184 results on '"Kyritsi, Konstantina"'
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2. Development of Scaffold-Free Three-Dimensional Cholangiocyte Organoids to Study the Progression of Primary Sclerosing Cholangitis
3. The Functional Roles of Immune Cells in Primary Liver Cancer
4. Prolonged Administration of Melatonin Ameliorates Liver Phenotypes in Cholestatic Murine Model
5. Kupffer Cells: Inflammation Pathways and Cell-Cell Interactions in Alcohol-Associated Liver Disease
6. Knockout of the Tachykinin Receptor 1 in the Mdr2−/− (Abcb4−/−) Mouse Model of Primary Sclerosing Cholangitis Reduces Biliary Damage and Liver Fibrosis
7. Biliary damage and liver fibrosis are ameliorated in a novel mouse model lacking l-histidine decarboxylase/histamine signaling
8. Amelioration of Large Bile Duct Damage by Histamine-2 Receptor Vivo-Morpholino Treatment
9. Downregulation of hepatic stem cell factor by Vivo-Morpholino treatment inhibits mast cell migration and decreases biliary damage/senescence and liver fibrosis in Mdr2−/− mice
10. Knockdown of vimentin reduces mesenchymal phenotype of cholangiocytes in the Mdr2−/− mouse model of primary sclerosing cholangitis (PSC)
11. Pinealectomy or light exposure exacerbates biliary damage and liver fibrosis in cholestatic rats through decreased melatonin synthesis
12. Knockout of α-calcitonin gene-related peptide attenuates cholestatic liver injury by differentially regulating cellular senescence of hepatic stellate cells and cholangiocytes
13. Knockout of secretin receptor reduces biliary damage and liver fibrosis in Mdr2−/− mice by diminishing senescence of cholangiocytes
14. The Secretin/Secretin Receptor Axis Modulates Ductular Reaction and Liver Fibrosis through Changes in Transforming Growth Factor-β1–Mediated Biliary Senescence
15. Regulation of Cellular Senescence by miR-34a in Alcoholic Liver Injury
16. Knockdown of Hepatic Gonadotropin-Releasing Hormone by Vivo-Morpholino Decreases Liver Fibrosis in Multidrug Resistance Gene 2 Knockout Mice by Down-Regulation of miR-200b
17. 662 INHIBITION OF THE GNRH/GNRHR1 AXIS AMELIORATES THE PHENOTYPES OF PRIMARY BILIARY CHOLANGITIS (PBC).
18. Knockout of microRNA-21 reduces biliary hyperplasia and liver fibrosis in cholestatic bile duct ligated mice
19. Loss of apical sodium bile acid transporter alters bile acid circulation and reduces biliary damage in cholangitis
20. Mast cells selectively target large cholangiocytes during biliary injury via H2HR‐mediated cAMP/pERK1/2 signaling
21. The Effects of Taurocholic Acid on Biliary Damage and Liver Fibrosis Are Mediated by Calcitonin-Gene-Related Peptide Signaling
22. Tu1297: MELATONIN RECEPTOR 1A (MT1) KNOCKOUT DECREASES BILIARY DAMAGE AND LIVER STEATOSIS VIA DOWNREGULATION OF MIR-200B IN HIGH FAT DIET (HFD) MODEL OF NONALCOHOLIC FATTY LIVER DISEASE (NAFLD)
23. Tu1316: CROMOLYN SODIUM AMELIORATES MAST CELL(MC)-MEDIATED HEPATIC DAMAGE IN A MURINE MODEL OF NON-ALCOHOLIC FATTY LIVER DISEASE (NAFLD)
24. Su1345: GROWTH HORMONE AND GROWTH HORMONE RECEPTOR SIGNALING AS A NOVEL THERAPEUTIC TARGET FOR PRIMARY SCLEROSING CHOLANGITIS
25. Mo1264: INHIBITION OF TRANSFORMING GROWTH FACTOR BETA 1 (TGFβ1) BY VIVO-MORPHOLINO (VM) AMELIORATES CHOLESTASIS IN THE MDR2-/- MOUSE MODEL OF PRIMARY SCLEROSING CHOLANGITIS (PSC)
26. FGF1 Signaling Modulates Biliary Injury and Liver Fibrosis in the Mdr2−/− Mouse Model of Primary Sclerosing Cholangitis
27. Mast cells (MCs) induce ductular reaction mimicking liver injury in mice via MC-derived TGF-β1 signaling
28. 233 KNOCKDOWN OF THE MT1 MELATONIN RECEPTOR AMELIORATES THE PHENOTYPES OF THE MDR2-/- MOUSE MODEL OF PRIMARY SCLEROSING CHOLANGITIS (PSC) BY DOWNREGULATION OF ORPHAN GPR50 RECEPTOR-PROMOTED CONSTITUTIVE TGF-β1 RECEPTOR SIGNALING.
