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2. o/nlythis / all/ein diese

Author

Grabher, Gudrun M., Amann, Sabine, Antretter, Martina, Krabichler, Maria, Oehlinger, Christine, Seeber, Barbara, Straif, Barbara, Strauss, Susann, and Kapfer, Cornelia

Published
1999

3. Me up at does / Mir zu her auf

Author

Grabher, Gudrun M., Amann, Sabine, Antretter, Martina, Krabichler, Maria, Oehlinger, Christine, Seeber, Barbara, Straif, Barbara, Strauss, Susann, and Kapfer, Cornelia

Published
1999

4. because it's / denn's ist ja

Author

Grabher, Gudrun M., Amann, Sabine, Antretter, Martina, Krabichler, Maria, Oehlinger, Christine, Seeber, Barbara, Straif, Barbara, Strauss, Susann, and Kapfer, Cornelia

Published
1999

5. timeless / immerwähr

Author

Grabher, Gudrun M., Amann, Sabine, Antretter, Martina, Krabichler, Maria, Oehlinger, Christine, Seeber, Barbara, Straif, Barbara, Strauss, Susann, and Kapfer, Cornelia

Published
1999

6. t,h;r:u;s,h;e:s / d,r;o:s;s,e;l:n

Author

Grabher, Gudrun M., Amann, Sabine, Antretter, Martina, Krabichler, Maria, Oehlinger, Christine, Seeber, Barbara, Straif, Barbara, Strauss, Susann, and Kapfer, Cornelia

Published
1999

7. silence / stille

Author

Grabher, Gudrun M., Amann, Sabine, Antretter, Martina, Krabichler, Maria, Oehlinger, Christine, Seeber, Barbara, Straif, Barbara, Strauss, Susann, and Kapfer, Cornelia

Published
1999

8. a great / ein großer

Author

Grabher, Gudrun M., Amann, Sabine, Antretter, Martina, Krabichler, Maria, Oehlinger, Christine, Seeber, Barbara, Straif, Barbara, Strauss, Susann, and Kapfer, Cornelia

Published
1999

10. Identification of ALK in Thinness

Author

Orthofer, Michael, primary, Valsesia, Armand, additional, Mägi, Reedik, additional, Wang, Qiao-Ping, additional, Kaczanowska, Joanna, additional, Kozieradzki, Ivona, additional, Leopoldi, Alexandra, additional, Cikes, Domagoj, additional, Zopf, Lydia M., additional, Tretiakov, Evgenii O., additional, Demetz, Egon, additional, Hilbe, Richard, additional, Boehm, Anna, additional, Ticevic, Melita, additional, Nõukas, Margit, additional, Jais, Alexander, additional, Spirk, Katrin, additional, Clark, Teleri, additional, Amann, Sabine, additional, Lepamets, Maarja, additional, Neumayr, Christoph, additional, Arnold, Cosmas, additional, Dou, Zhengchao, additional, Kuhn, Volker, additional, Novatchkova, Maria, additional, Cronin, Shane J.F., additional, Tietge, Uwe J.F., additional, Müller, Simone, additional, Pospisilik, J. Andrew, additional, Nagy, Vanja, additional, Hui, Chi-Chung, additional, Lazovic, Jelena, additional, Esterbauer, Harald, additional, Hagelkruys, Astrid, additional, Tancevski, Ivan, additional, Kiefer, Florian W., additional, Harkany, Tibor, additional, Haubensak, Wulf, additional, Neely, G. Gregory, additional, Metspalu, Andres, additional, Hager, Jorg, additional, Gheldof, Nele, additional, and Penninger, Josef M., additional

Published
2020
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11. Erratum. Adipocyte Glucocorticoid Receptor Deficiency Attenuates Aging- and HFD-Induced Obesity and Impairs the Feeding-Fasting Transition. Diabetes 2017;66:272–286

