165 results on '"Antonsson, Bruno"'
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2. Mitochondrial Release of Apoptosis-Inducing Factor Occurs Downstream of Cytochrome c Release in Response to Several Proapoptotic Stimuli
3. A Novel, High Conductance Channel of Mitochondria Linked to Apoptosis in Mammalian Cells and Bax Expression in Yeast
4. The Release of Cytochrome c from Mitochondria during Apoptosis of NGF-Deprived Sympathetic Neurons Is a Reversible Event
5. Bid-Induced Conformational Change of Bax Is Responsible for Mitochondrial Cytochrome c Release during Apoptosis
6. Bax-Induced Cytochrome C Release from Mitochondria Is Independent of the Permeability Transition Pore but Highly Dependent on Mg 2+ Ions
7. Catalytic Activation of the Phosphatase MKP-3 by ERK2 Mitogen-Activated Protein Kinase
8. Regulation of Microtubule Dynamics by the Neuronal Growth-Associated Protein SCG10
9. Dissecting Processing and Apoptotic Activity of a Cysteine Protease by Mutant Analysis
10. The Phosphoprotein Stathmin Is Essential for Nerve Growth Factor-Stimulated Differentiation
11. Inhibition of Bax Channel-Forming Activity by Bcl-2
12. tcBid promotes Ca2+ signal propagation to the mitochondria: control of Ca2+ permeation through the outer mitochondrial membrane
13. Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions
14. MAC Inhibitors Neutralize the Pro-Apoptotic Effects of Tbid
15. BAX channel activity mediates lysosomal disruption linked to Parkinson disease
16. Lysosome-Mediated Apoptosis is Associated with Cathepsin D-Specific Processing of Bid at Phe24,Trp48, and Phe183
17. Cathepsin D-mediated processing of Bid at Phe24, Trp48, and Phe183
18. Stimulation of Bax Mitochondrial Localization by Bcl-xL
19. tBid and cardiolipin - trying an insight to the interplay of cell apoptosis key players in a simple model system
20. Bax channel inhibitors prevent mitochondrion-mediated apoptosis and protect neurons in a model of global brain ischemia.
21. Differences in the glycosylation of recombinant proteins expressed in HEK and CHO cells
22. Bcl-xL Increases Bax Mitochondrial Localization and Activation in Non-Apoptotic Cells
23. Interactions Between Bcl-2, Bcl-xl, and Bax at the Mitochondria: Keep Your Friends Close but Your Enemies Closer
24. BAX insertion, oligomerization, and outer membrane permeabilization in brain mitochondria: Role of permeability transition and SH-redox regulation
25. Dissimilar mechanisms of cytochrome c release induced by octyl glucoside-activated BAX and by BAX activated with truncated BID
26. Bad Targets the Permeability Transition Pore Independent of Bax or Bak to Switch between Ca2+-Dependent Cell Survival and Death
27. Apoptosis in FL5.12 cells is suppressed by inhibitors of the Mitochondrial Apoptosis-induced Channel MAC
28. Endophilin B1/Bif-1 Stimulates BAX Activation Independently from Its Capacity to Produce Large Scale Membrane Morphological Rearrangements
29. Design Of An ELISA Protocol For A Rapid Quantification Of Activated/oligomerized Bax During Apoptosis
30. Oligomeric BAX induces mitochondrial permeability transition and complete cytochrome c release without oxidative stress
31. Mitochondrial Cholesterol Contributes to Chemotherapy Resistance in Hepatocellular Carcinoma
32. Mechanism of Mitochondrial Glutathione-Dependent Hepatocellular Susceptibility to TNF Despite NF-κB Activation
33. BIM and tBID Are Not Mechanistically Equivalent When Assisting BAX to Permeabilize Bilayer Membranes
34. Bax Channel Inhibitors Prevent Mitochondrion-mediated Apoptosis and Protect Neurons in a Model of Global Brain Ischemia
35. Oligomeric Bax Is a Component of the Putative CytochromecRelease Channel MAC, Mitochondrial Apoptosis-induced Channel
36. Lipidic Pore Formation by the Concerted Action of Proapoptotic BAX and tBID
37. Effects of cytochromecon the mitochondrial apoptosis-induced channel MAC
38. Bid, but Not Bax, Regulates VDAC Channels
39. Investigation of the role of the C-terminus of Bax and of tc-Bid on Bax interaction with yeast mitochondria
40. Bax oligomerization in mitochondrial membranes requires tBid (caspase-8-cleaved Bid) and a mitochondrial protein
41. Bax and heart mitochondria: uncoupling and inhibition of respiration without permeability transition
42. Bid induces cytochrome c-impermeable Bax channels in liposomes
43. Rapid Kinetics of tBid-induced Cytochrome c and Smac/DIABLO Release and Mitochondrial Depolarization
44. c‐Jun N‐terminal kinase‐3 (JNK3)/stress‐activated protein kinase‐β (SAPKβ) binds and phosphorylates the neuronal microtubule regulator SCG10
45. Oligomeric C‐terminal truncated Bax preferentially releases cytochrome c but not adenylate kinase from mitochondria, outer membrane vesicles and proteoliposomes
46. Phosphorylation of Bid by Casein Kinases I and II Regulates Its Cleavage by Caspase 8
47. Bax Is Present as a High Molecular Weight Oligomer/Complex in the Mitochondrial Membrane of Apoptotic Cells
48. The Destabilization of Lipid Membranes Induced by the C-terminal Fragment of Caspase 8-cleaved Bid Is Inhibited by the N-terminal Fragment
49. Rapid spectrophotometric method for quantitation of cytochrome c release from isolated mitochondria or permeabilized cells revisited
50. Bid Induces the Oligomerization and Insertion of Bax into the Outer Mitochondrial Membrane
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