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Results are presented on a method that uses laser-assisted reactivity in a constrained gas expansion to facilitate film growth. The 193 nm output of an ArF excimer laser is focused through a hole in a substrate into a gas pulse consisting of trimethylamine-alane (TMMA). As shown by mass spectral data, laser irradiation of the TMAA clearly results in significant gas-phase reactivity. While no discernible film growth takes place in the absence of laser radiation, the laser readily brings about film growth on a silicon substrate, even though the substrate is held at room temperature and no laser/substrate interactions occur. Atomic force microscopy is used to investigate the surface morphology present in different regions of the deposited film, and chemical composition in those regions is investigated with Auger electron spectroscopy.

A 2-dimensional waveguide model was successfully implemented to maximize the kink-free power from 980 nm pump lasers. Additionally, we show that high quality single crystal mirrors can be deposited epitaxially by MBE to eliminate COMD as a failure mode in these devices. Using state of the art manufacturing and packaging capabilities, the high power chip has been incorporated into the design of a high power wavelength stabilized 980 nm pump laser module. A typical curve showing fiber coupled power as a function of drive current for this family of laser modules is shown.

Summary Six patients with carcinomas of the cervix, uterus, or vagina that persisted or recurred after conventional surgical or radiation therapy were treated by continuous infusion of amethopterin into the hypogastric arteries. Citrovorum factor was administered intramuscularly to prevent generalized toxic manifestations. Hypogastric arteriograms in each case demonstrated evidence of flow of the concentrated drug directly to the tumor. For this therapy, normal or nearly normal renal function is an absolute prerequisite. The immediate effect of the hypogastric intra-arterial infusion on the tumor is profound necrosis, but any neoplastic cells not completely des troyed apparently begin to recover as soon as therapy is stopped.