Search

Purpose: The purpose of this research was to compare the thickness of different retinal layers between dyslexic subjects and controls at the macula by means of optical coherence tomography (OCT). Methods: Each patient underwent a complete ophthalmic examination including macular OCT in both eyes. The thickness of the fovea, four sectors of the parafovea and four sectors of the perifovea were obtained according to the Early Treatment Diabetic Retinopathy Study protocol in the following 13 segmentations: complete retina, outer retina, inner retina, retinal nerve fibre layer (RNFL), ganglion cell layer (GCL), inner plexiform layer (IPL), inner nuclear layer (INL), outer plexiform layer (OPL), outer nuclear layer (ONL), INL + OPL + ONL, IPL + INL, OPL + ONL and ganglion cell complex (RNFL + GCL + IPL). The results of the right and left eyes were independently compared between groups. Results: The thickness in all the four sectors of the parafovea was found significantly higher in the right and in the left eyes in the dyslexic group in the following segmentations: complete retina, inner retina, middle retinal layers and OPL + ONL. Several macular sectors were also thicker in IPL + INL, INL, IPL and ONL in dyslexia. In contrast, no thickness differences between both groups were found in any of the sectors of RNFL, GCL, GCC, or outer retina in any eye. Conclusions: Anatomy of the macula is different in dyslexic and normoreader subjects in middle retinal layers, especially in the parafovea. This fact suggests that the macula may play an important role in the development of dyslexia. [ABSTRACT FROM AUTHOR]

Purpose: To describe an observational case of closed‐angle glaucoma secondary to uveal effusion syndrome after a first episode of posterior scleritis. Methods: Morales Meseguer University Hospital. Ophthalmology service. Uveitis section. Results: A 76‐year‐old woman presented to the emergency department with a red, painful right eye (RO) and decreased visual acuity (VA). Initial VA was light perception and intraocular pressure was 40 mmHg, presenting a narrowed chamber and corticonuclear cataract. The fundus was not assessable due to media opacity. Optical coherence tomography showed choroidal folds in the left eye, and the OD was unapproachable. B‐mode ocular ultrasound showed scleral thickening of 2 mm in both eyes and a superior temporal hematic choroidal detachment in the left eye. She was diagnosed with acute glaucoma secondary to uveal effusion after the first outbreak of posterior scleritis. In the emergency department she was treated with intravenous mannitol, acetazolamide orally and hypotensive eye drops. Acute angle‐closure glaucoma and choroidal detachment remitted under systemic steroid treatment, cycloplegic eye drops and topical hypotensive agents. After screening for pathologies associated with posterior scleritis, idiopathic origin of the condition was concluded. Conclusions: Patients with posterior scleritis may develop uveal effusion syndrome, which can lead to secondary angle‐closure glaucoma. A screening for pathologies associated with posterior scleritis (Wegener's, rheumatoid arthritis, herpes zoster, tumours, porphyria, post‐surgical or pharmacological causes) should be performed, although most cases are idiopathic. Treatment differs from glaucoma due to pupillary block, being necessary the use of systemic corticosteroids. [ABSTRACT FROM AUTHOR]