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Intracluster light is thought to originate from stars that were ripped away from their parent galaxies by gravitational tides and galaxy interactions during the build up of the cluster. The stars from such interactions will accumulate over time, so semi-analytic models suggest that the abundance of intracluster stars is negligible in young proto-clusters at z ∼ 2 and grows to around a quarter of the stellar mass in the oldest, most mature clusters. In contrast to these theoretical expectations, we report on the detection of intracluster light within two proto-clusters at z = 2 using deep HST images. We use the colour of the intracluster light to estimate its mass-to-light ratio in annuli around the brightest cluster galaxies (BCG), up to a radius of 100 kpc. We find that 54 ± 5 per cent and 71 ± 3 per cent of the stellar mass in these regions is located more than 10 kpc away from the BCGs in the two proto-clusters. This low concentration is similar to BCGs in lower redshift clusters, and distinct from other massive proto-cluster galaxies. This suggests that intracluster stars are already present within the core 100 kpc of proto-clusters. We compare these observations to the Hydrangea hydrodynamical galaxy cluster simulations and find that intracluster stars are predicted to be a generic feature of group-sized haloes at z = 2. These intracluster stars will gradually move further away from the BCG as the proto-cluster assembles into a cluster. [ABSTRACT FROM AUTHOR]

Minor mergers have been proposed as the driving mechanism for the size growth of quiescent galaxies with decreasing redshift. The process whereby large star-forming galaxies quench and join the quiescent population at the large size end has also been suggested as an explanation for this size growth. Given the clear association of quenching with clusters, we explore this mechanism by studying the structural properties of 23 spectroscopically identified recently quenched (or 'post-starburst' (PSB)) cluster galaxies at z ∼ 1. Despite clear PSB spectral signatures implying rapid and violent quenching, 87 per cent of these galaxies have symmetric, undisturbed morphologies in the stellar continuum. Remarkably, they follow a mass–size relation lying midway between the star-forming and quiescent field relations, with sizes 0.1 dex smaller than z ∼ 1 star-forming galaxies at log(M */M⊙) = 10.5. This implies a rapid change in the light profile without directly effecting the stellar distribution, suggesting changes in the mass-to-light ratio gradients across the galaxy are responsible. We develop fading toy models to explore how star-forming galaxies move across the mass–size plane as their stellar populations fade to match those of the PSBs. 'Outside-in' fading has the potential to reproduce the contraction in size and increase in bulge-dominance observed between star-forming and PSB cluster galaxies. Since cluster PSBs lie on the large size end of the quiescent mass–size relation, and our previous work shows cluster galaxies are smaller than field galaxies, the sizes of quiescent galaxies must grow both from the quenching of star-forming galaxies and dry minor mergers. [ABSTRACT FROM AUTHOR]

Lymphangiomas or lymphatic malformations have a diverse presentation within the head and neck. This case report explores the use of cryotherapy as an alternative treatment strategy for such oral lesions with a review of literature. A 12 year‐old female presented with an uncommon lower lip lymphangioma. Due to aesthetic concerns she was treated with one session of cryotherapy as a relatively potential novel treatment. Clinically the patient had some reduction in size of the lesion with no significant post‐operative complications. The use of cryotherapy could be considered as a treatment option in small oral cavity lymphangiomas within the aesthetic zone but may not always provide complete resolution or success. [ABSTRACT FROM AUTHOR]

Minor mergers are thought to be responsible for the size growth of quiescent field galaxies with decreasing redshift. We test this hypothesis using the cluster environment as a laboratory. Satellite galaxies in clusters move at high velocities, making mergers between them rare. The stellar mass–size relation in 10 clusters and in the field is measured and compared at |$z$| ∼ 1. Our cluster sample contains 344 spectroscopically confirmed cluster members with Gemini/Gemini Multi-Object Spectrographs and 182 confirmed with Hubble Space Telescope /Wide Field Camera 3 G141 grism spectroscopy. On average, quiescent and star-forming cluster galaxies are smaller than their field counterparts by (0.08 ± 0.04) and (0.07 ± 0.01) dex, respectively. These size offsets are consistent with the average sizes of quiescent and star-forming field galaxies between 1.2 ≤ |$z$| ≤ 1.5, implying the cluster environment has inhibited size growth between this period and |$z$| ∼ 1. The negligible differences measured between the |$z$| ∼ 0 field and cluster quiescent mass–size relations in other works imply that the average size of quiescent cluster galaxies must rise with decreasing redshift. Using a toy model, we show that the disappearance of the compact cluster galaxies might be explained if, on average, |${\sim }40{{\ \rm per\ cent}}$| of them merge with their brightest cluster galaxies (BCGs) and |${\sim }60{{\ \rm per\ cent}}$| are tidally destroyed into the intracluster light (ICL) between 0 ≤ |$z$| ≤ 1. This is in agreement with the observed stellar mass growth of BCGs between 0 ≤ |$z$| ≤ 1 and the observed ICL stellar mass fraction at |$z$| ∼ 0. Our results support minor mergers as the cause for the size growth in quiescent field galaxies, with cluster-specific processes responsible for the similarity between the field and cluster quiescent mass–size relations at low redshift. [ABSTRACT FROM AUTHOR]

