Search

Your search keyword '"Giménez, M. I."' showing total 2 results
Start Over Author "Giménez, M. I." Search Limiters Full Text
2 results on '"Giménez, M. I."'

1. Classic, fuzzy and predictive dtc strategies for the PMSM using the bacterial foraging algorithmas an online parameter estimator

Author

Noriega, G., Restrepo, J., Bueno, A., Jose Aller, Giménez, M. I., and Guzmán, V.

Subjects
máquina sincrónica de imán permanente, sistema de inferencia difusa, lcsh:T, lcsh:TA1-2040, forraje bacterial, control directo de par, estimación paramétrica, lcsh:Engineering (General). Civil engineering (General), control predictivo, lcsh:Technology
Abstract

Este trabajo presenta una comparación entre cuatro técnicas de control directo de par (DTC) aplicadas al accionamiento de la máquina sincrónica deimán permanente: Clásico, Modificado con un sistema de inferencia difusa, Predictivo y Predictivo con inferencia difusa. La estimación de parámetros para las estrategias predictivas es realizado utilizando un algoritmo de búsqueda poblacional basado en Forraje Bacterial, el cuál es capaz de calcular en línea los parámetros de la máquina sincrónica de imán permanente. Los resultados experimentales del par eléctrico y de los enlaces de flujo del estator muestran mejoras significativas para las estrategias predictivas que utilizan algoritmos de estimación paramétrica mediante Forraje Bacterial cuando se comparan con técnicas no predictivas.

Published
2012

2. Erythrocyte sodium-lithium countertransport in non-modulating offspring and essential hypertensive individuals: response to enalapril.

Author

Sánchez RA, Giménez MI, Migliorini M, Giannone C, Ramírez AJ, and Weder AB

Subjects
Adult, Angiotensin-Converting Enzyme Inhibitors administration & dosage, Antihypertensive Agents administration & dosage, Biological Transport, Chlorides metabolism, Data Interpretation, Statistical, Enalapril administration & dosage, Female, Humans, Hypertension metabolism, Lithium analysis, Male, Middle Aged, Sodium analysis, Sodium metabolism, Sodium, Dietary adverse effects, Sodium-Potassium-Exchanging ATPase metabolism, Spectrophotometry, Atomic, Time Factors, Angiotensin-Converting Enzyme Inhibitors therapeutic use, Antihypertensive Agents therapeutic use, Enalapril therapeutic use, Erythrocytes metabolism, Hypertension drug therapy, Hypertension genetics, Lithium blood, Sodium blood
Abstract

Non-modulators are a subset of essential hypertensive individuals in whom renal hemodynamic and adrenal aldosterone responses to angiotensin II fail to modulate appropriately during high dietary salt intake. The main aim of this study was to investigate the familial aggregation of non-modulation and several erythrocyte Na+ transport systems in normotensive and hypertensive individuals as well as offspring of hypertensive parents. An additional aim was to evaluate the effect of treatment with enalapril on erythrocyte Na+ transport. We studied 15 normotensive subjects (6 males, 27+/-6 years), 14 untreated modulating essential hypertensive subjects (7 males, 38+/-7 years), 12 untreated non-modulating essential hypertensive subjects (7 males, 38+/-6 years), 14 modulating offspring of hypertensive parents (8 males, 25+/-6 years), and 14 non-modulating offspring of hypertensive parents (8 males, 26+/-4 years). Blood pressure was recorded with an oscillometric device and renal plasma flow and glomerular filtration rate by clearances of para-aminohippurate and inulin, respectively. Non-modulating subjects were identified as individuals who failed to increase effective renal plasma flow by 30% and decrease filtration fraction by at least 30% 10 days after changing from a low (20 mmol/d) to a high (250 mmol/d) sodium intake. Erythrocyte Na+ transport was characterized by measurements of the Na+-K+ pump, Na+-Li+ countertransport, Na+-K+-Cl- cotransport, passive Na+ permeability, and Na+ content. After the initial studies, hypertensive individuals were treated with enalapril (20 mg/d P.O.) for 6 months, after which erythrocyte Na+ transport measurements were again made. The main findings were that Na+-Li+ countertransport is increased in non-modulating hypertensive subjects and non-modulating offspring of hypertensive parents, that the increase in blood pressure in response to high salt intake is greater in non-modulating than modulating hypertensive subjects, and that enalapril decreases Na+-Li+ countertransport activity to normal in non-modulating hypertensive subjects. These findings provide support for a possible genetic role in the development of salt sensitivity and suggest that Na+-Li+ countertransport and non-modulation are related phenotypes.

Published
1997
Full Text
View/download PDF

Catalog

Books, media, physical & digital resources
See catalog results