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18 results on '"Maximilian Kauer"'

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1. Schwann cell plasticity regulates neuroblastic tumor cell differentiation via epidermal growth factor-like protein 8

2. EWS-FLI1 perturbs MRTFB/YAP-1/TEAD target gene regulation inhibiting cytoskeletal autoregulatory feedback in Ewing sarcoma

3. MEF2C-dysregulated pediatric T-cell acute lymphoblastic leukemia is associated withCDKN1Bdeletions and a poor response to glucocorticoid therapy

4. High-throughput RNAi screen in Ewing sarcoma cells identifies leucine rich repeats and WD repeat domain containing 1 (LRWD1) as a regulator of EWS-FLI1 driven cell viability

5. Metronomic topotecan impedes tumor growth of MYCN-amplified neuroblastoma cells in vitro and in vivo by therapy induced senescence

6. Genetic alterations in glucocorticoid signaling pathway components are associated with adverse prognosis in children with relapsedETV6/RUNX1-positive acute lymphoblastic leukemia

7. Variability in functional p53 reactivation by PRIMA-1Met/APR-246 in Ewing sarcoma

8. Notch signalling is off and is uncoupled from HES1 expression in Ewing's sarcoma

9. ETV6/RUNX1-positive relapses evolve from an ancestral clone and frequently acquire deletions of genes implicated in glucocorticoid signaling

10. Intercohort Gene Expression Co-Analysis Reveals Chemokine Receptors as Prognostic Indicators in Ewing's Sarcoma

11. The role of the Janus-faced transcription factor PAX5-JAK2 in acute lymphoblastic leukemia

12. Identification of RISC-associated adenoviral microRNAs, a subset of their direct targets, and global changes in the targetome upon lytic adenovirus 5 infection

13. Oncogenic ETS fusions deregulate E2F3 target genes in Ewing sarcoma and prostate cancer

14. Hypoxia modulates EWS-FLI1 transcriptional signature and enhances malignant properties of Ewing's sarcoma cells in-vitro

15. A molecular function map of Ewing's sarcoma

16. O-GlcNAcylation is involved in the transcriptional activity of EWS-FLI1 in Ewing’s sarcoma

17. Enriched Bone Marrow Derived Disseminated Neuroblastoma Cells Can Be a Reliable Source for Gene Expression Studies—A Validation Study

18. The Leukemia-Specific Fusion Gene ETV6/RUNX1 Perturbs Distinct Key Biological Functions Primarily by Gene Repression

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