29. Sa380 KNOCKDOWN OF P16 IMPROVES MAST CELL (MC)-MEDIATED BILIARY SENESCENCE AND NAFLD PHENOTYPES IN A DIET-INDUCED RODENT MODEL
30. Fr366 ADVANCED 3D HUMAN LIVER ORGANOIDS CREATED BY MULTIPLE-HEPATIC LINEAGE CELLS FOR THE STUDY OF LIVER DISEASES
31. 85 MAST CELLS REGULATE CHOLESTATIC LIVER INJURY AND INTESTINAL BILE ACID SIGNALING VIA MODIFICATION OF FXR/FGF15 AXIS
32. 86 LONG-TERM SECRETIN TREATMENT RESTORES THE DUCTULO-CANALICULAR JUNCTIONS (DCJ) AND AMELIORATES BILIARY DAMAGE AND LIVER FIBROSIS IN A MODEL OF LATE-STAGE PRIMARY BILIARY CHOLANGITIS (PBC).
33. 87 DIFFERENTIAL EFFECTS OF MELATONIN AND IL-33/ST-2/NFκB SIGNALING ON MAST CELL-INDUCED PRIMARY SCLEROSING CHOLANGITIS (PSC)
34. FGF1 Signaling Modulates Biliary Injury and Liver Fibrosis in the Mdr2−/− Mouse Model of Primary Sclerosing Cholangitis.
35. Mo1504 FORKHEAD BOX PROTEIN A2 (FOXA2) REGULATES LIVER PHENOTYPES IN THE MULTI-DRUG RESISTANT 2 KNOCKOUT (MDR2-/-) MOUSE MODEL OF PRIMARY SCLEROSING CHOLANGITIS (PSC) AND HUMAN CHOLANGIOCYTES FROM PSC LIVER EXPLANTS
36. 915 KNOCKOUT (KO) OF NEUROKININ RECEPTOR 1 (NK1R) AMELIORATES LIVER PHENOTYPES IN AN ACUTE MODEL OF ALCOHOL-INDUCED LIVER DISEASE (ALD)
37. 659 MELATONIN THERAPY REDUCES BILIARY SENESCENCE, LIVER INFLAMMATION AND FIBROSIS IN A MURINE MODEL OF PRIMARY BILIARY CHOLANGITIS (PBC).
38. Downregulation of p16 Decreases Biliary Damage and Liver Fibrosis in the Mdr2/ Mouse Model of Primary Sclerosing Cholangitis
39. Kupffer Cells
40. 693 CROSSTALK SIGNALING BETWEEN MAST CELLS, HISTAMINE AND STIMULATOR OF INTERFERON GENE (STING/TMEM173) PROMOTES CHOLANGIOCARCINOMA (CCA) TUMORIGENESIS
41. Sa1493 COORDINATED REGULATION OF SENSORY INNERVATION AND MAST CELL ACTIVATION CONTRIBUTES TO BILIARY DAMAGE AND HEPATIC FIBROSIS
42. Sa1491 CHOLANGIOCARCINOMA CELLS SECRETE EXTRACELLULAR VESICLES THAT CONTRIBUTE TO CYTOKINE EXPRESSION AND FIBROGENESIS DURING THE TUMOR MICROENVIRONMENT DEVELOPMENT.
43. Sa1490 PROLONGED ADMINISTRATION OF MELATONIN TO THE MDR2-/- MOUSE MODEL OF PRIMARY SCLEROSIS CHOLANGITIS (PSC) DECREASES BILIARY SENESCENCE AND LIVER FIBROSIS THROUGH RESTORATION OF THE CIRCADIAN RHYTHM.
44. Sa1502 PROMOTION OF M1 MACROPHAGE POLARIZATION AND DUCTULAR REACTION BY MICRORNA-34A DURING CHOLESTATIC LIVER INJURY
45. 454 LONG-TERM ADMINISTRATION OF THE SECRETIN RECEPTOR ANTAGONIST, SCT 5-27, INHIBITS BILIARY SENESCENCE AND LIVER FIBROSIS OF PRIMARY SCLEROSING CHOLANGITIS (PSC) IN THE MDR2-/- MOUSE MODEL.
46. Correction to: Knockout of α-calcitonin gene-related peptide attenuates cholestatic liver injury by differentially regulating cellular senescence of hepatic stellate cells and cholangiocytes
47. Sa1494 INHIBITION OF BILIARY ASBT VIA VIVO-MORPHOLINO TREATMENT DECREASES MAST CELL ACTIVATION AND SUBSEQUENT BILIARY DAMAGE, HEPATIC FIBROSIS AND BILE ACID SIGNALING IN MDR2-/- MICE
48. Sa1510 – Increased Serotonin (5HT) Biliary Synthesis Due to Enhanced Expression of Tryptophan Hydroxylase1 (TPH1) and Reduced Monoamine-Oxidase-A (MAO-A) Expression is Couple with Alcohol-Induced Liver Injury (ALI)
49. Mo1428 – Role of the Aanat/Melatonin/Mt1/Mt2/Per-1 Axis in the Regulation of Biliary Damage and Liver Fibrosis in Cholestatic Mice
50. Sa1502 – Knockout (KO) of the Melatonin Receptor, Mt2, Enhances Alcohol-Induced Ductular Reaction (DR), Biliary Senescence and Hepatic Fibrosis During Alcoholic Liver Disease (ALD)
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