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3. Proteoform-resolved profiling of plasminogen activation reveals novel abundant phosphorylation site and primary N-terminal cleavage site

4. Proteoform-resolved profiling of plasminogen activation reveals novel abundant phosphorylation site and primary N-terminal cleavage site

12. Model for surface-dependent factor XII activation: the roles of factor XII heavy chain domains

15. A Common Fold Mediates Vertebrate Defense and Bacterial Attack

20. Inside Cover: Application and Structural Analysis of Triazole‐Bridged Disulfide Mimetics in Cyclic Peptides (Angew. Chem. Int. Ed. 28/2020)

22. Effective targeting of intact and proteolysed CDCP1 for imaging and treatment of pancreatic ductal adenocarcinoma

23. Anti-CDCP1 immuno-conjugates for detection and inhibition of ovarian cancer

26. Structure and Function Characterization of the a1a2 Motifs of Streptococcus pyogenes M Protein in Human Plasminogen Binding

29. A cyclic peptide inhibitor of the iNOS–SPSB protein–protein interaction as a potential anti-infective agent

31. A Cyclic Peptide Inhibitor of the iNOS–SPSB Protein–Protein Interaction as a Potential Anti-Infective Agent

32. X-ray crystal structure of plasmin with tranexamic acid–derived active site inhibitors

33. Structural studies of plasmin inhibition.

36. Structure of the poly-C9 component of the complement membrane attack complex

38. Stability of the Octameric Structure Affects Plasminogen-Binding Capacity of Streptococcal Enolase

39. Conformational Changes during Pore Formation by the Perforin-Related Protein Pleurotolysin

41. Maspin is not required for embryonic development or tumour suppression

42. Defining the interaction of perforin with calcium and the phospholipid membrane

43. Perforin forms transient pores on the target cell plasma membrane to facilitate rapid access of granzymes during killer cell attack

45. The Subtilisin-Like Protease AprV2 Is Required for Virulence and Uses a Novel Disulphide-Tethered Exosite to Bind Substrates

49. The MACPF/CDC family of pore-forming toxins

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