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Primary/secondary functional injury of cell membranes with deterioration of ionic potential of membrane electrical potential (MEP) bio-electromagnetic field, quantum energy and quantum electromagnetic radiation, such as signal transfer, which involves pathophysiological responses of the pathophysiological cellular act is considered as Maria&Irina Vasilieva syndrome (Electro-Ion Membrane Distress Syndrome; EIMDs), which causes chronic fatigue syndrome/myalgic encephalomyelitis (CFS/ME). EIMDs and mitochondrial acidosis internally established by disordered depolarization decrease selectively MEP of the inner mitochondrial membrane, where ATP decreases ubiquinone (CoQ10 synthesis is reduced) and the mitochondrial energy deficiency is established, during which the pores are damaged, Ca++ uniporter mPT pore, channel KACh, ChitoATP/MitoATP ratio, receptors: adenosine A1; purine P2, the metabolome of nitric oxide as well as aqua pore are involved. Immunocompromise (IC) CHAOS dissonance creates an electro-storm disorder or electroparalysis of MEP, and the persistence of cytokines induces the postresuscitation disease described by G. Litarczek, P. Safar and V. Negovsky. In the IC CHAOS survivors, a marker of CFS/ME was the study of the reduced erythrocyte deformation, E from the blood, which accompanies EIMDs, evaluating the membrane permeability and the absorption capacity of E, an osmometer dependent on the urea concentration. The traditional therapy in IC CHAOS survivors with dissonance did not ensure a steady decrease in CF/ME, the effect of which was observed after MOST-ELSO therapy associated with cryobioxeno (thymomyelospleno) extracorporeal perfusion. The slightly symptomatic CFS/ME observed after cryobioxeno perfusion was also considered as a universal bio mechanism of cryotherapy expressed by the effect of compression and reduction of “synairesis” proteins, by separating the liquid from the gel, caused by the reduction of water and the cell membranes and cells from deuterium D/²H, “heavy water” which inhibits some cleavage reactions. [ABSTRACT FROM AUTHOR]