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322 results on '"Van Eldik, Linda J."'

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1. LATE-NC risk alleles (in TMEM106B, GRN, and ABCC9 genes) among persons with African ancestry.

2. Aβ efflux impairment and inflammation linked to cerebrovascular accumulation of amyloid-forming amylin secreted from pancreas

4. Cross species systems biology discovers glial DDR2, STOM, and KANK2 as therapeutic targets in progressive supranuclear palsy

6. Brain arteriolosclerosis

7. Analysis of shared heritability in common disorders of the brain

8. Analysis of shared heritability in common disorders of the brain.

9. ABCC9 gene polymorphism is associated with hippocampal sclerosis of aging pathology

10. Human cerebrospinal fluid contains diverse lipoprotein subspecies enriched in proteins implicated in central nervous system health

16. New directions for Alzheimer's disease research from the Jackson Laboratory Center for Alzheimer's and Dementia Research 2022 workshop.

23. A blunted TH17 cytokine signature in women with mild cognitive impairment: insights from inflammatory profiling of a community-based cohort of older adults

24. Neurodegenerative pathologies associated with behavioral and psychological symptoms of dementia in a community-based autopsy cohort.

40. The p38α mitogen-activated protein kinase as a central nervous system drug discovery target

42. Cyclopentenone prostaglandins suppress activation of microglia: down-regulation of inducible nitric-oxide synthase by 15-deoxy-delta(super 12,14)-prostaglandin J(sub 2)

43. Genetic and lifestyle risk factors for MRI-defined brain infarcts in a population-based setting

45. Amyloid beta-peptide stimulates nitric oxide production in astrocytes through an NF kappaB-dependent mechanism

47. Genetic meta-analysis of diagnosed Alzheimer's disease identifies new risk loci and implicates A beta, tau, immunity and lipid processing

48. In aged human brains without detected neuritic amyloid plaques, the severity of NFT pathology, and APOE genotype, are each associated with non-neuritic Aβ plaques

50. Microglial p38α MAPK is critical for LPS-induced neuron degeneration, through a mechanism involving TNFα

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