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Your search keyword '"Vynnychenko, Lb"' showing total 2 results
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2 results on '"Vynnychenko, Lb"'

1. [Application of "reytoil" in prevention of atherosclerosis in diabetes patients].

Author

Popov SV, Melekhovets OK, Demikhova NV, Vynnychenko LB, Khil'ko IeS, Tychyna DO, and Murga IV

Subjects
Atherosclerosis blood, Atherosclerosis etiology, Carotid Arteries drug effects, Carotid Arteries pathology, Cholesterol, HDL blood, Cholesterol, LDL blood, Cholesterol, VLDL blood, Diabetes Mellitus, Type 2 blood, Diabetes Mellitus, Type 2 complications, Dyslipidemias blood, Dyslipidemias complications, Humans, Risk Factors, Triglycerides blood, Tunica Intima drug effects, Tunica Intima pathology, Tunica Media drug effects, Tunica Media pathology, Atherosclerosis prevention & control, Carotid Intima-Media Thickness, Diabetes Mellitus, Type 2 diet therapy, Dietary Fats, Unsaturated pharmacology, Dyslipidemias diet therapy, Lipid Metabolism drug effects
Abstract

The paper was conducted sonographic study of extracranial atherosclerotic vascular changes in patients with diabetes mellitus, and lipid profile study of the dynamics under the influence of treatment with the inclusion of the drug "Reytoil". Structural changes of the vascular wall correlates with changes of lipid profile in patients with diabetes. Structural assessment of intima-media complex (IMC) can be used as a marker for early diagnosis of vascular remodeling in patients with diabetes, as well as the criterion of efficiency of a treatment. Detection of lipid exchange and evaluation of IMC in the early stages of diabetes mellitus will make it possible pathogenetic approach to providing comprehensive treatment of diabetes with dyslipidemia, including the drug "Reytoil".

Published
2012

2. [Drug with a high metabolic activity, cocarnit, in the treatment of diabetic cardiac autonomic neuropathy].

Author

Popov SV, Melekhovets' OK, Demikhova NV, and Vynnychenko LB

Subjects
Adenosine Triphosphate pharmacology, Adenosine Triphosphate therapeutic use, Adolescent, Adult, Autonomic Nervous System drug effects, Autonomic Nervous System metabolism, Cardiomyopathies complications, Cardiomyopathies metabolism, Cardiotonic Agents pharmacology, Diabetes Mellitus, Type 2 complications, Diabetes Mellitus, Type 2 metabolism, Diabetic Neuropathies complications, Diabetic Neuropathies metabolism, Drug Combinations, Fatty Acids metabolism, Female, Glucose metabolism, Heart drug effects, Heart Rate drug effects, Humans, Male, Middle Aged, Niacinamide pharmacology, Niacinamide therapeutic use, Oxidation-Reduction, Thiamine Pyrophosphate pharmacology, Thiamine Pyrophosphate therapeutic use, Ventricular Dysfunction, Left complications, Ventricular Dysfunction, Left metabolism, Ventricular Function, Left drug effects, Ventricular Function, Left physiology, Vitamin B 12 pharmacology, Vitamin B 12 therapeutic use, Cardiomyopathies drug therapy, Cardiotonic Agents therapeutic use, Diabetes Mellitus, Type 2 drug therapy, Diabetic Neuropathies drug therapy, Ventricular Dysfunction, Left drug therapy
Abstract

Left ventricular diastolic dysfunction in patients with diabetes is formed in the absence of atherosclerotic changes as a consequence of diabetic cardiac autonomic neuropathy in the early stages of diabetes. Progression of autonomic cardiac neuropathy in cardio-vascular type is associated with the violation of energy supply of cells, protein synthesis, electrolyte exchange, the exchange of trace elements, oxidation reduction processes, oxygen-transport function of blood, so that metabolic therapy is carried out to optimize the processes of formation and energy costs. The drug cocarnit activates processes of aerobic oxidation of glucose, as well as providing regulatory influence on the oxidation of fatty acids. Applying of cocarnit in complex therapy in patients with diabetic cardiac autonomic neuropathy found improvement of left ventricular diastolic function, and positive dynamics in the efferent activity balance of the sympathetic and parasympathetic control of heart rate variability, which provides the regression of clinical symptoms.

Published
2012

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