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3 results on '"Emma Burt"'

1. In Ph+BCR-ABL1P210+ acute lymphoblastic leukemia the e13a2 (B2A2) transcript is prevalent

Author

Daniel Coriu, Audrey Bidet, BJ Milner, Michele Baccarani, Ilaria Iacobucci, Sabina Chiaretti, Samia Menif, A Ayala, Stephen E. Langabeer, Beatrice Borsellino, Elisabeth Paietta, Emma Burt, Ana Ines Prado, Lorenzo Comba, Sabine Jeromin, Orietta Spinelli, Mario Luppi, Barbara Scappini, Monica Crugnola, Jiri Mayer, Letizia Foroni, Jacqueline Maier, Sara Galimberti, Irena Preložnik Zupan, Francesco Passamonti, Francesca Lunghi, Neelam Varma, Anna Candoni, Jeroen Janssen, Mario Annunziata, Francesco Albano, Poonkuzhali Balasubramanian, Thomas Lion, Giovanna Rege-Cambrin, Valentina Polli, Carolina Terragna, Behzad Poopak, Ombretta Annibali, Barbara Izzo, Renata Zadro, Victor Salinas-Viedma, Francesco Di Raimondo, Tulika Seth, Robin Foà, Baccarani, M., Iacobucci, I., Chiaretti, S., Foa', R., Balasubramanian, P., Paietta, E., Foroni, L., Jeromin, S., Izzo, B., Spinelli, O., Varma, N., Menif, S., Terragna, C., Seth, T., Bidet, A., Coriu, D., Lunghi, F., Mayer, J., Scappini, B., Langabeer, S., Maier, J., Burt, E., Candoni, A., Albano, F., Luppi, M., Zupan, I., Lion, T., Zadro, R., di Raimondo, F., Poopak, B., Rege-Cambrin, G., Annunziata, M., Ayala, A., Salinas-Viedma, V., Ines Prado, A., Milner, B., Galimberti, S., Janssen, J., Polli, V., Comba, L., Borsellino, B., Annibali, O., Crugnola, M., Passamonti, F., Hematology, and CCA - Cancer biology and immunology

Subjects
Adult, Male, Cancer Research, Adolescent, Lymphoblastic Leukemia, bcr-abl, Fusion Proteins, bcr-abl, Young Adult, Myelogenous, Text mining, Leukemia, Myelogenous, Chronic, BCR-ABL Positive, medicine, Humans, Age Factor, Chronic, Young adult, Child, Proto-Oncogene Proteins c-abl, Aged, B-Lymphocytes, Leukemia, business.industry, B-Lymphocyte, Age Factors, breakpoint cluster region, Fusion Proteins, Hematology, Middle Aged, Precursor Cell Lymphoblastic Leukemia-Lymphoma, medicine.disease, Fusion protein, Oncology, Multicenter study, Cancer research, Female, BCR-ABL Positive, business, Human
Published
2019

2. In Ph+BCR-ABL1

Author

Michele, Baccarani, Ilaria, Iacobucci, Sabina, Chiaretti, Robin, Foà', Poonkuzhali, Balasubramanian, Elisabeth, Paietta, Letizia, Foroni, Sabine, Jeromin, Barbara, Izzo, Orietta, Spinelli, Neelam, Varma, Samia, Menif, Carolina, Terragna, Tulika, Seth, Audrey, Bidet, Daniel, Coriu, Francesca, Lunghi, Jiri, Mayer, Barbara, Scappini, Stephen, Langabeer, Jacqueline, Maier, Emma, Burt, Anna, Candoni, Francesco, Albano, Mario, Luppi, Irena, Zupan, Thomas, Lion, Renata, Zadro, Francesco, di Raimondo, Behzad, Poopak, Giovanna, Rege-Cambrin, Mario, Annunziata, Ana, Ayala, Victor, Salinas-Viedma, Ana, Ines Prado, Benedict, Milner, Sara, Galimberti, Jeroen, Janssen, Valentina, Polli, Lorenzo, Comba, Beatrice, Borsellino, Ombretta, Annibali, Monica, Crugnola, and Francesco, Passamonti

Subjects
Adult, Male, B-Lymphocytes, Adolescent, Age Factors, Fusion Proteins, bcr-abl, Middle Aged, Precursor Cell Lymphoblastic Leukemia-Lymphoma, Young Adult, Leukemia, Myelogenous, Chronic, BCR-ABL Positive, Humans, Female, Child, Proto-Oncogene Proteins c-abl, Aged
Published
2019

3. Caenorhabditis elegans in the study of SMN-interacting proteins: a role for SMI-1, an orthologue of human Gemin2 and the identification of novel components of the SMN complex

Author

Paula R. Towers, Emma Burt, and David B. Sattelle

Subjects
animal diseases, Two-hybrid screening, Molecular Sequence Data, Nerve Tissue Proteins, Cellular and Molecular Neuroscience, Developmental Neuroscience, SMN complex, RNA interference, SMN Complex Proteins, Two-Hybrid System Techniques, Genetic model, medicine, Animals, Humans, Amino Acid Sequence, Cloning, Molecular, RNA, Small Interfering, Caenorhabditis elegans, Caenorhabditis elegans Proteins, Cyclic AMP Response Element-Binding Protein, Gene, Genetics, Sequence Homology, Amino Acid, biology, Reverse Transcriptase Polymerase Chain Reaction, RNA-Binding Proteins, Spinal muscular atrophy, biology.organism_classification, medicine.disease, nervous system diseases, nervous system
Abstract

Spinal muscular atrophy is a common neuromuscular disorder caused by mutations in the survival motor neuron (SMN) gene. In mammals, SMN is tightly associated with Gemin2. To gain further insight into the functions of SMN and Gemin2, we have cloned and sequenced smi-1 (Survival of Motor neuron-Interacting protein 1), a C. elegans homologue of the human Gemin2 gene. We show that the SMI-1 expression pattern and RNA interference phenotype show considerable overlap with that previously reported for SMN-1. Finally, we demonstrate that the SMN-1 and SMI-1 proteins directly interact. Having demonstrated the utility of the C. elegans genetic model for investigating genes encoding SMN-interacting proteins, we have undertaken a yeast two-hybrid screen of a C. elegans cDNA library to identify novel proteins that interact with SMN-1. We show the direct interaction of SMN-1 with nine novel proteins, several of which may be involved in RNA metabolism.

Published
2006

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