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1. A case report of an asymptomatic necrotic Meckel's diverticulum in an inguinal hernia during elective surgery in a resource limited setting: Littre's hernia

Author

Odongo, Charles Newton, Godier-Furnemont, Amandine, Moro, Joshua, and Oyania, Felix

Subjects
Biomedical and Clinical Sciences, Clinical Sciences, Digestive Diseases, Littre's hernia, Meckel's diverticulum, Diverticulectomy, Segmental resection, Case report, Clinical sciences
Abstract

Introduction and importanceAlthough the common complications of Meckel's diverticulum (MD) are well known, that these congenital intestinal outpouchings may become involved as the content of abdominal hernia sacs is not well appreciated. MD is the most prevalent congenital abnormality of the gastrointestinal tract, but involvement in a hernia, known as Littre's hernia (LH), accounts for less than 1 % of MD cases. Incarcerated LH has been reported sporadically in the literature, with MD found in the sacs of paraumbilical, femoral, inguinal, and incisional hernias.Presentation of caseWe report a LH in a 3-year-old male child who was scheduled for elective herniotomy for a reducible left inguinal hernia. Intraoperatively we found the hernia sac contained a necrotic and perforated MD with viable associated bowel loop. The patient was successfully managed by diverticulectomy and primary repair through a trans-inguinal incision and herniotomy was performed.Clinical discussionLH is a rare presentation of MD, and preoperative diagnosis of LH is challenging. Even in the case of a strangulated MD, a patient may not present with the typical signs and symptoms associated with compromised viscous. Once identified, repair of Littre hernia consists of resection of the diverticulum, or segmental bowel resection if necessary, and herniotomy.ConclusionThe finding of a perforated MD during elective hernia repair emphasizes the importance of awareness of unusual variants of inguinal hernia, and the necessity of identifying a MD given the risk of sequelae in the case of necrosis or perforation, if not repaired.

Published
2023

11. Etiology, Clinical Presentations, and Short-Term Treatment Outcomes of Extrahepatic Obstructive Jaundice in South-Western Uganda

Author

Odongo,Charles Newton, Dreque,Carlos Cabrera, Mutiibwa,David, Bongomin,Felix, Oyania,Felix, Sikhondze,Mvuyo Maqhawe, Acan,Moses, Atwine,Raymond, Kirya,Fred, Situma,Martin, Odongo,Charles Newton, Dreque,Carlos Cabrera, Mutiibwa,David, Bongomin,Felix, Oyania,Felix, Sikhondze,Mvuyo Maqhawe, Acan,Moses, Atwine,Raymond, Kirya,Fred, and Situma,Martin

Abstract

Charles Newton Odongo,1 Carlos Cabrera Dreque,2 David Mutiibwa,2 Felix Bongomin,3 Felix Oyania,4 Mvuyo Maqhawe Sikhondze,2 Moses Acan,2 Raymond Atwine,2 Fred Kirya,1 Martin Situma2 1Department of Anatomy, Faculty of Medicine, Soroti University, Soroti, Uganda; 2Faculty of Medicine, Mbarara University of Science and Technology, Mbarara, Uganda; 3Department of Microbiology and Immunology, and Internal Medicine, Faculty of Medicine, Gulu University, Gulu, Uganda; 4Department of Surgery, Faculty of Medicine, Kabale University, Kabale, UgandaCorrespondence: Charles Newton Odongo, Tel +256774849032, Email cnodongo@gmail.comBackground: The diagnosis of extrahepatic obstructive jaundice (EHOJ) remains a challenge and is often made late in low-resource settings. Systematic data are limited on the etiology and prognosis of patients with obstructive jaundice in Uganda. The objective of this study was to determine the etiology, clinical presentations, and short-term treatment outcomes of patients managed for EHOJ at Mbarara Regional Referral Hospital (MRRH) in south-western Uganda.Methods: Between September 2019 and May 2020, we prospectively enrolled a cohort of patients who presented with EHOJ at MRRH. A pretested, semi-structured data collection tool was used to abstract data from both the study participants and their files.Results: A total of 72 patients, 42 (58.3%) of whom were male with a median age of 56 (range of 2 months to 95 years) were studied. Forty-two (58.3%) participants had malignancies: Pancreatic head tumors 20 (27.8%), cholangiocarcinoma 13 (18.1%), duodenal cancers 5 (6.94%), and gall bladder cancer 4 (5.6%). The remaining 30 (41.7%) participants had benign etiologies: choledocholithiasis 10 (13.9%), biliary atresia 7 (9.7%), pancreatic pseudo cyst 6 (8.3%), Mirizzi syndrome 5 (6.9%) and 1 (1.4%) each of chronic pancreatitis and choledochal cyst. Sixty-seven (93.1%) patients presented with right upper quadrant tenderness, 65 (90.3%) abdominal pain and 55 (7

Published
2022

12. Adenomatous Hyperplasia of the Gallbladder in the Setting of Mirizzi Syndrome, Mimicking Adenocarcinoma of the Gallbladder

Author

Odongo,Charles Newton, Dreque,Carlos Cabrera, Bongomin,Felix, Oyania,Felix, Situma,Martin, and Atwine,Raymond

