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26 results on '"Bito, Virginie"'

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1. Glycolaldehyde-Derived High-Molecular-Weight Advanced Glycation End-Products Induce Cardiac Dysfunction through Structural and Functional Remodeling of Cardiomyocytes.

2. Differentiation of Human Cardiac Atrial Appendage Stem Cells into Adult Cardiomyocytes: A Role for the Wnt Pathway?

3. Acute exposure to glycated proteins reduces cardiomyocyte contractile capacity.

4. Reduced mitochondrial respiration in the ischemic as well as in the remote nonischemic region in postmyocardial infarction remodeling.

5. Possibilities and limitations for co-transplantation of cardiac atrial appendage stem cells and mesenchymal stem cells for myocardial repair.

6. Cardiac atrial appendage stem cells engraft and differentiate into cardiomyocytes in vivo: A new tool for cardiac repair after MI.

7. Measuring Ca²⁺ sparks in cardiac myocytes.

8. Assessing Ca²⁺-removal pathways in cardiac myocytes.

9. A systematic approach for assessing Ca²⁺ handling in cardiac myocytes.

10. Characterizing the trigger for sarcoplasmic reticulum Ca2+ release in cardiac myocytes.

11. Measuring sarcoplasmic reticulum Ca2+ content, fractional release, and Ca2+ buffering in cardiac myocytes.

12. Basic methods for monitoring intracellular Ca2+ in cardiac myocytes using Fluo-3.

13. Selective modulation of coupled ryanodine receptors during microdomain activation of calcium/calmodulin-dependent kinase II in the dyadic cleft.

14. ACE-inhibition, but not weight reduction restores cardiomyocyte response to β-adrenergic stimulation in the metabolic syndrome.

15. T-tubule remodelling and ryanodine receptor organization modulate sodium-calcium exchange.

16. Transcriptome characterization of estrogen-treated human myocardium identifies myosin regulatory light chain interacting protein as a sex-specific element influencing contractile function.

17. Subcellular heterogeneity of ryanodine receptor properties in ventricular myocytes with low T-tubule density.

18. Inhibition of the calcium-activated chloride current in cardiac ventricular myocytes by N-(p-amylcinnamoyl)anthranilic acid (ACA).

19. Ultrastructural and functional remodeling of the coupling between Ca2+ influx and sarcoplasmic reticulum Ca2+ release in right atrial myocytes from experimental persistent atrial fibrillation.

20. Pharmacological inhibition of na/ca exchange results in increased cellular Ca2+ load attributable to the predominance of forward mode block.

21. Remodeling of T-tubules and reduced synchrony of Ca2+ release in myocytes from chronically ischemic myocardium.

22. Window Ca2+ current and its modulation by Ca2+ release in hypertrophied cardiac myocytes from dogs with chronic atrioventricular block.

23. Reduced force generating capacity in myocytes from chronically ischemic, hibernating myocardium.

24. The amiodarone derivative KB130015 [2-methyl-3-(3,5-diiodo-4-carboxymethoxybenzyl)benzofuran] induces an Na+-dependent increase of [Ca2+] in ventricular myocytes.

25. Cellular mechanisms of contractile dysfunction in hibernating myocardium.

26. Reduced synchrony of Ca2+ release with loss of T-tubules-a comparison to Ca2+ release in human failing cardiomyocytes.

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