18 results on '"Pietrzik, Claus U"'
Search Results
2. Tethering soluble meprin α in an enzyme complex to the cell surface affects IBD‐associated genes
3. Ectodomain shedding of CD99 within highly conserved regions is mediated by the metalloprotease meprin β and promotes transendothelial cell migration
4. O4-11-05: Endothelial-LRP1 Clears Major Amounts of Abeta 1-42 Across the Blood-Brain Barrier
5. Low-density lipoprotein receptor-related protein 1 is a novel modulator of radial glia stem cell proliferation, survival, and differentiation
6. Presenilin-1 but not amyloid precursor protein mutations present in mouse models of Alzheimer’s disease attenuate the response of cultured cells to γ-secretase modulators regardless of their potency and structure
7. α-secretase mediated conversion of the amyloid precursor protein derived membrane stub C99 to C83 limits Aβ generation
8. P4-244: Progranulin (GRN), a protein mutated in frontotemporal dementia with ubiquitin-positive inclusions (FTLD-U), is abundantly expressed in human gliomas
9. P3-271: Presenilin-1 (PS1) and amyloid precursor protein (APP) mutations present in mouse models of Alzheimer's disease in their response to γ-secretase inhibitors and modulators
10. P4-295: Small vessel disease-induced microvascular protein leakage provides a possible pathogenetic link to Alzheimer's disease
11. P4-192: Mechanism of γ-secretase cleavage: Evidence for independent generation of Aβ42 and Aβ38 peptide species
12. P3-344: γ-secretase-dependent APP processing occurs either at the plasma membrane or in the endocytic compartments independent of the APP wild-type or APP Swedish variant
13. O1–02–06: Genetic dissection of the PS1–ΔExon9 mutation and its attenuated response to Aβ42–lowering NSAIDs
14. P3-244: The impact of LRP on APP metabolism along the secretory pathway
15. P3–247: Functional consequences of LRP expression on familiar Alzheimer disease APP mutations
16. P4–070: Amyloid precursor–like proteins 1 and 2 influence proteolytic processing of the amyloid precursor protein by alpha– and beta–secretase
17. Calculation of red blood cell folate steady state conditions after supplementation with different folate forms and dosages
18. Inhibitors of Rho‐kinase modulate amyloid‐β (Aβ) secretion but lack selectivity for Aβ42
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