Reduced Synchrony of Ca 2+ Release in Myocytes From Chronically Ischemic Myocardium
94. Cellular basis for triggered ventricular arrhythmias that occur in the setting of compensated hypertrophy and heart failure: considerations for diagnosis and treatment
95. Abstract 319: Cardiomyocyte-specific Overexpression of Type 5 Phosphodiesterase Impairs Postinfarction Myocardial Function and Left Ventricular Remodeling.
96. Role of PI3K in mediating short- and long-term effects of IGF-1 on calcium handling in adult cardiac myocytes
97. Relation between loss of t-tubules, Na/Ca exchange and ryanodine receptor function in myocardial infarction
98. Inactivation of Smad5 in Endothelial Cells and Smooth Muscle Cells Demonstrates that Smad5 Is Required for Cardiac Homeostasis
99. Early Exercise Training Normalizes Myofilament Function and Attenuates Left Ventricular Pump Dysfunction in Mice With a Large Myocardial Infarction
100. Reduced Force Generating Capacity in Myocytes From Chronically Ischemic, Hibernating Myocardium
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