220 results on '"Peng, Tianqing"'
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2. Inhibition of NK cell cytotoxicity by tubular epithelial cell expression of Clr-b and Clr-f
3. Myeloid cell-specific deletion of Capns1 prevents macrophage polarization toward the M1 phenotype and reduces interstitial lung disease in the bleomycin model of systemic sclerosis
4. MLKL-mediated necroptosis is a target for cardiac protection in mouse models of type-1 diabetes
5. Correction: Enhancing Specific-Antibody Production to the ragB Vaccine with GITRL That Expand Tfh, IFN-γ+ T Cells and Attenuates Porphyromonas gingivalis Infection in Mice
6. Protective role of endothelial calpain knockout in lipopolysaccharide-induced acute kidney injury via attenuation of the p38-iNOS pathway and NO/ROS production
7. Abstract P1149: Junctophilin-2 Promotes Cardiomyocyte Survival By Blocking Murf1-mediated Junctin Ubiquitination And Proteasome-dependent Degradation
8. Lipocalin 10 is essential for protection against inflammation-triggered vascular leakage by activating LDL receptor-related protein 2-slingshot homologue 1 signalling pathway
9. Calpain Activation and Organ Failure in Sepsis: Molecular Insights and Therapeutic Perspectives
10. An Hsp20-FBXO4 Axis Regulates Adipocyte Function through Modulating PPARγ Ubiquitination
11. Selective deletion of endothelial cell calpain in mice reduces diabetic cardiomyopathy by improving angiogenesis
12. Administration of losartan preserves cardiomyocyte size and prevents myocardial dysfunction in tail-suspended mice by inhibiting p47phox phosphorylation, NADPH oxidase activation and MuRF1 expression
13. Molecular Determinants of Calpain-dependent Cleavage of Junctophilin-2 Protein in Cardiomyocytes
14. Synthesis of N, N, N-trimethyl chitosan-based nanospheres for the prolonged release of curcumin
15. Renewal of embryonic and neonatal-derived cardiac-resident macrophages in response to environmental cues abrogated their potential to promote cardiomyocyte proliferation via Jagged-1–Notch1
16. Abstract 20228: Nicotinamide Riboside Improves Autophagic Flux and Prevents Doxorubicin-Induced Cardiac Injury
17. Loss of Lipocalin 10 Exacerbates Diabetes-Induced Cardiomyopathy via Disruption of Nr4a1-Mediated Anti-Inflammatory Response in Macrophages
18. Additional file 1 of MLKL-mediated necroptosis is a target for cardiac protection in mouse models of type-1 diabetes
19. Renewal of embryonic and neonatal-derived cardiac-resident macrophages in response to environmental cues abrogated their potential to promote cardiomyocyte proliferation via Jagged-1–Notch1.
20. Silencing of miR-195 reduces diabetic cardiomyopathy in C57BL/6 mice
21. Gamma-Aminobutyrate Transaminase Protects against Lipid Overload-Triggered Cardiac Injury in Mice
22. Therapeutic effects of continuous infusion of brain natriuretic peptides on postmyocardial infarction ventricular remodelling in rats
23. Calpain-Mediated Mitochondrial Damage: An Emerging Mechanism Contributing to Cardiac Disease
24. Administration of losartan preserves cardiomyocyte size and prevents myocardial dysfunction in tail-suspended mice by inhibiting p47 phox phosphorylation, NADPH oxidase activation and MuRF1 expression
25. Correction: Enhancing Specific-Antibody Production to the ragB Vaccine with GITRL That Expand Tfh, IFN-γ+ T Cells and Attenuates Porphyromonas gingivalis Infection in Mice.
26. Sectm1a Facilitates Protection against Inflammation-Induced Organ Damage through Promoting TRM Self-Renewal
27. Administration of GDF3 Into Septic Mice Improves Survival via Enhancing LXRα-Mediated Macrophage Phagocytosis
28. Calpain activation mediates microgravity-induced myocardial abnormalities in mice via p38 and ERK1/2 MAPK pathways
29. Over-expression of calpastatin aggravates cardiotoxicity induced by doxorubicin
30. Rac1 signalling mediates doxorubicin-induced cardiotoxicity through both reactive oxygen species-dependent and -independent pathways
31. Targeted Inhibition of Calpain Reduces Myocardial Hypertrophy and Fibrosis in Mouse Models of Type 1 Diabetes
32. MicroRNA-195 promotes palmitate-induced apoptosis in cardiomyocytes by down-regulating Sirt1
33. Tsg101 Is Involved in the Sorting and Re-Distribution of Glucose Transporter-4 to the Sarcolemma Membrane of Cardiac Myocytes
34. Tsg101 positively regulates P62-Keap1-Nrf2 pathway to protect hearts against oxidative damage
35. Sectm1a deficiency aggravates inflammation-triggered cardiac dysfunction through disruption of LXRα signalling in macrophages
36. GDF3 Protects Mice against Sepsis-Induced Cardiac Dysfunction and Mortality by Suppression of Macrophage Pro-Inflammatory Phenotype
37. Deficiency of Rac1 Blocks NADPH Oxidase Activation, Inhibits Endoplasmic Reticulum Stress, and Reduces Myocardial Remodeling in a Mouse Model of Type 1 Diabetes
38. Rac1 Is Required for Cardiomyocyte Apoptosis During Hyperglycemia
39. Over-expression of calpastatin inhibits calpain activation and attenuates myocardial dysfunction during endotoxaemia
40. JNK1/c-fos inhibits cardiomyocyte TNF-α expression via a negative crosstalk with ERK and p38 MAPK in endotoxaemia
41. Disruption of phospholipase Cγ1 signalling attenuates cardiac tumor necrosis factor-α expression and improves myocardial function during endotoxemia
42. Corrigendum: Calpain activation contributes to hyperglycemia-induced apoptosis in cardiomyocytes (Cardiovascular Research (2009) 84 (100-110) DOI: 10.1093/cvr/cvp189)
43. Identifying the enzyme involved in Vacuolar ATPase acetylation during doxorubicin-induced cardiotoxicity
44. MicroRNA-223 is essential for maintaining functional β-cell mass during diabetes through inhibiting both FOXO1 and SOX6 pathways
45. Prevention of hyperglycemia-induced myocardial apoptosis by gene silencing of Toll-like receptor-4
46. Sectm1a deficiency aggravates inflammation-triggered cardiac dysfunction through disruption of LXRα signalling in macrophages.
47. Calpain Activation and Organ Failure in Sepsis: Molecular Insights and Therapeutic Perspectives
48. Circulating Exosomes Isolated from Septic Mice Induce Cardiovascular Hyperpermeability Through Promoting Podosome Cluster Formation
49. Mitochondrial calpain-1 disrupts ATP synthase and induces superoxide generation in type 1 diabetic hearts: A novel mechanism contributing to diabetic cardiomyopathy
50. Disruption of calpain reduces lipotoxicity-induced cardiac injury by preventing endoplasmic reticulum stress
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