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2018 JUL 28 (NewsRx) -- By a News Reporter-Staff News Editor at Obesity, Fitness & Wellness Week -- A new study on Aldehydes - Malondialdehyde is now available. According to [...]

2018 MAY 26 (NewsRx) -- By a News Reporter-Staff News Editor at Obesity, Fitness & Wellness Week -- Researchers detail new data in Caenorhabditis elegans. According to news reporting from [...]

ABSTRACT. The effect of sulfur dioxide (S[O.sup.2]) on brain antioxidant status, lipid peroxidation, and somatosensory evoked potentials (SEPs) was investigated in diabetic rats. A total of 40 rats were divided [...]

A foodborne disease outbreak characterized by vomiting, abdominal pain and diarrhea, involving mostly children was investigated. Epidemiological, hospital and laboratory investigations indicated that the disease outbreak was associated with consumption of rancid biscuits abandoned in the street corner in a crowded locality of old city Hyderabad. The offensive flavors of rancidity were masked by the strong pineapple flavor used in the biscuits. Rancidity of the biscuits was confirmed by high peroxide value and acidity of extracted fat. Bacterial contamination was excluded by the total aerobic plate counts and negative tests for Staphylococcus and Salmonella., (Key Words: foodborne disease; lipid peroxidation; rancidity.) INTRODUCTION Foodborne diseases are a serious health hazard and important cause of morbidity and mortality in developing countries. Most cases go unreported and [...]

The ability of [beta]-carotene (BC) to reduce lipid peroxidation in humans was investigated. In this randomized double-blind controlled trial, 42 nonsmokers and 28 smokers received either 20 mg BC or placebo daily for 4 wk. Twenty-five smokers and 38 nonsmokers completed the trial. Changes in plasma BC concentrations increased significantly (P < 0.0005) and to the same extent in both groups supplemented with BC. There were no significant changes among the placebo groups. At baseline, lipid peroxidation measured by breath-pentane output (BPO) was significantly higher in the two smoking groups (BC: 8.8 [+ or -] 1.1, placebo: 9.4 [+ or -] 1.4 pmol [kg.sup.-1][multiplied by][min.sup.-1]) than in the nonsmoking groups (BC: 5.7 [+ or -] 0.5, placebo: 5.9 [+ or -] 0.6 pmol[multiplied by][kg.sup.-1][multiplied by][min.sup.-1]) (P < 0.005). BPO decreased significantly only in smokers receiving BC (6.5 [+ or -] 0.7 pmol[multiplied by][kg.sup.-1][multiplied by][min.sup < 0.04). Changes in breath-ethane output were not significant. Therefore, lipid peroxidation measured by BPO is significantly higher in smokers than in nonsmokers and is reduced by BC supplementation in smokers. There was no significant change (95% CI - 1.26, 1.12) in BPO when nonsmokers received BC.