Author

Mueller, Kristina M., primary, Hartmann, Kerstin, additional, Kaltenecker, Doris, additional, Vettorazzi, Sabine, additional, Bauer, Mandy, additional, Mauser, Lea, additional, Amann, Sabine, additional, Jall, Sigrid, additional, Fischer, Katrin, additional, Esterbauer, Harald, additional, Müller, Timo D., additional, Tschöp, Matthias H., additional, Magnes, Christoph, additional, Haybaeck, Johannes, additional, Scherer, Thomas, additional, Bordag, Natalie, additional, Tuckermann, Jan P., additional, and Moriggl, Richard, additional

Published
2017
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12. Adipocyte Glucocorticoid Receptor Deficiency Attenuates Aging- and HFD-Induced Obesity and Impairs the Feeding-Fasting Transition

Author

Mueller, Kristina M., primary, Hartmann, Kerstin, additional, Kaltenecker, Doris, additional, Vettorazzi, Sabine, additional, Bauer, Mandy, additional, Mauser, Lea, additional, Amann, Sabine, additional, Jall, Sigrid, additional, Fischer, Katrin, additional, Esterbauer, Harald, additional, Müller, Timo D., additional, Tschöp, Matthias H., additional, Magnes, Christoph, additional, Haybaeck, Johannes, additional, Scherer, Thomas, additional, Bordag, Natalie, additional, Tuckermann, Jan P., additional, and Moriggl, Richard, additional

Published
2016
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13. Untersuchung zur Klassifizierung, Identifizierung und Differenzierung von Yersinia Arten

Author

Amann, Sabine

Abstract

Zur Identifizierung und Differenzierung von Y. pestis wurden mehrere PCR-Methoden entwickelt sowie eine Multiplex-PCR zur gleichzeitigen Identifizierung von Stämmen der Spezies Y. pestis und Y. pseudotuberculosis. Weiters wurde das Genus Yersinia in Hinblick auf atypische Y. pseudotuberculosis Stämme, die möglicherweise eine neue, mit Y. pestis und Y. pseudotuberculosis nah verwandte Spezies darstellen, und die Spezies Y. ruckeri, deren Zugehörigkeit zum Genus Yersinia kontrovers betrachtet wird und die phylogenetisch klar von anderen Yersinia Spezies abgetrennt werden kann, chemotaxonomisch untersucht. Eine Differenzierung dieser als Y. pseudotuberculosis "pseudopest" bezeichneten atypischen Stämme von Y. pestis und Y. pseudotuberculosis war durch das Lipidprofil möglich, während die Ergebnisse der Chinonanalyse und der DNA-DNA Hybridisierung eine Zuordnung zur Spezies Y. pseudotuberculosis ergaben. Basierend auf der Analyse des Chinonsystems sowie der polaren Lipide war eine Differenzierung zu anderen Yersinia Spezies nicht eindeutig möglich. Dagegen konnten beim Polyaminmuster von Y. ruckeri deutliche Unterschiede beobachtet werden, was die bereits gezeigte phylogenetisch separate Position dieser Spezies unterstützt und auf eine mögliche Reklassifizierung in ein neues Genus innerhalb der Enterobacteriaceae hindeutet.

Published
2007
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14. Adipocyte Glucocorticoid Receptor Deficiency Attenuates Aging- and HFD-Induced Obesity and Impairs the Feeding-Fasting Transition.

Author

Mueller, Kristina M., Hartmann, Kerstin, Kaltenecker, Doris, Vettorazzi, Sabine, Bauer, Mandy, Mauser, Lea, Amann, Sabine, Jall, Sigrid, Fischer, Katrin, Esterbauer, Harald, Müller, Timo D., Tschöp, Matthias H., Magnes, Christoph, Haybaeck, Johannes, Scherer, Thomas, Bordag, Natalie, Tuckermann, Jan P., and Moriggl, Richard

Subjects
GLUCOCORTICOIDS, ENERGY metabolism, PATHOLOGICAL physiology, METABOLOMICS, OBESITY, FAT cells, ADIPOSE tissues, AGING, ANIMAL experimentation, BETA adrenoceptors, BIOCHEMISTRY, HUMAN body composition, CELL receptors, CELLULAR signal transduction, DIET, FASTING, FATTY liver, FOOD habits, GENETIC disorders, HYPERTROPHY, INSULIN, LIPID metabolism disorders, LIQUID chromatography, LIVER, MASS spectrometry, MICE, WESTERN immunoblotting
Abstract

