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A model of the geological evolution of the Japan and Okhotsk seas was developed based on radioisotope age, mineralogical, and isotope-geochemical study of the Late Mesozoic–Cenozoic volcanic rocks. The geodynamic settings of volcanic stages were determined as follows: (1) the Late Cretaceous continental-margin (calc-alkaline), (2) the Eocene transform-margin (adakite) in the Sea of Okhotsk, (3) the Miocene–Pliocene marginal sea (alkaline basaltic) in the Sea of Japan, and (4) the Pliocene–Pleistocene island arc (calc-alkaline) in the southern Sea of Okhotsk. It has been established that parental magmas were derived from subcontinental lithosphere, oceanic asthenosphere, and lower-mantle plume-continental (CAB) and plume-oceanic (OIB) sources. Geodynamic settings changed from the Late Cretaceous subduction to the Maastrichtian–Pliocene transform margin. The transform margin setting involved destruction and extension, maximum oceanic-margin spreading (end of the Early Miocene–beginning of the Middle Miocene), and post-spreading lower mantle plume upwelling (the Middle Miocene – Pliocene). It was completed by the resumption of the Pliocene–Pleistocene subduction of the Pacific plate beneath the Eurasian continent. [ABSTRACT FROM AUTHOR]

Since the late 1980s, a birnaviral gill disease has been occurring in Japanese eels Anguilla japonica reared in warmwater ponds in western regions in Japan. Diseased eels mostly displayed marked formations of aneurysmal hematomas within gill lamellae and high mortalities. Histological examination revealed necrosis of pillar cells and subsequent aggregation of erythrocytes inside the lamellar capillaries, and proliferation of interlamellar epithelia onto the lamellae. Gastric gland cells were also necrotized. Electron microscopy revealed birnavirus infection in lamellar pillar cells. The causative birnavirus was isolated and cultured in fish cell lines and was found to be related to an infectious pancreatic necrosis virus (IPNV) Sp serotype by neutralization tests. The viral pathogenicity was confirmed by the results of histopathological examinations and infectivity experiments.