Abstract: Diffuse sclerosing osteomyelitis (DSO) is a rare inflammatory disease of bone. The underlying cause is poorly understood and traditional management techniques have struggled to provide patients with adequate relief. A number of reports in the literature have described the use of bisphosphonates to manage DSO; however, these have predominantly been associated with the intravenous formulation. Understandably, there is an element of angst due to its underlying and well‐recognised risk of osteonecrosis of the jaw. In this article, we review the literature for the management of DSO including the novel approach of using bisphosphonates. As an example, we present a case of a 56‐year‐old female with DSO where conventional therapies failed to manage her symptoms; however, treatment with oral alendronic acid led to complete and sustained resolution. This case in conjunction with additional reported cases provides further evidence of the value of bisphosphonates in DSO and highlights that the use of oral bisphosphonates can be successful and could be a viable consideration prior to the use of intravenous versions. [ABSTRACT FROM AUTHOR]

Benign parotid tumours have historically often been managed surgically by superficial parotidectomy. While this approach usually gives a generous cuff of surrounding normal parotid tissue to increase tumour margins, it requires a much larger incision than the increasingly used extracapsular dissection ( ECD) technique. Furthermore, superficial parotidectomy can result in marked facial hollowing, Frey syndrome and an increased risk of both temporary and permanent facial nerve weakness. ECD has been popularised as a safe alternative to parotidectomy primarily for the removal of mobile, benign parotid tumours with safe outcomes and reduced risk to the facial nerve. In this article, we review the growing body of evidence for ECD and include our own experience confirming the move away from superficial parotidectomy in contemporary practice for the treatment of benign parotid tumours. [ABSTRACT FROM AUTHOR]

Thirty-three cases of infective endocarditis presenting during a 6.5 year period to a district general hospital were analysed retrospectively. The annual incidence was 22 cases per million population. Twenty-two cases had pre-existing cardiac disease, mainly valvular disease-usually rheumatic (nine cases) and prosthetic valves (10 cases). Recognizable precipitants such as recent surgery were uncommon. Two cases presented after deliberate drug overdose possibly due to depression exacerbated by systemic disease. Symptoms were usually non-specific. All but two cases had murmurs and most were pyrexial. Splinter haemorrhages and clubbing were seen in about 20% of cases. Viridans-type streptococci were the commonest infecting organisms (14 cases). Staphylococcal infection (six cases) was confined to intravenous drug abusers and patients with prosthetic valves. Five cases were culture negative. Cardiac failure was present in 13 cases at presentation and developed in seven others during treatment. Acute valve replacement was necessary in eight cases, and late replacement in three. Renal impairment (plasma urea > 8 mmol/l and/or plasma creatinine > 120 mumol/l) occurred in 19 cases during the course of their illness. Embolic phenomena occurred in 12 patients and mostly involved the central nervous system. In the 8 fatal cases, the cause of death was cardiac failure in six, cerebrovascular accident in one, and myocardial infarction in one. Four of the six patients who subsequently died of cardiac failure had been referred for surgery. Both those who were not referred had coexisting medical problems. Factors associated with increased mortality were age, male sex, cardiac failure (P < 0.01), renal impairment (P < 0.05), and embolic phenomena (P < 0.01).(ABSTRACT TRUNCATED AT 250 WORDS) [ABSTRACT FROM AUTHOR]

We describe a previously unreported anatomical variant (found during a routine neck dissection) in which the lower third of the sternocleidomastoid was innervated by the C1 branch of the ansa cervicalis (descendens hypoglossi). [ABSTRACT FROM AUTHOR]