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As the core of the national administration, the resolutions of the Executive Yuan Session are the basis for the subsequent actions of other executive authorities at all levels, and the relevant written records not only serve as the basis for the overall administration of the country, but are also important for the continuous development of the records of the past sessions, which are a reflection of the trajectory of the national development. In view of this, this study focuses on the Executive Yuan Session's Minutes of the National Government (...), and explores the functions, rules, and roles of Executive Yuan Session and the contents, structure, and characteristics of Executive Yuan Session's Minutes through documentary crawling and collation. This paper also discusses the editions, current conditions, and value of the Executive Yuan Session's Minutes held by National Archives Administration, National Development Council (NAA), and serves as a reference for the relevant institutions and subsequent research. [ABSTRACT FROM AUTHOR]

Cooking oil fumes (COF) exposure was demonstrated to be associated with lung cancer development in Taiwanese nonsmoking women. Previous studies identified Cox-2 overexpression and oxidative DNA damage in lung adenocarcinoma cells after exposure to COF. Involvement of COF in lung tumorigenesis may be associated with cell survival, as well as proliferation of lung adenocarcinoma. To test this hypothesis, A549, a lung adenocarcinoma cell line, was used, and MTT assay data showed that the cell viability of A549 was significantly increased in a concentration-dependent manner by COF treatment for 48h. Flow cytometery results indicated that the proportion of A549 cell at S-phase was markedly increased after exposure of COF. To elucidate whether the antiapoptotic c-IAP2 (IAP2) was involved in COF-improved cell survival, IAP2 protein levels was determined by Western blot, and the results showed it was significantly induced by COF in a concentration-dependent manner. Moreover, the suppression of BAY, a nuclear factor (NF)-κB binding inhibitor, or the COF-induced IAP2 protein levels indicated that NF-κB activation by COF may partly be involved in IAP2 induction. These results showed that the positive impact of COF on cell survival and proliferation of A549 lung tumor cells may be through an induction of IAP2 overexpression. [ABSTRACT FROM AUTHOR]