Glucocorticoids (GCs) are important regulators of systemic energy metabolism, and aberrant GC action is linked to metabolic dysfunctions. Yet, the extent to which normal and pathophysiological energy metabolism depend on the GC receptor (GR) in adipocytes remains unclear. Here, we demonstrate that adipocyte GR deficiency in mice significantly impacts systemic metabolism in different energetic states. Plasma metabolomics and biochemical analyses revealed a marked global effect of GR deficiency on systemic metabolite abundance and, thus, substrate partitioning in fed and fasted states. This correlated with a decreased lipolytic capacity of GR-deficient adipocytes under postabsorptive and fasting conditions, resulting from impaired signal transduction from β-adrenergic receptors to adenylate cyclase. Upon prolonged fasting, the impaired lipolytic response resulted in abnormal substrate utilization and lean mass wasting. Conversely, GR deficiency attenuated aging-/diet-associated obesity, adipocyte hypertrophy, and liver steatosis. Systemic glucose tolerance was improved in obese GR-deficient mice, which was associated with increased insulin signaling in muscle and adipose tissue. We conclude that the GR in adipocytes exerts central but diverging roles in the regulation of metabolic homeostasis depending on the energetic state. The adipocyte GR is indispensable for the feeding-fasting transition but also promotes adiposity and associated metabolic disorders in fat-fed and aged mice. [ABSTRACT FROM AUTHOR]

Published
2017
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15. Heme Oxygenase-1 Drives Metaflammation and Insulin Resistance in Mouse and Man

Author

Jais, Alexander, primary, Einwallner, Elisa, additional, Sharif, Omar, additional, Gossens, Klaus, additional, Lu, Tess Tsai-Hsiu, additional, Soyal, Selma M., additional, Medgyesi, David, additional, Neureiter, Daniel, additional, Paier-Pourani, Jamile, additional, Dalgaard, Kevin, additional, Duvigneau, J. Catharina, additional, Lindroos-Christensen, Josefine, additional, Zapf, Thea-Christin, additional, Amann, Sabine, additional, Saluzzo, Simona, additional, Jantscher, Florian, additional, Stiedl, Patricia, additional, Todoric, Jelena, additional, Martins, Rui, additional, Oberkofler, Hannes, additional, Müller, Simone, additional, Hauser-Kronberger, Cornelia, additional, Kenner, Lukas, additional, Casanova, Emilio, additional, Sutterlüty-Fall, Hedwig, additional, Bilban, Martin, additional, Miller, Karl, additional, Kozlov, Andrey V., additional, Krempler, Franz, additional, Knapp, Sylvia, additional, Lumeng, Carey N., additional, Patsch, Wolfgang, additional, Wagner, Oswald, additional, Pospisilik, J. Andrew, additional, and Esterbauer, Harald, additional

Published
2014
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16. Hedgehog partial agonism drives Warburg-like metabolism in muscle and brown fat

Author

UCL - SSS/LDRI - Louvain Drug Research Institute, Teperino, Raffaele, Amann, Sabine, Bayer, Martina, McGee, Sean L, Loipetzberger, Andrea, Connor, Timothy, Jaeger, Carsten, Kammerer, Bernd, Winter, Lilli, Wiche, Gerhard, Dalgaard, Kevin, Selvaraj, Madhan, Gaster, Michael, Lee-Young, Robert S, Febbraio, Mark A, Knauf, Claude, Cani, Patrice D., Aberger, Fritz, Penninger, Josef M, Pospisilik, J Andrew, Esterbauer, Harald, UCL - SSS/LDRI - Louvain Drug Research Institute, Teperino, Raffaele, Amann, Sabine, Bayer, Martina, McGee, Sean L, Loipetzberger, Andrea, Connor, Timothy, Jaeger, Carsten, Kammerer, Bernd, Winter, Lilli, Wiche, Gerhard, Dalgaard, Kevin, Selvaraj, Madhan, Gaster, Michael, Lee-Young, Robert S, Febbraio, Mark A, Knauf, Claude, Cani, Patrice D., Aberger, Fritz, Penninger, Josef M, Pospisilik, J Andrew, and Esterbauer, Harald

