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1. Distinct pattern of genomic breakpoints in CML and BCR::ABL1-positive ALL: analysis of 971 patients

2. Erratum to: Genomic breakpoint-specific monitoring of measurable residual disease in pediatric non-standard risk acute myeloid leukemia

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3. P315: TP53 ALTERATIONS AND MRD REFINE PROGNOSIS OF ADULT KMT2A-REARRANGED B-ALL

4. P337: IKZF1 DELETIONS IN B-ALL: FROM ITS GENETIC BASIS TO DIAGNOSTIC ENHANCEMENT

5. Genomic breakpoint-specific monitoring of measurable residual disease in pediatric non-standard-risk acute myeloid leukemia

6. The immune checkpoint ICOSLG is a relapse-predicting biomarker and therapeutic target in infant t(4;11) acute lymphoblastic leukemia

7. Novel Diagnostic and Therapeutic Options for KMT2A-Rearranged Acute Leukemias

8. IKZF1 Deletions with COBL Breakpoints Are Not Driven by RAG-Mediated Recombination Events in Acute Lymphoblastic Leukemia

9. Evolution of AF6-RAS association and its implications in mixed-lineage leukemia

10. Adversarial Multi-Task Deep Learning for Noise-Robust Voice Activity Detection with Low Algorithmic Delay

15. Evaluation of gene expression signatures predictive of cytogenetic and molecular subtypes of pediatric acute myeloid leukemia

17. Epigenetic regulator genes direct lineage switching in MLL/AF4 leukemia

18. The EGR3 regulome of infant KMT2A-r acute lymphoblastic leukemia identifies differential expression of B-lineage genes predictive for outcome

19. Data from Targeted Next-Generation Sequencing for Detecting MLL Gene Fusions in Leukemia

20. Figure S1 from Targeted Next-Generation Sequencing for Detecting MLL Gene Fusions in Leukemia

21. Table S1 from Targeted Next-Generation Sequencing for Detecting MLL Gene Fusions in Leukemia

22. Data from Effective Targeting of the P53–MDM2 Axis in Preclinical Models of Infant MLL-Rearranged Acute Lymphoblastic Leukemia

23. Supplementary Methods, Figures S1-S8, Tables S1-S7 from Effective Targeting of the P53–MDM2 Axis in Preclinical Models of Infant MLL-Rearranged Acute Lymphoblastic Leukemia

24. The recombinome of IKZF1 deletions in B-ALL

25. KMT2A-CBL rearrangements in acute leukemias: clinical characteristics and genetic breakpoints

26. Clinical Implications of Minimal Residual Disease Detection in Infants With KMT2A-Rearranged Acute Lymphoblastic Leukemia Treated on the Interfant-06 Protocol

27. Novel Diagnostic and Therapeutic Options for

28. Human MLL/KMT2A gene exhibits a second breakpoint cluster region for recurrent MLL–USP2 fusions

29. A new complex rearrangement in infant ALL: t(X;11;17)(p11.2;q23;q12)

30. Prenatal origin of <scp>KRAS</scp> mutation in a child with an acute myelomonocytic leukaemia bearing the KMT2A / MLL‐AFDN / MLLT4 / AF6 fusion transcript

31. Targeted Next-Generation Sequencing for Detecting MLL Gene Fusions in Leukemia

32. Implication of ICOSLG on Relapse in Infant T(4;11) Acute Lymphoblastic Leukemia

33. Targeted Next Generation Sequencing Reveals a Third Breakpoint Cluster Region and New Partner Genes in the KMT2A Recombinome

34. t(11;16)(q23;q24) KMT2A/USP10

35. IKZF1 Deletions with COBL Breakpoints Are Not Driven by RAG-Mediated Recombination Events in Acute Lymphoblastic Leukemia

36. Effective Targeting of the P53–MDM2 Axis in Preclinical Models of Infant MLL-Rearranged Acute Lymphoblastic Leukemia

37. Subclonality and prenatal origin ofRASmutations inKMT2A (MLL)-rearranged infant acute lymphoblastic leukaemia

38. Targeted Next-Generation Sequencing for Detecting

39. Evolution of AF6-RAS association and its implications in mixed-lineage leukemia

40. t(2;11)(q33;q23) KMT2A/ABI2

41. Functional analysis of the two reciprocal fusion genes MLL-NEBL and NEBL-MLL reveal their oncogenic potential

42. The distribution ofMLLbreakpoints correlates with outcome in infant acute leukaemia

43. COBL is a novel hotspot for IKZF1 deletions in childhood acute lymphoblastic leukemia

44. Involvement of theMLLgene in adult T-lymphoblastic leukemia

45. TOP3A, a new partner gene fused to MLL in an adult patient with de novo acute myeloid leukaemia

46. ETV6/RUNX1-positive relapses evolve from an ancestral clone and frequently acquire deletions of genes implicated in glucocorticoid signaling

47. Contents Vol. 126, 2011

48. MLL-USP2: An Underestimated New Entity of MLL-Rearranged Leukemia Identified By NGS Analysis

49. Quantitative Analysis of MLL Fusion Transcripts By Droplet Digital PCR to Monitor Minimal Residual Disease in MLL-Rearranged Acute Myeloid Leukemia

50. Nebulette is the second member of the nebulin family fused to the MLL gene in infant leukemia