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2. Hydrogen Sulfide-Releasing Indomethacin-Derivative (ATB-344) Prevents the Development of Oxidative Gastric Mucosal Injuries

5. Tu1534: INTESTINAL ALKALINE PHOSPHATASE THERAPY COUNTERACTS THE INTENSIFICATION OF COLITIS IN OBESE MICE SUBJECTED TO FORCED TREADMILL EXERCISE BY SHAPING THE INTESTINAL MICROBIOTA AND ATTENUATING OF PRO-INFLAMMATORY AND OXIDATIVE STRESS BIOMARKERS

6. Sa1129: THERAPEUTIC EFFECT OF MITOCHONDRIA-TARGETING HYDROGEN SULFIDE PRODRUG, AP39 IN THE HEALING OF CHRONIC GASTRIC ULCERS

7. Mo1618: EXPRESSION OF TIGHT JUNCTION PROTEINS IS MODULATED BY MATERNAL OBESITY INDUCED BY DIFFERENT DIET PROGRAM. EVIDENCE FROM INTESTINE OF PREGNANT DAMS AND THEIR MALE AND FEMALE RAT OFFSPRING

8. Tu1167b: INTRODUCTION TO THE THERAPEUTIC POTENTIAL OF CARBON MONOXIDE (CO) RELEASED FROM PHARMACOLOGICAL DONORS AGAINST BARRETT'S METAPLASIA AND ESOPHAGEAL ADENOCARCINOMA (EAC) DEVELOPMENT

9. Role of Obesity, Physical Exercise, Adipose Tissue-Skeletal Muscle Crosstalk and Molecular Advances in Barrett’s Esophagus and Esophageal Adenocarcinoma

11. Novel Hydrogen Sulfide (H2S)-Releasing BW-HS-101 and Its Non-H2S Releasing Derivative in Modulation of Microscopic and Molecular Parameters of Gastric Mucosal Barrier

12. Fr129 IMPACT OF NOVEL HYDROGEN SULFIDE-RELEASING KETOPROFEN (ATB-352) ON MOLECULAR PATHWAYS AND MICROBIOME PROFILE IN GASTROINTESTINAL (GI) TRACT WITH POSSIBLE DEVELOPMENT OF GI-SAFE POLYPHARMACY WITH OTHER NON-STEROIDAL ANTI INFLAMMATORY DRUGS (NSAIDS)

13. Fr157 DECREASED ACTIVITY OF ENDOGENOUS HYDROGEN SULFIDE (H2S)-PRODUCING ENZYME IN BARRETT'S METAPLASIA PROGRESSION WITH POSSIBLE MODULATION OF METALLOPEPTIDASE INHIBITOR (TIMP1) AND SIGNAL TRANSDUCER AND ACTIVATOR OF TRANSCRIPTION 3 (STAT3) PATHWAYS

14. Sa629 THE COMBINATION OF ALKALINE PHOSPHATASE TREATMENT WITH VOLUNTARY PHYSICAL ACTIVITY AMELIORATES THE INFLAMMATION OF LIVER AND ADIPOSE TISSUE IN MURINE COLITIS WITH ABDOMINAL OBESITY. IMPORTANCE OF CYTOKINES, OXIDATIVE STRESS AND ADIPOMYOKINES

15. Su241 APELIN/AJP SYSTEM PROTECTS THE GASTRIC MUCOSA AGAINST ISCHEMIA-REPERFUSION INJURY VIA ACTIVATION OF CNOS/NO SYSTEM, SENSORY NEUROPEPTIDES AND SUPPRESSION OF OXIDATIVE STRESS

16. Intestinal Alkaline Phosphatase Combined with Voluntary Physical Activity Alleviates Experimental Colitis in Obese Mice. Involvement of Oxidative Stress, Myokines, Adipokines and Proinflammatory Biomarkers

17. Organic carbon monoxide prodrug, BW-CO-111, in protection against chemically-induced gastric mucosal damage

20. Molecular Profile of Barrett’s Esophagus and Gastroesophageal Reflux Disease in the Development of Translational Physiological and Pharmacological Studies

21. Evidence for Cytoprotective Effect of Carbon Monoxide Donor in the Development of Acute Esophagitis Leading to Acute Esophageal Epithelium Lesions

22. 834 EFFECT OF MATERNAL OBESITY INDUCED BY DIFFERENT DIET PROGRAM ON THE PLASMA LEVELS OF APPETITE HORMONES AND INTESTINAL EXPRESSION OF ADIPOKINES AND ADIPO RECEPTORS 1 AND 2 IN MALE AND FEMALE RAT OFFSPRING

23. Mo1034 INTESTINAL PROTECTION INDUCED BY TREATMENT WITH ALKALINE PHOSPHATASE ON THE SEVERITY OF EXPERIMENTAL COLITIS IN OBESE MICE SUBJECTED TO FORCED TREADMILL RUNNING. ROLE OF PROINFLAMMATORY BIOMARKERS, INTESTINAL BARRIER PROTEINS AND OXIDATIVE STRESS

