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1. Molecular and behavioral consequences of Ube3a gene overdosage in mice

2. Dual-isoform hUBE3A gene transfer improves behavioral and seizure outcomes in Angelman syndrome model mice

3. Dual-isoform hUBE3A gene transfer improves behavioral and seizure outcomes in Angelman syndrome model mice

4. Ube3a reinstatement mitigates epileptogenesis in Angelman syndrome model mice

5. Subcellular organization of UBE3A in human cerebral cortex

6. Enhanced Operant Extinction and Prefrontal Excitability in a Mouse Model of Angelman Syndrome

7. Decreased Axon Caliber Underlies Loss of Fiber Tract Integrity, Disproportional Reductions in White Matter Volume, and Microcephaly in Angelman Syndrome Model Mice

8. Delayed loss of UBE3A reduces the expression of Angelman syndrome-associated phenotypes

9. GABAergic Neuron-Specific Loss of Ube3a Causes Angelman Syndrome-Like EEG Abnormalities and Enhances Seizure Susceptibility

10. Allelic specificity of Ube3a Expression In The Mouse Brain During Postnatal Development

11. Loss of UBE3A from TH-expressing neurons suppresses GABA co-release and enhances VTA-NAc optical self-stimulation

12. A new synaptic player leading to autism risk: Met receptor tyrosine kinase

13. Conserved Subcortical and Divergent Cortical Expression of Proteins Encoded by Orthologs of the Autism Risk Gene MET

14. Evidence of cell-nonautonomous changes in dendrite and dendritic spine morphology in the met-signaling-deficient mouse forebrain

15. Dissociation of locomotor and cerebellar deficits in a murine Angelman syndrome model

16. Subcellular organization of UBE3A in neurons

17. Angelman syndrome: insights into genomic imprinting and neurodevelopmental phenotypes

18. Dynamic gene and protein expression patterns of the autism-associated met receptor tyrosine kinase in the developing mouse forebrain

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