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Overall, we suggest that PARK2 gene mutations should be considered in dystonia-parkinsonism with an onset in the infantile period to childhood and adolescence and with clinical features that in some cases may mimic CP. Recently, an increasing number of genetic conditions presenting with childhood dystonia-parkinsonism have been recognized, mainly caused by mutations in genes encoding for enzymes involved in in the biosynthesis of monoaminergic neurotransmitters, such as GCH1, SPR, or TH or in brain iron accumulation syndromes [[6]]. In our patient, the presence of dystonia and parkinsonism and the positive SPECT led us to perform an extensive genetic panel for movement disorders. [Extracted from the article]

The first video with the patient seated on a chair is supplementary video 2: I Post-DBS neurological examination i . The second video where the patient is seated on a wheelchair with pink t- shirt is supplementary video 1: I Pre-DBS neurological examination i . Correction to: A case of childhood-onset dystonia-parkinsonism due to homozygous parkin mutations and effect of globus pallidus deep brain stimulation. [Extracted from the article]