226 results on '"Chin, Michael T"'
Search Results
2. Altered intercellular communication and extracellular matrix signaling as a potential disease mechanism in human hypertrophic cardiomyopathy
3. A novel αB-crystallin R123W variant drives hypertrophic cardiomyopathy by promoting maladaptive calcium-dependent signal transduction.
4. A Barth Syndrome Patient-Derived D75H Point Mutation in TAFAZZIN Drives Progressive Cardiomyopathy in Mice.
5. Role of the Alpha-B-Crystallin Protein in Cardiomyopathic Disease
6. Abstract 13383: Is Genotype Negative Hypertrophic Cardiomyopathy Really Benign? The Increased Risk of Sudden Death and Advanced Heart Failure in the Absence of Pathogenic Sarcomere Mutations
7. Relationship Between Genotype Status and Clinical Outcome in Hypertrophic Cardiomyopathy.
8. Plasma metabolomic profiling of hypertrophic cardiomyopathy patients before and after surgical myectomy suggests postoperative improvement in metabolic function
9. A method for cryopreservation and single nucleus RNA-sequencing of normal adult human interventricular septum heart tissue reveals cellular diversity and function
10. Differential effects of various genetic mouse models of the mechanistic target of rapamycin complex I inhibition on heart failure
11. Editorial: Molecular pathogenesis and novel treatments for inherited cardiomyopathies
12. Spatial Transcriptomic Analysis of Focal and Normal Areas of Myocyte Disarray in Human Hypertrophic Cardiomyopathy
13. Abstract EC.09: Single Nuclei RNA Sequencing Analysis Of Mybpc-3- Associated HCM To Identify Common Pathological Mechanisms In Human, Feline, And Murine Hearts
14. Abstract P1016: Single Cell Transcriptomic Analysis Of Tafazzin Knockout Mice For Pathway And Therapeutic Discovery In Barth Syndrome
15. Abstract GS.08: Cardiolipin Is A Novel Target Of Therapy To Mitigate Myocardial Injury Due To Veno-arterial Extracorporeal Membrane Oxygenation (va-ecmo)
16. Abstract P2171: Lentivirus TAZ Gene Therapy Restores Metabolic Functions In Taz Ko Mice Model Of Barth Syndrome
17. Abstract P2046: The Role Of A Novel Alpha-crystallin B Chain Variant In Hypertrophic Cardiomyopathy
18. Single Cell Transcriptomic Analysis in a Mouse Model of Barth Syndrome Reveals Cell-Specific Alterations in Gene Expression and Intercellular Communication
19. A novel αB-crystallin R123W variant drives hypertrophic cardiomyopathy by promoting maladaptive calcium-dependent signal transduction
20. Abstract 17402: Single Nuclei RNA-sequencing of Human Hypertrophic Cardiomyopathy Myectomy Samples Reveals Common Novel Mechanisms of Pathogenesis and Potential Therapeutic Targets Regardless of Genotype
21. Ventricular Septal Defect and Cardiomyopathy in Mice Lacking the Transcription Factor CHF1/Hey2
22. Multi-Omics Profiling of Hypertrophic Cardiomyopathy Reveals Altered Mechanisms in Mitochondrial Dynamics and Excitation–Contraction Coupling
23. In utero exposure to diesel exhaust is associated with alterations in neonatal cardiomyocyte transcription, DNA methylation and metabolic perturbation
24. Modeling heart failure with preserved ejection fraction in rodents: Where do we stand?
25. Abstract P2011: The Role Of A Novel Alpha-crystallin B Chain Variant In Hypertrophic Cardiomyopathy
26. Abstract 20732: Identification of a Novel Mitochondrial Targeting Sequence in Tafazzin, the Mitochondrial Enzyme Associated With the Cardiomyopathic Disorder Barth Syndrome
27. Common and Distinctive Intercellular Communication Patterns in Human Obstructive and Nonobstructive Hypertrophic Cardiomyopathy
28. Amino acid primed mTOR activity is essential for heart regeneration
29. Pathogenic Mechanisms of Hypertrophic Cardiomyopathy beyond Sarcomere Dysfunction
30. Plasma Proteomic Profiling in Hypertrophic Cardiomyopathy Patients before and after Surgical Myectomy Reveals Post-Procedural Reduction in Systemic Inflammation
31. Is it possible to transform cardiac scar tissue into beating heart muscle in humans?
32. Mitochondrial proteome remodelling in pressure overload-induced heart failure: the role of mitochondrial oxidative stress
33. Role of Tafazzin in Mitochondrial Function, Development and Disease
34. Deficiency of cathepsin S reduces atherosclerosis in LDL receptor-deficient mice
35. Abstract 771: Identification of Pathological Pathways in Hypertrophic Cardiomyopathy via Single Nuclei RNA-Sequencing
36. Abstract 113: Recombinant Tafazzin Enzyme Replacement Therapy Rescues Metabolic and Functional Defects in a Mouse Model of Barth Syndrome
37. Abstract 16604: Recombinant Tafazzin Enzyme Replacement Therapy Rescues Metabolic and Functional Defects in a Mouse Model of Barth Syndrome
38. Placental Vascular Calcification and Cardiovascular Health: It Is Time to Determine How Much of Maternal and Offspring Health Is Written in Stone
39. Abstract 430: In Utero Exposure to Diesel Exhaust Reprograms the Metabolic Phenotype of Neonatal Cardiomyocytes
40. Air pollution and adverse cardiac remodeling: clinical effects and basic mechanisms
41. Smooth Muscle Notch1 Mediates Neointimal Formation Following Vascular Injury
42. Neonatal Diesel Exhaust Particulate Exposure Does Not Predispose Mice to Adult Cardiac Hypertrophy or Heart Failure.
43. Reprogramming cell fate: a changing story
44. An Optimized and Simplified System of Mouse Embryonic Stem Cell Cardiac Differentiation for the Assessment of Differentiation Modifiers
45. In Utero Exposure to Diesel Exhaust Air Pollution Promotes Adverse Intrauterine Conditions, Resulting in Weight Gain, Altered Blood Pressure, and Increased Susceptibility to Heart Failure in Adult Mice
46. In utero and early life exposure to diesel exhaust air pollution increases adult susceptibility to heart failure in mice
47. Sodium-Dependent Phosphate Cotransporters and Phosphate-Induced Calcification of Vascular Smooth Muscle Cells
48. Inhalation of diesel exhaust does not exacerbate cardiac hypertrophy or heart failure in two mouse models of cardiac hypertrophy
49. Endothelial deletion of ADAM17 in mice results in defective remodeling of the semilunar valves and cardiac dysfunction in adults
50. Intracellular Delivery of Proteins with Cell-Penetrating Peptides for Therapeutic Uses in Human Disease.
Catalog
Books, media, physical & digital resources
Discovery Service for Jio Institute Digital Library
For full access to our library's resources, please sign in.