Abstract

Diabetes, obesity, and cancer affect upward of 15% of the world's population. Interestingly, all three diseases juxtapose dysregulated intracellular signaling with altered metabolic state. Exactly which genetic factors define stable metabolic set points in vivo remains poorly understood. Here, we show that hedgehog signaling rewires cellular metabolism. We identify a cilium-dependent Smo-Ca(2+)-Ampk axis that triggers rapid Warburg-like metabolic reprogramming within minutes of activation and is required for proper metabolic selectivity and flexibility. We show that Smo modulators can uncouple the Smo-Ampk axis from canonical signaling and identify cyclopamine as one of a new class of "selective partial agonists," capable of concomitant inhibition of canonical and activation of noncanonical hedgehog signaling. Intriguingly, activation of the Smo-Ampk axis in vivo drives robust insulin-independent glucose uptake in muscle and brown adipose tissue. These data identify multiple noncanonical endpoints that are pivotal for rational design of hedgehog modulators and provide a new therapeutic avenue for obesity and diabetes.

Published
2012

17. Hedgehog partial agonism drives warburg-lie metabolism in muscle and brown fat

Author

Teperino, Raffaele, Amann, Sabine, Bayer, Martina, McGee, Sean, Loipetzberger, Andrea, Connor, Timothy, Jaeger, Carsten, Kammerer, Bernd, Winter, Lilli, Wiche, Gerhard, Dalgaard, Kevin, Selvaraj, Madhan, Gaster, Michael, Lee-Young, Robert, Febbraio, Mark, Knauf, Claude, Cani, Patrice, Aberger, Fritz, Penninger, Josef M., Pospisilik, J. Andrew, Esterbauer, Harald, Teperino, Raffaele, Amann, Sabine, Bayer, Martina, McGee, Sean, Loipetzberger, Andrea, Connor, Timothy, Jaeger, Carsten, Kammerer, Bernd, Winter, Lilli, Wiche, Gerhard, Dalgaard, Kevin, Selvaraj, Madhan, Gaster, Michael, Lee-Young, Robert, Febbraio, Mark, Knauf, Claude, Cani, Patrice, Aberger, Fritz, Penninger, Josef M., Pospisilik, J. Andrew, and Esterbauer, Harald

Abstract

Diabetes, obesity, and cancer affect upward of 15% of the world’s population. Interestingly, all three diseases juxtapose dysregulated intracellular signaling with altered metabolic state. Exactly which genetic factors define stable metabolic set points in vivo remains poorly understood. Here, we show that hedgehog signaling rewires cellular metabolism. We identify a cilium-dependent Smo-Ca2+-Ampk axis that triggers rapid Warburg-like metabolic reprogramming within minutes of activation and is required for proper metabolic selectivity and flexibility. We show that Smo modulators can uncouple the Smo-Ampk axis from canonical signaling and identify cyclopamine as one of a new class of “selective partial agonists,” capable of concomitant inhibition of canonical and activation of noncanonical hedgehog signaling. Intriguingly, activation of the Smo-Ampk axis in vivo drives robust insulin-independent glucose uptake in muscle and brown adipose tissue. These data identify multiple noncanonical endpoints that are pivotal for rational design of hedgehog modulators and provide a new therapeutic avenue for obesity and diabetes.

Published
2012

18. Hedgehog Partial Agonism Drives Warburg-like Metabolism in Muscle and Brown Fat

Author

Teperino, Raffaele, primary, Amann, Sabine, additional, Bayer, Martina, additional, McGee, Sean L., additional, Loipetzberger, Andrea, additional, Connor, Timothy, additional, Jaeger, Carsten, additional, Kammerer, Bernd, additional, Winter, Lilli, additional, Wiche, Gerhard, additional, Dalgaard, Kevin, additional, Selvaraj, Madhan, additional, Gaster, Michael, additional, Lee-Young, Robert S., additional, Febbraio, Mark A., additional, Knauf, Claude, additional, Cani, Patrice D., additional, Aberger, Fritz, additional, Penninger, Josef M., additional, Pospisilik, J. Andrew, additional, and Esterbauer, Harald, additional

