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1. Early dynamic orchestration of immunologic mediators identifies multiply injured patients who are tolerant or sensitive to hemorrhage

2. Quality Control Measures and Validation in Gene Association Studies: Lessons for Acute Illness

3. The Use of Multiplexing to Identify Cytokine and Chemokine Networks in the Immune-Inflammatory Response to Trauma

4. A road map from single-cell transcriptome to patient classification for the immune response to trauma

5. Analysis of the Plasma Metabolome after Trauma, Novel Circulating Sphingolipid Signatures, and In-Hospital Outcomes

6. Unsupervised Clustering Analysis Based on MODS Severity Identifies Four Distinct Organ Dysfunction Patterns in Severely Injured Blunt Trauma Patients

7. An Enrichment Strategy Yields Seven Novel Single Nucleotide Polymorphisms Associated With Mortality and Altered Th17 Responses Following Blunt Trauma

8. What's New in Shock, June 2018?

9. Diurnal Variation in Systemic Acute Inflammation and Clinical Outcomes Following Severe Blunt Trauma

10. MPPED2 Polymorphism Is Associated With Altered Systemic Inflammation and Adverse Trauma Outcomes

11. An Aging-Related Single-Nucleotide Polymorphism is Associated With Altered Clinical Outcomes and Distinct Inflammatory Profiles in Aged Blunt Trauma Patients

12. Inflammation and Disease: Modelling and Modulation of the Inflammatory Response to Alleviate Critical Illness

13. Individual-specific principal component analysis of circulating inflammatory mediators predicts early organ dysfunction in trauma patients

14. Insights into the association between coagulopathy and inflammation: abnormal clot mechanics are a warning of immunologic dysregulation following major injury

15. Elevations in Circulating sST2 Levels Are Associated With In-Hospital Mortality and Adverse Clinical Outcomes After Blunt Trauma

16. Prehospital Hypotension Is Associated With Altered Inflammation Dynamics and Worse Outcomes Following Blunt Trauma in Humans*

17. The early evolving sex hormone environment is associated with significant outcome and inflammatory response differences after injury

18. IL33-mediated ILC2 activation and neutrophil IL5 production in the lung response after severe trauma: A reverse translation study from a human cohort to a mouse trauma model

19. From static to dynamic: a sepsis-specific dynamic model from clinical criteria in polytrauma patients

20. X Chromosome-Linked IRAK-1 Polymorphism Is a Strong Predictor of Multiple Organ Failure and Mortality Postinjury

22. Computational evidence for an early, amplified systemic inflammation program in polytrauma patients with severe extremity injuries

23. Young and Aged Blunt Trauma Patients Display Major Differences in Circulating Inflammatory Mediator Profiles after Severe Injury

24. Computational Analysis Supports an Early, Type 17 Cell-Associated Divergence of Blunt Trauma Survival and Mortality

25. [Untitled]

26. Persistence of Elevated Plasma CXCL8 Concentrations Following Red Blood Cell Transfusion in a Trauma Cohort

27. A Biohybrid Device for the Systemic Control of Acute Inflammation

28. Sepsis: From Pattern to Mechanism and Back

30. Elevated Admission Base Deficit Is Associated with a Complex Dynamic Network of Systemic Inflammation Which Drives Clinical Trajectories in Blunt Trauma Patients

31. Temporal Patterns of Circulating Inflammation Biomarker Networks Differentiate Susceptibility to Nosocomial Infection Following Blunt Trauma in Humans

32. Trauma in silico: Individual-specific mathematical models and virtual clinical populations

33. Inducible protein-10, a potential driver of neurally controlled interleukin-10 and morbidity in human blunt trauma

34. Racial Disparities and Sex-Based Outcomes Differences after Severe Injury

35. Central Role for MCP-1/CCL2 in Injury-Induced Inflammation Revealed by In Vitro, In Silico, and Clinical Studies

36. [Untitled]

37. Combined network and mathematical modeling suggests novel inflammation positive feedback circuit and role for IL-1α in experimental trauma/hemorrhage and hepatocyte hypoxia

38. Sepsis: Something old, something new, and a systems view

39. Induction and activation of macrophage transforming growth factor-β1 by extracellular nicotinamide adenine dinucleotide (NAD+) and its metabolite, cyclic ADP ribose: In vitro and in silico studies

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