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1. Effects of Cellular Pathway Disturbances on Misfolded Superoxide Dismutase-1 in Fibroblasts Derived from ALS Patients.

2. Expression of wild-type human superoxide dismutase-1 in mice causes amyotrophic lateral sclerosis.

3. Mutant superoxide dismutase-1 indistinguishable from wild-type causes ALS.

4. A novel ALS SOD1 C6S mutation with implications for aggregation related toxicity and genetic counseling.

5. A novel SOD1 splice site mutation associated with familial ALS revealed by SOD activity analysis.

6. Large-scale SOD1 mutation screening provides evidence for genetic heterogeneity in amyotrophic lateral sclerosis.

7. Comprehensive analysis to explain reduced or increased SOD1 enzymatic activity in ALS patients and their relatives.

8. A 50 bp deletion in the SOD1 promoter lowers enzyme expression but is not associated with ALS in Sweden.

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