Published
2012
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19. A Global In Vivo Drosophila RNAi Screen Identifies NOT3 as a Conserved Regulator of Heart Function

Author

Neely, G. Gregory, primary, Kuba, Keiji, additional, Cammarato, Anthony, additional, Isobe, Kazuya, additional, Amann, Sabine, additional, Zhang, Liyong, additional, Murata, Mitsushige, additional, Elmén, Lisa, additional, Gupta, Vaijayanti, additional, Arora, Suchir, additional, Sarangi, Rinku, additional, Dan, Debasis, additional, Fujisawa, Susumu, additional, Usami, Takako, additional, Xia, Cui-ping, additional, Keene, Alex C., additional, Alayari, Nakissa N., additional, Yamakawa, Hiroyuki, additional, Elling, Ulrich, additional, Berger, Christian, additional, Novatchkova, Maria, additional, Koglgruber, Rubina, additional, Fukuda, Keiichi, additional, Nishina, Hiroshi, additional, Isobe, Mitsuaki, additional, Pospisilik, J. Andrew, additional, Imai, Yumiko, additional, Pfeufer, Arne, additional, Hicks, Andrew A., additional, Pramstaller, Peter P., additional, Subramaniam, Sai, additional, Kimura, Akinori, additional, Ocorr, Karen, additional, Bodmer, Rolf, additional, and Penninger, Josef M., additional

Published
2010
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20. Erratum. Adipocyte Glucocorticoid Receptor Deficiency Attenuates Aging- and HFD-Induced Obesity and Impairs the Feeding-Fasting Transition. Diabetes 2017;66:272-286.

Author

Mueller, Kristina M, Hartmann, Kerstin, Kaltenecker, Doris, Vettorazzi, Sabine, Bauer, Mandy, Mauser, Lea, Amann, Sabine, Jall, Sigrid, Fischer, Katrin, Esterbauer, Harald, Müller, Timo D, Tschöp, Matthias H, Magnes, Christoph, Haybaeck, Johannes, Scherer, Thomas, Bordag, Natalie, Tuckermann, Jan P, and Moriggl, Richard

Subjects
OBESITY treatment, GLUCOCORTICOID receptors
Abstract

A correction to the article "Adipocyte Glucocorticoid Receptor Deficiency Attenuates Aging- and HFD-Induced Obesity and Impairs the Feeding-Fasting Transition" by M. Kristina et al. that was published in a 2017 issue of the journal "Diabetes" is presented.

Published
2018
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21. Yersinia similis sp. nov.

Author

Sprague LD, Scholz HC, Amann S, Busse HJ, and Neubauer H

Subjects
Base Sequence, DNA, Bacterial genetics, Genes, Bacterial, Genetic Markers, Lipid Metabolism, Molecular Sequence Data, Phenotype, Phylogeny, RNA, Bacterial genetics, RNA, Ribosomal, 16S genetics, Species Specificity, Terminology as Topic, Yersinia metabolism, Yersinia classification, Yersinia genetics
Abstract

Strains originally identified phenotypically as members of the species Yersinia pseudotuberculosis were subjected to a more detailed classification employing 16S rRNA gene sequence analysis, DNA-DNA hybridization, determination of the DNA base composition and phenotypic characterization. The quinone system, consisting of the predominant compound ubiquinone Q-8 and minor amounts of menaquinone MK-8, the major components of the polar lipid profile, as well as the polyamine pattern, with putrescine as the major compound, supported the assignment of the strains to the genus Yersinia. Based on DNA-DNA relatedness, a specific 16S rRNA gene sequence type, absence of melibiose fermentation and a polar lipid profile lacking phosphatidylmonomethylethanolamine and two aminolipids, the strains were identified as members of a novel species for which the name Yersinia similis sp. nov. is proposed. The type strain of Yersinia similis sp. nov. is strain Y228T (=CCUG 52882T=LMG 23763T).

Published
2008
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