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1. Adaptation to persistent growth in the H9 cell line renders a primary isolate of human immunodeficiency virus type 1 sensitive to neutralization by vaccine sera

2. Restoration of virulence in the attenuated Candid#1 vaccine virus requires reversion at both positions 168 and 427 in the envelope glycoprotein GPC.

3. Fusogenic structural changes in arenavirus glycoproteins are associated with viroporin activity.

4. Second-Generation Live-Attenuated Candid#1 Vaccine Virus Resists Reversion and Protects against Lethal Junín Virus Infection in Guinea Pigs.

5. Epistastic Interactions within the Junín Virus Envelope Glycoprotein Complex Provide an Evolutionary Barrier to Reversion in the Live-Attenuated Candid#1 Vaccine.

6. Convergent immunological solutions to Argentine hemorrhagic fever virus neutralization.

7. Myristoylation of the Arenavirus Envelope Glycoprotein Stable Signal Peptide Is Critical for Membrane Fusion but Dispensable for Virion Morphogenesis.

8. Small-Molecule Fusion Inhibitors Bind the pH-Sensing Stable Signal Peptide-GP2 Subunit Interface of the Lassa Virus Envelope Glycoprotein.

9. Arenavirus infection induces discrete cytosolic structures for RNA replication.

10. Dissection of the role of the stable signal peptide of the arenavirus envelope glycoprotein in membrane fusion.

11. The curious case of arenavirus entry, and its inhibition.

12. Biochemical reconstitution of hemorrhagic-fever arenavirus envelope glycoprotein-mediated membrane fusion.

13. A specific interaction of small molecule entry inhibitors with the envelope glycoprotein complex of the Junín hemorrhagic fever arenavirus.

14. T-705 (favipiravir) inhibition of arenavirus replication in cell culture.

15. Structure of a zinc-binding domain in the Junin virus envelope glycoprotein.

16. An antibody directed against the fusion peptide of Junin virus envelope glycoprotein GPC inhibits pH-induced membrane fusion.

17. Assembly of arenavirus envelope glycoprotein GPC in detergent-soluble membrane microdomains.

18. Intersubunit interactions modulate pH-induced activation of membrane fusion by the Junin virus envelope glycoprotein GPC.

19. pH-induced activation of arenavirus membrane fusion is antagonized by small-molecule inhibitors.

20. Unique small molecule entry inhibitors of hemorrhagic fever arenaviruses.

21. A novel zinc-binding domain is essential for formation of the functional Junín virus envelope glycoprotein complex.

22. Bitopic membrane topology of the stable signal peptide in the tripartite Junín virus GP-C envelope glycoprotein complex.

23. Transferrin receptor 1 is a cellular receptor for New World haemorrhagic fever arenaviruses.

24. Distinct requirements for signal peptidase processing and function in the stable signal peptide subunit of the Junín virus envelope glycoprotein.

25. Oligomerization of hepatitis C virus core protein is crucial for interaction with the cytoplasmic domain of E1 envelope protein.

26. Role of the stable signal peptide of Junín arenavirus envelope glycoprotein in pH-dependent membrane fusion.

27. Furin cleavage of the SARS coronavirus spike glycoprotein enhances cell-cell fusion but does not affect virion entry.

28. Role of the stable signal peptide and cytoplasmic domain of G2 in regulating intracellular transport of the Junín virus envelope glycoprotein complex.

29. Genetic analysis of heptad-repeat regions in the G2 fusion subunit of the Junín arenavirus envelope glycoprotein.

30. Serine-scanning mutagenesis studies of the C-terminal heptad repeats in the SARS coronavirus S glycoprotein highlight the important role of the short helical region.

31. The signal peptide of the Junín arenavirus envelope glycoprotein is myristoylated and forms an essential subunit of the mature G1-G2 complex.

32. Role of hydrophobic residues in the central ectodomain of gp41 in maintaining the association between human immunodeficiency virus type 1 envelope glycoprotein subunits gp120 and gp41.

33. Genetic evidence that interhelical packing interactions in the gp41 core are critical for transition of the human immunodeficiency virus type 1 envelope glycoprotein to the fusion-active state.

35. Structural and functional analysis of interhelical interactions in the human immunodeficiency virus type 1 gp41 envelope glycoprotein by alanine-scanning mutagenesis.

36. Antibody binding and neutralization of primary and T-cell line-adapted isolates of human immunodeficiency virus type 1.

37. Turning a corner on HIV neutralization?

38. Fusion-competent vaccines: broad neutralization of primary isolates of HIV.

39. Continued utilization of CCR5 coreceptor by a newly derived T-cell line-adapted isolate of human immunodeficiency virus type 1.

40. Coreceptor utilization by human immunodeficiency virus type 1 is not a primary determinant of neutralization sensitivity.

41. Adaptation to persistent growth in the H9 cell line renders a primary isolate of human immunodeficiency virus type 1 sensitive to neutralization by vaccine sera.

42. L-696,229 specifically inhibits human immunodeficiency virus type 1 reverse transcriptase and possesses antiviral activity in vitro.

43. Prevention of HIV-1 infection in chimpanzees by gp120 V3 domain-specific monoclonal antibody.

44. Viral resistance to human immunodeficiency virus type 1-specific pyridinone reverse transcriptase inhibitors.

45. Pyridinone derivatives: specific human immunodeficiency virus type 1 reverse transcriptase inhibitors with antiviral activity.

46. Recombinant feline herpesviruses expressing feline leukemia virus envelope and gag proteins.

47. Correlation of dihydrofolate reductase elevation with gene amplification in a homogeneously staining chromosomal region in L5178Y cells.

48. Isolation of a cDNA clone for the human HLA-DR antigen alpha chain by using a synthetic oligonucleotide as a hybridization probe.

49. Phenotypic expression in E. coli of a DNA sequence coding for mouse dihydrofolate reductase.

50. Interleukin 2 acts as an adjuvant to increase the potency of inactivated rabies virus vaccine.

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