24. Su1122 DEVELOPMENT OF BARRETT'S ESOPHAGUS (BE) AND GASTROESOPHAGEAL REFLUX DISEASE (GERD) EXPERIMENTAL MODELS FOR TRANSLATIONAL PHARMACOLOGICAL STUDIES RELATED TO THE POSSIBLE NOVEL TREATMENT METHODS OF THESE PATHOLOGIES

25. Interaction of epidermal growth factor with COX-2 products and peroxisome proliferator-activated receptor-γ system in experimental rat Barrett’s esophagus

26. Effect of forced physical activity on the severity of experimental colitis in normal weight and obese mice : involvement of oxidative stress and proinflammatory biomarkers

27. Exploiting Significance of Physical Exercise in Prevention of Gastrointestinal Disorders

29. Effect of Forced Physical Activity on the Severity of Experimental Colitis in Normal Weight and Obese Mice. Involvement of Oxidative Stress and Proinflammatory Biomarkers

30. 281 – Role of Endogenous and Exogenous Carbon Monoxide (CO) Released from Co Donor in Esophagoprotection Against Experimental Reflux Esophagitis. Importance of Heme Oxygenase-1 (HO-1), Prostaglandins and Sensory Afferent Neuropeptides

31. Su1102 – Carbon Monoxide Released from Its Pharmacological Donor Prevents Gastric Mucosa Against Oxidative Ischemi/Areperfusion-Induced Injury by the Involvement of the Atpdependent Potassium Channels and Nitric Oxide Synthase Activity

33. The Role of Intestinal Alkaline Phosphatase in Inflammatory Disorders of Gastrointestinal Tract

34. Alterations in Gastric Mucosal Expression of Calcitonin Gene-Related Peptides, Vanilloid Receptors, and Heme Oxygenase-1 Mediate Gastroprotective Action of Carbon Monoxide against Ethanol-Induced Gastric Mucosal Lesions

35. Melatonin in Prevention of the Sequence from Reflux Esophagitis to Barrett’s Esophagus and Esophageal Adenocarcinoma: Experimental and Clinical Perspectives

37. Mo1952 - Curcumin Accelerates the Healing of Experimental Colitis via Mechanism Involving Endogenous Prostaglandins, Nitirc Oxide and the Modulation of Gut Microbiota

38. The Protective Role of Carbon Monoxide (CO) Produced by Heme Oxygenases and Derived from the CO-Releasing Molecule CORM-2 in the Pathogenesis of Stress-Induced Gastric Lesions: Evidence for Non-Involvement of Nitric Oxide (NO)

39. Beneficial Effect of Voluntary Exercise on Experimental Colitis in Mice Fed a High-Fat Diet: The Role of Irisin, Adiponectin and Proinflammatory Biomarkers

40. Role of Gut-Adipose-muscle Axis in Beneficial Effect of Voluntary Exercise on Experimental Colitis in Mice Fed a Diet-Induced Obesity. Involvement of Protective Irisin and Proinflammatory Biomarkers Released from Mesenteric Fat and Colonic Mucosa

41. Sensory Nerves Releasing Calcitonin Gene Related Peptide, Lipid Peroxidation and Expression and Activity of Antioxidative Enzymes in the Mechanism of Gastroprotection Induced by Carbon Monoxide Against Stress Ulcerogenesis

43. Exogenous and Endogenous Hydrogen Sulfide Protects Gastric Mucosa against the Formation and Time-Dependent Development of Ischemia/Reperfusion-Induced Acute Lesions Progressing into Deeper Ulcerations

44. Moderate exercise training attenuates the severity of experimental rodent colitis : the importance of crosstalk between adipose tissue and skeletal muscles

45. Effect of Forced Physical Activity on the Severity of Experimental Colitis in NormalWeight and Obese Mice. Involvement of Oxidative Stress and Proinflammatory Biomarkers

46. 682 Coffee Protects Gastric Mucosa Against Acute Gastric Lesions Induced By Acid-Independent and Acid-Dependent Ulcerogenes. Involvement of Prostaglandins, Nitric Oxide, Sensory Neuropeptides and Vanilloid Receptors

47. Tu1858 Role of Heme Oxygenase-1 and Nuclear Factor (Erythroid-Derived 2)-Like 2 Factor (NRF2) in Pathogenesis of Aspirin-Induced Gastric Damage and Protection by Carbon Monoxide

48. 199 Adiponectin, the Secretory Hormone of Adipocytes, Prevents the Formation of Ischemia-Reperfusion Gastric Lesions via Anti-Inflammatory and Antioxidative Activity Mediated by cNOS/No System and Sensory Afferent Nerves

49. Mo1257 Crosstalk Between Carbon Monoxide (CO) Released From Tricarbonyldichlororuthenium (II) Dimer (Corm-2) and Endogenous Nitric Oxide (NO) in Gastroprotection Against Stress-Induced Gastric Lesions

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