21 results on '"Ermak, Gennady"'
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2. RCAN1-1L is overexpressed in neurons of Alzheimerʼs disease patients
3. RCAN1 (DSCR1 or Adapt78) stimulates expression of GSK-3β
4. Genetic Aberrations in Chernobyl-Related Thyroid Cancers: Implications for Possible Future Nuclear Accidents or Nuclear Attacks
5. Oxidative and calcium stress regulate DSCR1 (Adapt78/MCIP1) protein
6. DSCR1(Adapt78)—A Janus Gene Providing Stress Protection but Causing Alzheimerʼs Disease?
7. Calcium and oxidative stress: from cell signaling to cell death
8. Production of POMC, CRH-R1, MC1, and MC2 Receptor mRNA and Expression of Tyrosinase Gene in Relation to Hair Cycle and Dexamethasone Treatment in the C57BL/6 Mouse Skin
9. Chronic Expression of RCAN1-1L Protein Induces Mitochondrial Autophagy and Metabolic Shift from Oxidative Phosphorylation to Glycolysis in Neuronal Cells.
10. Amyloid-β Toxicity and Tau Hyperphosphorylation are Linked Via RCAN1 in Alzheimer's Disease.
11. Do RCAN1 proteins link chronic stress with neurodegeneration?
12. Mitochondrial fission and cristae disruption increase the response of cell models of Huntington's disease to apoptotic stimuli.
13. Regulator of Calcineurin (RCAN1-1L) Is Deficient in Huntington Disease and Protective against Mutant Huntingtin Toxicity in Vitro.
14. Renaming the DSCR1/Adapt78 gene family as RCAN: regulators of calcineurin.
15. DSCR1(Adapt78) modulates expression of SOD1.
16. Cytotoxic effect of doxycycline and its implications for tet-on gene expression systems
17. The essential role of calcium in induction of the DSCR1 (ADAPT78) gene.
18. Ultraviolet B and Melanocyte-Stimulating Hormone (MSH) Stimulate mRNA Production for ∝MSH Receptors and Proopiomelanocortin-Derived Peptides in Mouse Melanoma Cells and Transformed Keratinocytes.
19. Ultraviolet B stimulates production of corticotropin releasing factor (CRF) by human melanocytes
20. HSP70 mediates dissociation and reassociation of the 26S proteasome during adaptation to oxidative stress
21. Tau protein degradation is catalyzed by the ATP/ubiquitin-independent 20S proteasome under normal cell conditions
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