245 results on '"Yusa, Kosuke"'
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2. Genome-wide screening identifies Polycomb repressive complex 1.3 as an essential regulator of human naïve pluripotent cell reprogramming
3. RENGE infers gene regulatory networks using time-series single-cell RNA-seq data with CRISPR perturbations
4. B1 SINE-binding ZFP266 impedes mouse iPSC generation through suppression of chromatin opening mediated by reprogramming factors
5. Sphingomyelin synthase 1 supports two steps of rubella virus life cycle
6. Transposon delivery for CRISPR-based loss-of-function screen in mice identifies NF2 as a cooperating gene involved with the canonical WNT signaling molecular class of hepatocellular carcinoma
7. The CADM1 tumor suppressor gene is a major candidate gene in MDS with deletion of the long arm of chromosome 11
8. Author Correction: Pooled extracellular receptor-ligand interaction screening using CRISPR activation
9. Minimal genome-wide human CRISPR-Cas9 library
10. Genome-wide CRISPR Screens in T Helper Cells Reveal Pervasive Crosstalk between Activation and Differentiation
11. Genome‐wide loss‐of‐function genetic screen identifies INSIG2 as the vulnerability of hepatitis B virus‐integrated hepatoma cells
12. Genome-wide CRISPR-KO Screen Uncovers mTORC1-Mediated Gsk3 Regulation in Naive Pluripotency Maintenance and Dissolution
13. Prioritization of cancer therapeutic targets using CRISPR–Cas9 screens
14. Constitutively Active SMAD2/3 Are Broad-Scope Potentiators of Transcription-Factor-Mediated Cellular Reprogramming
15. Molecular synergy underlies the co-occurrence patterns and phenotype of NPM1-mutant acute myeloid leukemia
16. Low rates of mutation in clinical grade human pluripotent stem cells under different culture conditions
17. Agreement between two large pan-cancer CRISPR-Cas9 gene dependency data sets
18. Functional linkage of gene fusions to cancer cell fitness assessed by pharmacological and CRISPR-Cas9 screening
19. Structural rearrangements generate cell-specific, gene-independent CRISPR-Cas9 loss of fitness effects
20. RENGE infers gene regulatory networks using time-series single-cell RNA-seq data with CRISPR perturbations
21. ATM orchestrates the DNA-damage response to counter toxic non-homologous end-joining at broken replication forks
22. SRPK1 maintains acute myeloid leukemia through effects on isoform usage of epigenetic regulators including BRD4
23. Pooled extracellular receptor-ligand interaction screening using CRISPR activation
24. Unsupervised correction of gene-independent cell responses to CRISPR-Cas9 targeting
25. A Genetic Progression Model of BrafV600E-Induced Intestinal Tumorigenesis Reveals Targets for Therapeutic Intervention
26. The critical role of histone H2A-deubiquitinase Mysm1 in hematopoiesis and lymphocyte differentiation
27. Peroxisomal Membrane Protein PMP34 Is Involved in the Human Papillomavirus Infection Pathway
28. Interhomolog recombination and loss of heterozygosity in wild-type and Bloom syndrome helicase (BLM)-deficient mammalian cells
29. A hyperactive piggyBac transposase for mammalian applications
30. PiggyBac Transposon Mutagenesis: A Tool for Cancer Gene Discovery in Mice
31. Pharmacologic characterization of TBP1901, a prodrug form of aglycone curcumin, and CRISPR-Cas9 screen for therapeutic targets of aglycone curcumin
32. Inhibition of Lysine Acetyltransferases p300/CBP Overcomes BTK Inhibitor Resistance in Mantle Cell Lymphoma Cells
33. CNOT4 Knockout Induces Proteasome Inhibitor Resistance in Multiple Myeloma Cells
34. Targeted gene correction of α1-antitrypsin deficiency in induced pluripotent stem cells
35. Additional file 1 of Minimal genome-wide human CRISPR-Cas9 library
36. Genome-wide phenotype analysis in ES cells by regulated disruption of Bloom's syndrome gene
37. Measurement of the nuclear concentration of α-ketoglutarate during adipocyte differentiation by using a fluorescence resonance energy transfer-based biosensor with nuclear localization signals
38. Mobilization of giant piggyBac transposons in the mouse genome
39. Rad54 is dispensable for the ALT pathway
40. KAT7 is a genetic vulnerability of acute myeloid leukemias driven by MLL rearrangements
41. Additional file 1: of Structural rearrangements generate cell-specific, gene-independent CRISPR-Cas9 loss of fitness effects
42. Genetic Vulnerabilities of DNMT3AR882H in Myeloid Malignancies
43. CRISPR-Knockout Screen Identifies Dmap1 as a Regulator of Chemically Induced Reprogramming and Differentiation of Cardiac Progenitors
44. CRISPR-Knockout Screen Identifies Dmap1 as a Regulator of Chemically Induced Reprogramming and Differentiation of Cardiac Progenitors
45. The CADM1tumor suppressor gene is a major candidate gene in MDS with deletion of the long arm of chromosome 11
46. JACKS: joint analysis of CRISPR/Cas9 knockout screens
47. Genome-scale identification of cellular pathways required for cell surface recognition
48. Genome-wide and high-density CRISPR-Cas9 screens identify point mutations in PARP1 causing PARP inhibitor resistance
49. A CRISPR knockout screen identifies SETDB1-target retroelement silencing factors in embryonic stem cells
50. Author Correction: Optimised metrics for CRISPR-KO screens with second-generation gRNA libraries
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