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BACKGROUND Pilonidal sinus disease (PSD) is a benign disease caused by hair follicles in sacrococcygeal -region. Despite the use of different methods in treatment, there is no consensus reached for treatment modalities. In this study, we used a whole natal cleft excision and flap (WNCEF) method to remove the natal cleft in cases of extensive sacrococcygeal PSD. METHODS 243 patients with PSD were evaluated retrospectively. 47 patients with extensive sacrococcygeal PSD were included in the study. In these patients, the natal cleft was excised by using a whole natal cleft excision and flap -(WNCEF) method which was performed by an incision to include all the natal cleft (Kite incision) and the remaining defect was closed with a single fasciocutaneous flap by sliding along the midline intergluteal sulcus. RESULTS The mean age of patients was 25.7 years (17-43). The average hospital stay of patients was 2.7 days (2-4), mean operative time was 59 min (35-80), mean duration of drain removal was 2.7 days (2-4), mean postoperative follow-up was 16.4 months (3-24) and mean BMI was 26.9 (22-30). Flap edema occured in one patient (2.1%), seroma in three patients (6.3%) and surgical site infection requiring re-use of antibiotics were observed in two patients (4.2%). Flap -necrosis and failure did not occur in our patients. No recurrence was observed in any patient during an average follow-up of 16 months. CONCLUSIONS In sacrococcygeal PSD treatment, removal of the natal cleft with Kite incision by WNCEF method and shifting of the midline completely are thought to be an effective method that can reduce recurrence.

The intravitreal dexamethasone implant Ozurdex is indicated for the treatment of macular edema due to diabetes and branch retinal vein occlusion. While the most common ocular side effects are elevated intraocular pressure and cataract formation, rare complications related to the injection have been reported. We present a case with extramacular retinal hole after Ozurdex injection. © 2019 by Turkish Ophthalmological Association.

BioMed Central Ltd., Background: Orthodontic appliances can enhance plaque accumulation, and this can cause gingival inflammation. Halitosis of oral origin is associated with microbial metabolism on the tongue and in the saliva, dental plaque, and the amount of volatile sulfide-containing compounds. This study used a Halimeter to investigate fixed orthodontic therapy-associated increases in the oral malodor over a year. Methods: Thirteen orthodontic patients with Angle Class I malocclusions receiving fixed orthodontic therapy formed the study group, and 12 dental students without any dental treatment formed the control group. The Halimeter was used to examine oral malodor by detecting volatile sulfur compounds (VSC). Plaque index (PI), gingival index (GI), and probing pocket depth (PPD) were also measured in both groups. The subjects in the study group had one visit before the orthodontic treatment started and seven visits during orthodontic therapy (1, 3, 5, 7, 9, 11, and 13 months after bonding), while the subjects in the control group had three visits, once per subsequent month. Results: Oral malodor was significantly increased in the fixed orthodontic treatment group during treatment (p < .05). Increases were also observed in the PI, GI, and PPD measures (p < .05). The results of the control group were stable (p > .05). Conclusion: Oral malodor increased during fixed orthodontic treatments and reached a critical level 7 months later. © 2016 Sökücü et al., Birlik, M.; University of Bezmialem, Department of Orthodontics, Faculty of DentistryTurkey; email: muhammetbirlik@hotmail.com

Objective: To compare oral and nasal malodor in patients with and without cleft lip and palate (CLP) who had undergone orthodontic therapy. Setting: University of Cumhuriyet, Sivas, Turkey. Patients: The study included 33 patients. Group 1 was composed of 11 patients with CLP. Measurements were taken while they were undergoing treatment with orthodontic fixed appliances. Group 2 included 11 individuals without CLP; measurements were taken during fixed orthodontic therapy. Group 3 consisted of 11 individuals without CLP who did not receive orthodontic therapy. Intervention: Oral malodor, nasal malodor, Gingival Index, Plaque Index, and probing depths were evaluated in each group. Results: Plaque Index, Gingival Index, and probing depths showed no significant differences between groups 1 and 2. In contrast, oral malodor was significantly different between the two groups (p, Oztürk, F.; Cumhuriyet Üniversitesi, Dişhekimli?i Fakültesi, Ortodonti AD 58140 Sivas, Turkey; email: dtfirat@yahoo.com

Cumhuriyet University Faculty of Dentistry, Objective: Ozone accelerates wound healing and increases oxygen supply. The purpose of this study was to investigate the effect of ozone therapy combined with bone allograft on bone regeneration in rats with calvarial defects, histomorphometrically and histopathologically. Material-Method: Twenty four male Wistar rats were used in this study. A 5 mm diameter critical-size defects were created in all rats using a trephine bur. The rats were divided into 4 groups; empty defect (control) (n=6), ozone application into the control defect (control+ ozone) group (n=6), defect filled with allograft (Allograft) group (n=6), ozone combined with allograft application (allograft + ozone) group (n=6). Gaseous ozone administered at 80% density for 14 days. The animals were euthanized at 8-week. Total bone area was measured histomorphometrically and osteoclast and osteoblast number were analyzed histopathologically. Results: The osteoblast numbers increased in allograft + ozone group compared to the allograft group (p < 0.05). The control defects showed significantly less new bone formation than those of the allograft groups at 8 weeks (p < 0.05). Total bone area was significantly higher in allograft+ozone group than those of the allograft group (p < 0.05). Conclusions: Within the limitations of this study, we suggested that allograft+ozone combination increased osteoblast number and new bone area. However, further studies are needed to investigate the mechanisms of action of ozone on bone regeneration., Firat University Scientific Research Projects Management Unit: DIS-133, This study was supported by the Cumhuriyet University Unit of Scientific Research Projects with a project number DIS-133., Toker, H.; Department of Periodontology, Faculty of Dentistry, Cumhuriyet UniversityTurkey; email: htoker@gmail.com

Cumhuriyet University Faculty of Dentistry, Objectives: To evaluate the effect of periodontal therapy on clinical parameters as well as on myeloperoxidase (MPO) activity in the gingival crevicular fluid (GCF) of patients with type 2 diabetes mellitus and of systemically healthy individuals. Materials and Methods: 24 type 2 DM patients subjects, and 21 systemically healthy individuals, both groups with chronic periodontitis, and systemically and periodontally healthy individuals were enrolled. Periodontal clinical parameters, namely periodontal probing depth (PD), plaque index (PI), gingival index (GI), clinical attachment level (CAL), and gingival bleeding time index (GBI), as well as GCF MPO activity, were assessed before and 3 months after non-surgical periodontal therapy. GCF enzyme activity was spectrophotometrically analyzed. Possible correlation between clinical periodontal status and MPO activity was also evaluated. Results: Despite the relatively stable clinical measures and GCF MPO content in the periodontally-healthy subjects, the clinical periodontal status improved, as significant reductions were observed in all of the clinical parameters in periodontitis patients with and without diabetes (p, Özdemir, H.; Department of Periodontology, Faculty of Dentistry, Osmangazi University, Eskişehir, Turkey; email: hozdemir52@hotmail.com