21 results on '"Vassar, Robert"'
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2. Behavioral, biochemical, and neuropathological outcomes in amyloid‐bearing mice treated long‐term with a range of doses of BACE inhibitor
3. O5‐06‐02: AXONAL ORGANIZATION DEFECTS IN THE HIPPOCAMPUS OF ADULT CONDITIONAL BACE1 KNOCKOUT MICE: IMPLICATIONS FOR BACE1 INHIBITOR CLINICAL TRIALS IN HUMANS
4. FTS3-01-01: UMIBECESTAT (CNP520) IS NOT ASSOCIATED WITH CHANGES IN HIPPOCAMPAL MORPHOLOGY IN RATS OR CHANGES IN CSF AD BIOMARKERS IN HUMANS TREATED FOR 3 MONTHS
5. P2-057: 3K3A-ACTIVATED PROTEIN C BLOCKS AMYLOIDOGENIC BACE1 PATHWAY AND IMPROVES FUNCTIONAL OUTCOME IN MICE
6. P1‐027: KINETIC BEHAVIOR OF NEWLY GENERATED BACE1‐CLEAVED APP IN THE HUMAN CENTRAL NERVOUS SYSTEM IN ALZHEIMER'S DISEASE
7. P1‐163: MOLECULAR MECHANISMS OF THE ALZHEIMER'S RISK GENE UNC5C IN NEURONAL DEATH
8. P2‐167: EXPRESSION OF THE DUAL LEUCINE ZIPPER KINASE IN DYSTROPHIC NEURITES AND AXONAL BEADING DURING PATHOLOGY PROGRESSION IN ALZHEIMER'S DISEASE
9. EC‐01‐03: Bace1, The Beta‐Secretase Enzyme: a Leading Therapeutic Target for Alzheimer's Disease
10. P1‐040: Quantitative comparison of dense‐core amyloid plaque burdens in Alzheimer's disease patients and amyloid precursor protein transgenic mice
11. Function, therapeutic potential and cell biology of BACE proteases: current status and future prospects
12. PL-04-01: Targeting beta-secretase
13. O1‐02‐06: The Alzheimer's beta‐secretase enzyme BACE1 is required for accurate axon guidance of olfactory sensory neurons and normal glomerulus formation in the olfactory bulb
14. Identification and biology of β-secretase
15. O1-03-01: Phosphorylation of the translation initiation factor eIF2alpha increases BACE1 levels and promotes amyloidogenesis
16. S3-03-01: BACE1 levels are elevated in a rapid amyloid pathology transgenic mouse model of Alzheimer's disease
17. O2-03-01: Exploring β-secretase activity in lipid raft microdomains
18. P3-337: S-palmitoylation targets BACE-1 to lipid rafts
19. P4-047: Energy inhibition elevates beta-secretase levels and activity and is potentially amyloidogenic in APP transgenic mice: Possible early events in Alzheimer's disease pathogenesis
20. O4–06–04: Intraneuronal Abeta42 aggregates seed amyloid plaques and cause neuronal loss in transgenic mice with five familial Alzheimer's disease mutations
21. Temporal memory deficits in Alzheimer's mouse models: rescue by genetic deletion of BACE1
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