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This study aimed to examine the effects of manipulating the rest intervals during sprint interval training (SIT) on post-exercise hypotension and within-session oxygen consumption.Thirty healthy, trained adults (aged 30.9 ± 8.7 years; 14 males, 16 females; BMI 22.1 ± 2.3 kg/m 2 ; VO 2 max 50.7 ± 7.8 ml/kg/min) completed two different SIT protocols (4x 30-seconds all-out cycling sprints) with a one-week washout period. Sprint bouts were separated by either 1 (R1) or 3 (R3) minutes of active recovery. Both before and throughout the 45 min after the training, peripheral systolic (pSBP) and diastolic (pDBP) blood pressure, central systolic (cSBP) and diastolic (cDBP) blood pressure, aortic pulse wave velocity (aPWV), stroke volume (SV), and heart rate (HR) were assessed. Throughout the SIT protocols, oxygen consumption (VO 2 ) was monitored.There were no significant differences in time spent at 75%, 85%, 95%, and 100% of maximal VO 2 between R1 and R3. After R3, there was a significant reduction in pSBP, pDBP, cSBP, cDBP, and aPWV. After R1, there were no changes in the respective parameters. There were significant interaction effects in pSBD ( p  < 0.001), pDBP ( p  < 0.001), cSBP ( p  < 0.001), cDBP ( p  = 0.001), and aPWV ( p  = 0.033). HR significantly increased after both conditions. Only R1 resulted in a significant reduction in SV.Longer resting intervals during SIT bouts seem to result in more substantial post-exercise hypotension effects. Time spent at a high percentage of maximal VO 2 was not affected by rest interval manipulation.

Purpose: To identify whether the use of active recovery (ACT) the day after high-intensity interval training (HIIT) benefits recovery and to assess whether individual responses to ACT are repeatable., Methods: Eleven well-trained, male intermittent-sport athletes (age: 25.5 ± 1.8 y) completed 4 HIIT sessions, each separated by a 2-week washout period. Of the 4 sessions, 2 were followed by passive recovery (PAS) and 2 by 60 minutes of moderate biking (ACT) 24 hours postexercise in the following sequences: ACT→PAS→ACT→PAS or PAS→ACT→PAS→ACT. Before and after HIIT and after 24 and 48 hours of recovery, maximal voluntary isometric strength (MVIC), countermovement jump height (CMJ), tensiomyographic markers of muscle fatigue (TMG), serum concentration of creatine kinase (CK), muscle soreness (MS), and perceived stress state (PS) were determined., Results: A 3-way repeated-measure analysis of variance with a triple-nested random effects model revealed a significant (P < .05) fatigue-related time effect of HIIT on markers of fatigue (MVIC↓; CMJ↓; TMG↑; CK↑; MS↑; PS↑). No significant (P > .05) main effect of recovery strategy was detected. In 9 subjects, the individual results revealed inconsistent and nonrepeatable responses to ACT, while a consistent and repeatable positive or negative response to ACT was found in 2 individuals., Conclusions: The repeated failure of ACT to limit the severity of fatigue was found both at the group level and with most individuals. However, a small percentage of athletes may be more likely to benefit repeatedly from either ACT or PAS. Therefore, the use of ACT should be individualized.