41 results on '"Nenquin, Myriam"'
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2. In vitro insulin secretion by pancreatic tissue from infants with diazoxide-resistant congenital hyperinsulinism deviates from model predictions
3. Activators of PKA and Epac Distinctly Influence Insulin Secretion and Cytosolic Ca2+ in Female Mouse Islets Stimulated by Glucose and Tolbutamide
4. Glucose Stimulates Ca2+ Influx and Insulin Secretion in 2-Week-old β-Cells Lacking ATP-sensitive K+ Channels
5. Congenital Hyperinsulinism Caused by Hexokinase I Expression or Glucokinase-Activating Mutation in a Subset of β-Cells
6. cAMP-Mediated and Metabolic Amplification of Insulin Secretion Are Distinct Pathways Sharing Independence of β-Cell Microfilaments
7. Glucose Controls Cytosolic Ca2+ and Insulin Secretion in Mouse Islets Lacking Adenosine Triphosphate-Sensitive K+ Channels Owing to a Knockout of the Pore-Forming Subunit Kir6.2
8. Nutrient Control of Insulin Secretion in Isolated Normal Human Islets
9. In Vivo and In Vitro Glucose-Induced Biphasic Insulin Secretion in the Mouse: Pattern and Role of Cytoplasmic Ca2+ and Amplification Signals in β-Cells
10. SERCA3 Ablation Does Not Impair Insulin Secretion but Suggests Distinct Roles of Different Sarcoendoplasmic Reticulum Ca2+ Pumps for Ca2+ Homeostasis in Pancreatic β-cells
11. Signals and Pools Underlying Biphasic Insulin Secretion
12. Inhibition of Protein Synthesis Sequentially Impairs Distinct Steps of Stimulus-secretion Coupling in Pancreatic β Cells*
13. Immaturity of insulin secretion by pancreatic islets isolated from one human neonate
14. Dynamics and Regulation of Insulin Secretion in Pancreatic Islets from Normal Young Children
15. Dynamics of glucose-induced insulin secretion in normal human islets
16. Immaturity of insulin secretion by pancreatic islets isolated from one human neonate.
17. Disruption and stabilization of β-cell actin microfilaments differently influence insulin secretion triggered by intracellular Ca 2+ mobilization or store-operated Ca 2+ entry
18. Inhibition of protein synthesis sequentially impairs distinct steps of stimulus-secretion coupling in pancreatic beta cells
19. Disruption and stabilization of β‐cell actin microfilaments differently influence insulin secretion triggered by intracellular Ca2+ mobilization or store‐operated Ca2+ entry
20. Metabolic amplification of insulin secretion by glucose is independent of β-cell microtubules
21. Metabolic amplifying pathway increases both phases of insulin secretion independently of β-cell actin microfilaments
22. Glucose Controls Cytosolic Ca2+ and Insulin Secretion in Mouse Islets Lacking Adenosine Triphosphate-Sensitive K+ Channels Owing to a Knockout of the Pore-Forming Subunit Kir6.2
23. Glucose-induced Cytosolic pH Changes in β-Cells and Insulin Secretion Are Not Causally Related
24. Overnight Culture Unmasks Glucose-induced Insulin Secretion in Mouse Islets Lacking ATP-sensitive K+ Channels by Improving the Triggering Ca2+ Signal
25. Both Triggering and Amplifying Pathways Contribute to Fuel-induced Insulin Secretion in the Absence of Sulfonylurea Receptor-1 in Pancreatic β-Cells
26. The Elevation of Glutamate Content and the Amplification of Insulin Secretion in Glucose-stimulated Pancreatic Islets Are Not Causally Related
27. The Oscillatory Behavior of Pancreatic Islets from Mice with Mitochondrial Glycerol-3-phosphate Dehydrogenase Knockout
28. Relative contribution of Ca2+-dependent and Ca2+-independent mechanisms to the regulation of insulin secretion by glucose
29. Disruption and stabilization of β-cell actin microfilaments differently influence insulin secretion triggered by intracellular Ca2+ mobilization or store-operated Ca2+ entry
30. Overnight Culture Unmasks Glucose-induced Insulin Secretion in Mouse Islets Lacking ATP-sensitive K+ Channels by Improving the Triggering Ca2+ Signal.
31. Glucose Stimulates Ca2+ Influx and Insulin Secretion in 2-Week-old β-CelIs Lacking ATP-sensitive K+ Channels.
32. In vivo and in vitro glucose-induced biphasic insulin secretion in the mouse: pattern and role of cytoplasmic Ca2+ and amplification signals in beta-cells.
33. SERCA3 ablation does not impair insulin secretion but suggests distinct roles of different sarcoendoplasmic reticulum Ca(2+) pumps for Ca(2+) homeostasis in pancreatic beta-cells.
34. Activators of PKA and Epac Distinctly Influence Insulin Secretion and Cytosolic Ca2+in Female Mouse Islets Stimulated by Glucose and Tolbutamide
35. Magnesium uptake by pancreatic islet cells is modulated by stimulators and inhibitors of the B-cell function.
36. Relative contribution of Ca 2+-dependent and Ca 2+-independent mechanisms to the regulation of insulin secretion by glucose
37. Dynamics of glucose-induced insulin secretion in normal human islets.
38. Metabolic amplification of insulin secretion by glucose is independent of β-cell microtubules.
39. Metabolic amplifying pathway increases both phases of insulin secretion independently of beta-cell actin microfilaments.
40. Glucose-induced cytosolic pH changes in beta-cells and insulin secretion are not causally related: studies in islets lacking the Na+/H+ exchangeR NHE1.
41. Both triggering and amplifying pathways contribute to fuel-induced insulin secretion in the absence of sulfonylurea receptor-1 in pancreatic beta-cells.
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