12 results on '"Peng, Tianqing"'
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2. Calpain‐2 protects against heat stress‐induced cardiomyocyte apoptosis and heart dysfunction by blocking p38 mitogen‐activated protein kinase activation
3. HMGB1 blockade attenuates experimental autoimmune myocarditis and suppresses Th17-cell expansion
4. Lipopolysaccharide plus hypoxia and reoxygenation synergistically reduce electrical coupling between microvascular endothelial cells by dephosphorylating Connexin40
5. Glycogen synthase kinase-3beta suppresses tumor necrosis factor-alpha expression in cardiomyocytes during lipopolysaccharide stimulation
6. Abrupt Reoxygenation of Microvascular Endothelial Cells After Hypoxia Activates ERK1/2 and JNK1, Leading to NADPH Oxidase-Dependent Oxidant Production
7. Inhibition of phospholipase C decreased cardiac COX‐2 expression and attenuated myocardial dysfunction during endotoxemia
8. Activation of GSK‐3β inhibited TNF‐α expression in cardiomyocytes during LPS stimulation
9. GSK‐3b inactivation in preventing the myocardium from I/R‐induced injury: Role of eNOS‐derived NO
10. Pivotal role of phospholipase C in cardiac TNF‐α expression during endotoxemia
11. Activation of NADH oxidase protects cardiomyocytes from apoptosis during anoxia and reoxygenation
12. NADH oxidase signaling induces cyclooxygenase‐2 expression during lipopolysaccharide stimulation in cardiomyocytes
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