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Your search keyword '"Chung, Js"' showing total 14 results

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14 results on '"Chung, Js"'

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1. DC-HIL/Gpnmb Is a Negative Regulator of Tumor Response to Immune Checkpoint Inhibitors.

2. Blocking Monocytic Myeloid-Derived Suppressor Cell Function via Anti-DC-HIL/GPNMB Antibody Restores the In Vitro Integrity of T Cells from Cancer Patients.

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3. Melanoma-Derived Soluble DC-HIL/GPNMB Promotes Metastasis by Excluding T-Lymphocytes from the Pre-Metastatic Niches.

4. DC-HIL-expressing myelomonocytic cells are critical promoters of melanoma growth.

5. DC-HIL+ CD14+ HLA-DR no/low cells are a potential blood marker and therapeutic target for melanoma.

6. The DC-HIL/syndecan-4 pathway regulates autoimmune responses through myeloid-derived suppressor cells.

7. The DC-HIL ligand syndecan-4 is a negative regulator of T-cell allo-reactivity responsible for graft-versus-host disease.

8. Sézary syndrome cells overexpress syndecan-4 bearing distinct heparan sulfate moieties that suppress T-cell activation by binding DC-HIL and trapping TGF-beta on the cell surface.

9. DC-HIL/glycoprotein Nmb promotes growth of melanoma in mice by inhibiting the activation of tumor-reactive T cells.

10. Binding of DC-HIL to dermatophytic fungi induces tyrosine phosphorylation and potentiates antigen presenting cell function.

11. Gpnmb is a melanosome-associated glycoprotein that contributes to melanocyte/keratinocyte adhesion in a RGD-dependent fashion.

12. The DC-HIL/syndecan-4 pathway inhibits human allogeneic T-cell responses.

13. Syndecan-4 mediates the coinhibitory function of DC-HIL on T cell activation.

14. DC-HIL is a negative regulator of T lymphocyte activation.