Subjects
International Medical Case Reports Journal
Abstract

Charles Newton Odongo,1 Carlos Cabrera Dreque,1 Felix Bongomin,2 Felix Oyania,3 Martin Situma,1 Raymond Atwine4 1Department of Surgery, Faculty of Medicine, Mbarara University of Science and Technology, Mbarara, Uganda; 2Departments of Medical Microbiology & Immunology, and Internal Medicine, Faculty of Medicine, Gulu University, Gulu, Uganda; 3Department of Surgery, Faculty of Medicine, Kabale University, Kabale, Uganda; 4Department of Pathology, Faculty of Medicine, Mbarara University of Science and Technology, Mbarara, UgandaCorrespondence: Charles Newton OdongoDepartment of Surgery, Faculty of Medicine, Mbarara University of Science and Technology, Mbarara, UgandaEmail cnodongo@gmail.comBackground: Adenomatous hyperplasia (AH) is an uncommon benign lesion of the gallbladder (GB), predominantly seen in men. AH is commonly confused with malignant GB neoplasms in the setting of chronic cholecystitis and gallstones. There is a scarcity of published literature on AH, suggesting its rarity and the need for this case report.Case Presentation: A 24-year-old woman from Western Uganda presented with signs and symptoms consistent with extrahepatic biliary obstruction. Trans-abdominal ultrasound scan revealed cholecystomegaly (13.9 cm by 4.29 cm), thickened GB wall at 5.2 mm, with a poorly defined hypoechoic polypoid solid mass involving the fundal body of the GB. Explorative laparotomy with cholecystectomy and lymph node sampling was performed. Histopathological examination was consistent with adenomatous hyperplasia of the GB. The symptoms and laboratory values improved on follow-up in the clinic after laparotomy.Conclusion: Adenomatous hyperplasia may be misdiagnosed as a malignant GB neoplasm, especially in the setting of chronic cholecystitis and gallstones. If a correct histopathological diagnosis is made, no further diagnostic work-up is necessary following surgical interventions.Keywords: adenomatous hyperplasia, gallbladder, Mirizzi syndrome

Published
2021

19. Injury and violence in the context of sustainable development: The first Bethune Round Table in Africa, Bethune Round Table 2024, Conference on Global Surgery, May 16-18, 2024, Addis Ababa, Ethiopia.

Author

Lett R, Bartolomeos K, Lett R, Laeke T, Tirsit A, Kassahun A, Boniface R, Munthali V, Razek T, Deckelbaum D, Bracco D, Presser E, Belay E, Kifle F, Weiser TG, Iverson K, Takoutsing BD, Dalle DU, Mbangtang CB, Nyalundja AD, Macaraeg J, Dzirasa I, Kanmounye US, Dovlo D, Koram K, Nyarko E, Jumbam DT, Shimber ET, Jaraczewski T, Sgro M, Basmayor AM, Ergete A, Iverson K, Schroeder M, Dodgion C, Gyedu A, Nakua E, Donkor P, Mock C, Awedew A, Laeke T, Yibeltal M, Ayele M, Melkamu H, Bekele S, Hailemariam B, Shiferaw E, Shiferaw Y, Yirdaw W, Sawhney R, Patel S, Basak D, Veetil DK, Roy N, Wärnberg MG, Rath S, Abdullahi MAS, Mbaya K, Kakasanda A, Danjuma S, Olasoji H, Ameh E, Bedada A, Motsumi MJ, Hamda SG, Ibro S, Amdisa D, Tilahun G, Abeza M, Laeke T, Boroditsky M, Hill M, Hilzenrat R, Livergant R, Adams J, Binda C, Chhor A, Hsiao H, Haji F, Chin E, Oyania F, Stephens CQ, Ullrich S, Kotagal M, Bajunirwe F, Ozgediz DE, Poenaru D, Kravarioti D, Wong LY, Teklemariam TL, Tirsit A, Liyew T, Ferguson M, Plackett T, Henry JC, Dodgion C, Abeza MA, Minas SM, Bouchard M, Tennakoon D, Sawhney R, Burra R, Mathew F, Jones A, Virk S, Patel S, Vaghaiwalla T, Hudspeth J, Rabin T, Rowthorn V, Price RR, Raykar N, Eamer G, Mutiso S, Kisaka Y, Gathecha G, Lett R, Onu C, Ameh E, Igoche M, Anyanwu P, Onuh E, Ojeamen O, Yawe ET, Abubakar A, Ashoms Y, Suleiman H, Musa N, Kyengera DK, Abebe N, Gardener R, Berhanu D, Abebe NS, Zeleke HT, Sawhney R, Proulx KR, Gerk A, Guadagno E, Poenaru D, Kundu S, Laor B, Sawhney R, Wurdeman T, Botelho F, Gerk A, Guadagno E, Poenaru D, Ayele M, Kassahun A, Laeke T, Yibeltal M, Hailu B, Fikru E, Ibro SA, Workineh A, Balcha F, Abamecha F, Shemsi S, Alruwaili AS, Rodriguez G, Jose A, Ebied S, Girma S, Abiy A, Assen HE, Tesfaye K, Demeke K, Yiheyis A, Jemal K, Yilkal D, Amsalu A, Derseh L, W/Gerima Y, Belayneh T, Tiruneh M, Bitew A, Yitayih S, Awoke T, Worku C, Mohammed A, Alemu M, Yesuf M, Mamo F, Shitu K, Liyew B, Gucho A, Tilahun G, Love T, Chew A, Kasagga B, Takoutsing B, Ekwaro O, Elobu E, Mengistu DD, Zhuang A, Shiferew B, Mengistu G, Zewdie A, Tadelle N, Gebreyesus A, Presser E, Iverson K, Dodgion C, Weiser TG, Koch R, Starr N, Lau D, Zivkovic I, Joharifard S, Joos E, Garraway N, Vituci F, O'Flynn E, Péric I, Simon L, Ibbotson G, Seyoum T, Azazh A, Beza L, Onah I, Chukwuma C, Berhanu D, Shenoi J, Sears N, Bedore Y, Caplan R, and Shale WT

Published
2024
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