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3 results on '"Skeidsvoll, Håvard"'

1. Repeated magnetic resonance imaging and cerebral performance after cardiac arrest : a pilot study

Author

Heradstveit, Bård E, Larsson, Elna-Marie, Skeidsvoll, Håvard, Hammersborg, Stig-Morten, Wentzel-Larsen, Tore, Guttormsen, Anne Berit, Heltne, Jon-Kenneth, Heradstveit, Bård E, Larsson, Elna-Marie, Skeidsvoll, Håvard, Hammersborg, Stig-Morten, Wentzel-Larsen, Tore, Guttormsen, Anne Berit, and Heltne, Jon-Kenneth

Abstract

AIM OF THE STUDY: Prognostication may be difficult in comatose cardiac arrest survivors. Magnetic resonance imaging (MRI) is potentially useful in the prediction of neurological outcome, and it may detect acute ischemia at an early stage. In a pilot setting we determined the prevalence and development of cerebral ischemia using serial MRI examinations and neurological assessment. METHODS: Ten witnessed out-of-hospital cardiac arrest patients were included. MRI was carried out approximately 2h after admission to the hospital, repeated after 24h of therapeutic hypothermia and 96 h after the arrest. The images were assessed for development of acute ischemic lesions. Neurophysiological and cognitive tests as well as a self-reported quality-of-life questionnaire, Short Form-36 (SF-36), were administered minimum 12 months after discharge. RESULTS: None of the patients had acute cerebral ischemia on MRI at admission. Three patients developed ischemic lesions after therapeutic hypothermia. There was a change in the apparent diffusion coefficient, which significantly correlated with the temperature (p < 0.001). The neurophysiological tests appeared normal. The patients scored significantly better on SF 36 than the controls as regards both bodily pain (p = 0.023) and mental health (p = 0.016). CONCLUSIONS: MRI performed in an early phase after cardiac arrest has limitations, as MRI performed after 24 and 96 h revealed ischemic lesions not detectable on admission. ADC was related to the core temperature, and not to the volume distributed intravenously. Follow-up neurophysiologic tests and self-reported quality of life were good.

Published
2011
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2. [Neurologic decompression sickness in sports divers].

Author

Bjørnstad J, Nyland H, Skeidsvoll H, Aanderud L, and Eidsvik S

Subjects
Adult, Cognition Disorders diagnosis, Cognition Disorders etiology, Decompression Sickness complications, Decompression Sickness physiopathology, Decompression Sickness therapy, Follow-Up Studies, Humans, Hyperbaric Oxygenation, Nervous System Diseases diagnosis, Retrospective Studies, Decompression Sickness etiology, Diving injuries, Nervous System Diseases etiology
Abstract

Background: Sports diving is a popular recreational activity. Sports divers presenting with acute decompression sickness may exhibit residual neurologic and neuropsychological symptoms during follow-up, in spite of appropriate treatment., Material and Methods: A retrospective review of medical records was carried out for sports divers admitted to the department of neurology at Haukeland University Hospital during 1997., Results: 11 out of 20 divers experienced residual neurological symptoms after treatment. Five responded poorly to treatment, with 50% or more residual clinical score. These patients tended to be older, had performed deeper dives, and more repetitive diving. Seven divers had increased slow wave activity in EEG on initial recording, in two the EEG changes persisted after treatment., Interpretation: In this small series of sports divers with decompression sickness and arterial gas embolism, most neurologic symptoms responded to hyperbaric treatment. However, more than one half of the divers had residual neurological symptoms on discharge. Sensory loss and asymmetrical reflexes were the most common residual findings.

Published
2002

3. [Spasticity treated with selective posterior rhizotomy].

Author

Grønning M, Svendsen F, Skeidsvoll H, Jones KØ, and Nilsen PT

Subjects
Adult, Female, Humans, Treatment Outcome, Multiple Sclerosis surgery, Paraplegia surgery, Rhizotomy methods
Abstract

Background: Spasticity is often seen in patients with central nervous lesions. Some patients with severe spasticity are not optimally treated with physiotherapy and medication., Material and Methods: We present a case history of a 41-year-old woman with multiple sclerosis and severe painful spasticity in her lower limbs. Her spasticity did not respond to treatment with physiotherapy, spasmolytic medication, botulinum toxin A, intrathecal baclofen or epidural spinal cord stimulation., Result: The patient was treated with selective posterior rhizotomy S1-L1. Section of 60% of the rootlets on the right side and 40% on left the side resulted in a good outcome with less spasticity and pain. Finally her contractures were treated with tenotomy and myotomy, also with good functional result., Interpretation: Patients suffering from severe painful spasticity and who do not respond to physiotherapy in combination with other spasmolytic medication should be considered for surgical treatment. In some patients posterior rhizotomy is the treatment of choice.

Published
2002

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