120 results on '"Bajpai, Manisha"'
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2. EPR and optical studies of VO 2+ doped diglycine calcium chloride tetrahydrate single crystals
3. Gene Expression in Barrett’s Esophagus Cell Lines Resemble Esophageal Squamous Cell Carcinoma Instead of Esophageal Adenocarcinoma
4. Barrett’s Epithelium to Esophageal Adenocarcinoma: Is There a “Point of No Return”?
5. Recurring Translocations in Barrett’s Esophageal Adenocarcinoma
6. Repeated exposure to acid and bile selectively induces colonic phenotype expression in a heterogeneous Barrett's epithelial cell line
7. Prolonged exposure to acid and bile induces chromosome abnormalities that precede malignant transformation of benign Barrett’s epithelium
8. Improved Grain distribution in polymer thin films after electric polarization
9. Tu1160 – Gene Expression Signature of the “Point of No Return” in Barrett’s Epithelial Carcinogenesis
10. Sa2015 – 5-Aminosalicylic Acid Inhibits Xenobiotic Metabolism in Patients with Ulcerative Colitis: A Novel Pathway for Chemoprevention
11. Tu1841 – Comparative Analysis of Blood and Mucosal Mir-206 in Ulcerative Colitis Patients Treated with 5-Aminosalicylic Acid (5-ASA)
12. 19 - In vitro Studies of HTERT Immortalized Normal Esophageal Squamous Epithelial Cells show Progressive Morphological and Molecular Characteristics of Inflammation:Metaplasia: Neoplasia Sequence
13. Review on Optical and Electrical Properties of Conducting Polymers
14. Barrett’s metaplasia develops from cellular reprograming of esophageal squamous epithelium due to gastroesophageal reflux
15. In Non-Neoplastic Barrett's Epithelial Cells, Acid and Bile Salts Induce Epithelial Mesenchymal Transition (EMT) through a Hif-Mediated Reduction in Microrna-200a and -200b Expression
16. Novel Recurring Fusions: Predictive Markers of Esophageal Adenocarcinoma
17. Curcumin, a Nutraceutical, Prevents Acid and Bile Induced Colonic Phenotype of Metaplasia in Bar-T Cells
18. Effect of Benzo[A]Pyrene Along with Acid and Bile is Highly Carcinogenic as Shown in Thein-Vitro Barrett's Esophagus Carcinogenesis (BEC) Model
19. Acid and Bile Induced TGFB1 and NOTCH1 Regulates Epithelial Mesenchymal Transition in Barrett’s Carcinogenesis
20. Su2061 Nano-Curcumin Potentiates the Anti-Neoplastic Effects of Mesalamine in Colorectal Cancer
21. Su2052 NOTCH1 Is Overexpressed in Barrett's Epithelial Neoplasia and Can Be Inhibited by Curcumin
22. Tu2055 BMP Pathway Inhibitors May Prevent Neoplastic Progression of Barrett's Epithelium
23. Sa1886 Esophageal Squamous Cells Exposed Repeatedly to Acid and Bile Salts In Vitro Exhibit Columnar Differentiation With Features of Barrett's Metaplasia
24. 544 In Non-Neoplastic Barrett's Epithelial Cells, Acid and Bile Salts Activate VEGF Signaling to Induce Epithelial Mesenchymal Transition Through ZEB2, a Transcriptional Repressor of E-Cadherin
25. Sa1957 Nano-Curcumin Inhibits Neoplastic Process in the Barrett's Epithelial Carcinogenesis Model
26. Sa1915 Chromosome Translocation T(2,10,16) Marks ‘Point of No-Return’ in Barrett's Epithelium Carcinogenesis, In-Vitro Model
27. Tu1992 - Novel Recurring Fusions: Predictive Markers of Esophageal Adenocarcinoma
28. Mo2055 - Curcumin, a Nutraceutical, Prevents Acid and Bile Induced Colonic Phenotype of Metaplasia in Bar-T Cells
29. Mo2048 - Effect of Benzo[A]Pyrene Along with Acid and Bile is Highly Carcinogenic as Shown in Thein-Vitro Barrett's Esophagus Carcinogenesis (BEC) Model
30. Mo1030 - In Non-Neoplastic Barrett's Epithelial Cells, Acid and Bile Salts Induce Epithelial Mesenchymal Transition (EMT) through a Hif-Mediated Reduction in Microrna-200a and -200b Expression
31. Sa1158 - Acid and Bile Induced TGFB1 and NOTCH1 Regulates Epithelial Mesenchymal Transition in Barrett’s Carcinogenesis
32. Sa1960 Carcinogenesis-Genes Identified in Colon Tissue As Possible Targets for Chemo Preventive Effect of Mesalamine in Patients With Chronic Ulcerative Colitis
33. Sa1954 Benzo[a]Pyrene-Induced CYP 1A1/1B1 Genes May Promote Development of Esophageal Adenocarcinoma While Curcumin Attenuates Such Induction
34. Tu1667 Genomic Changes Correlate With Transformed Phenotype in a Dynamic in-Vitro Model of Barrett's Carcinogenesis
35. 930 Acid and Bile Salts Induce Features of Epithelial Mesenchymal Transition in Non-Neoplastic Barrett's Epithelial Cells Through Increased VEGFSignaling
36. Mo1935 Exposure to Acid and Bile Salts Induces Features of Epithelial Mesenchymal Transition in Non-Neoplastic Barrett's Epithelial Cells: A Potential Mechanism for Reflux-Induced Remodeling and Carcinogenesis in Barrett's Esophagus
37. Mo1946 Progressive Increase in Proliferation, Reduced Apoptosis, and Development of Serum Dependence in Neoplastic Cells of the Barrett's Epithelium Carcinogenesis Model
38. Mo1163 Carcinogenic Effects of Benzo[a]Pyrene (Cyp 450 Activation) on HET-1A and Bar-T Cell Line Could Be Reversed by Curcumin
39. Mo1938 Differential Response of Normal Esophageal Squamous Cells to Acid and Bile Salt-Induced Injury May Predispose to Barrett's Metaplasia
40. Mo1903 Molecular Effect of Acid and Bile Suppression on Progression of Be to Neoplasia
41. Su1179 Acid Suppression Can Impede Progression of Barrett's Epithelium to Dysplasia
42. Su1178 High Resolution Integrative Analysis Reveals Widespread Genetic and Epigenetic Alterations in Barrett's Epithelial Cells Exposed to Acid and Bile
43. TC22, a Novel Colorectal Cancer Biomarker Remains Continuously Suppressed Upon Repeated Mesalamine Exposure to Colorectal Cancer Cells
44. Aberrant, DNA Damage-Response May Contribute in the Carcinogenesis Process in Barrett's Epithelium
45. Continued Mesalamine Exposure is Required for Sustained Anti-Inflammatory and Chemoprevention Effects: Molecular Evidence Using an in-Vitro Model
46. Cellular Phenotypic Changes With Long-Term Acid and Bile Exposure to a Barrett's Epithelial Cell Line
47. S1066 Increase Oncogenecity of Bar-T Cells With Continued Exposure to Acid Plus Bile Beyond 56 Weeks
48. S1058 Modulation of Cell Cycle Pathways in an in-Vitro Chronic Acid Plus Bile Exposure Model of Barrett's Epithelium
49. S1962 Tropomyosin Isoform, TC22, a Novel Biomarker Associated With Neoplasia and Carcinoma
50. EPR and optical studies of VO2+ doped diglycine calcium chloride tetrahydrate single crystals
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