3 results on '"Armstrong, B."'
Search Results
2. Correction to: Early-stage studies to larger-scale trials: investigators' perspectives on scaling-up childhood obesity interventions.
- Author
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von Klinggraef, L., Dugger, R., Okely, A. D., Lubans, D., Jago, R., Burkart, S., Weaver, R. G., Armstrong, B., Pfedderer, C. D., and Beets, M. W.
- Subjects
CHILDHOOD obesity ,BEHAVIORAL assessment - Abstract
ID5
7b "In the pilot study we had teachers delivering intervention and in the main trial we went to the core non-degree staff delivering the intervention. We were embedded in the local school boards and they saw it as very useful from both a health and an educational point of view and the larger scale trial was really just a larger scale longer version of the pilot study." ID13 Challenges Challenges encountered when scaling pilot studies 12a "In the pilot we had a small amount of group of teachers who were very motivated and who want to bring about a change but in our larger trial we had multiple teachers, multiple schools. [Extracted from the article] - Published
- 2022
- Full Text
- View/download PDF
3. Cortisol reactivity and weight gain among adolescents who vary in prenatal drug exposure.
- Author
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Armstrong, B., Buckingham‐Howes, S., and Black, M. M.
- Subjects
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ADRENAL gland physiology , *HYPOTHALAMUS physiology , *RISK of childhood obesity , *PITUITARY gland physiology , *CHILDHOOD obesity , *SALIVA analysis , *BIOMARKERS , *HYDROCORTISONE , *NEUROPSYCHOLOGICAL tests , *SEX distribution , *PHYSIOLOGICAL stress , *SUBSTANCE abuse in pregnancy , *BODY mass index , *DIAGNOSIS - Abstract
Summary: Objective: Low inhibitory control is linked with weight gain among youth. Inhibitory problems are associated with disruption to the hypothalamic–pituitary–adrenal axis cortisol response. Increased cortisol predicts appetite and weight gain (though may be gender specific). This study hypothesized that cortisol reactivity explains the association between inhibition and weight gain while considering the moderating factors of early stressors to the hypothalamic–pituitary–adrenal axis (e.g. prenatal‐drug exposure) and gender. Methods: Adolescents with prenatal‐drug exposure (n = 76) and non‐exposed comparison adolescents (NE; n = 61) completed the Conner's Continuous Performance Test and provided salivary cortisol samples. BMI z‐score were measured at the initial and 12‐month follow‐up evaluations. A bootstrapped moderated mediation analysis was conducted to test for conditional indirect effects of cortisol reactivity. Results: Lower inhibition was associated with increased cortisol reactivity among youth who were NE, and increased cortisol reactivity was associated with weight gain among girls. Cortisol reactivity mediated the relation between inhibition and BMI z‐score change for the girls in the group who was NE. Conclusion: Increased cortisol reactivity may play a mechanistic role in predicting weight gain among non‐prenatally drug‐exposed girls. Cortisol reactivity may be a biomarker for targeted interventions to improve biological regulation and ultimately health risk among girls. [ABSTRACT FROM AUTHOR]
- Published
- 2018
- Full Text
- View/download PDF
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