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2. Multiple biomarkers improve diagnostic accuracy across Lewy body and Alzheimer's disease spectra

3. Lewy body dementia: Overcoming barriers and identifying solutions

5. Mediterranean diet protects against neuroinflammation and degeneration in female nonhuman primates

7. Cerebral amyloid angiopathy and neuritic plaques impact tangle burden and cognitive decline in a heterogeneous clinical population

12. Novel quantitative method for characterization of hippocampal sclerosis and its association with other neuropathologies and cognitive deficits

13. Characteristics of oldest‐old individuals with ‘Pure’ Limbic‐predominant age‐related TDP‐43 encephalopathy: a 90+ Study case series

15. Illusory Responses across the Lewy Body Disease Spectrum

16. Prediction of neuropathologic lesions from clinical data

17. Hemispheric asymmetry and atypical lobar progression of Alzheimer‐type tauopathy

18. Illusory responses across the Alzheimer’s disease spectrum and Lewy body disease spectrum

25. A consensus proteomic analysis of Alzheimer’s disease brain and cerebrospinal fluid reveals early changes in energy metabolism associated with microglia and astrocyte activation

26. Multimodal genome‐wide meta‐analysis of brain amyloidosis reveals heterogeneity across CSF, PET, and pathological amyloid measures

27. Genome‐wide association analysis of neurofibrillary tangle burden identifies novel risk loci in the adult changes of thought (ACT) and the religious orders study and memory and aging project (ROSMAP) autopsy cohorts

31. DT-01-05: TEMPORAL LOBE QUANTITATION OF FLORTAUCIPIR PET IMAGES MAY IMPROVE DETECTION OF INTERMEDIATE NEUROFIBRILLARY TANGLE PATHOLOGY IN AUTOPSY-VALIDATED CASES

32. S4-01-01: IDENTIFYING ELDERS AT RISK FOR COGNITIVE DECLINE USING THE BRAIN HEALTH REGISTRY (BHR)

34. A nonhuman primate model of early Alzheimer's disease pathologic change: Implications for disease pathogenesis

36. P4‐333: AUTOPSY FINDINGS OF CEREBRAL β‐AMYLOIDOSIS, TAUOPATHY, AND NEURODEGENERATION (A, T AND N) IN A COMMUNITY‐BASED SAMPLE: IMPLICATIONS FOR DEMENTIA MITIGATION STRATEGIES

37. P1‐105: A NON‐HUMAN PRIMATE MODEL OF EARLY ALZHEIMER'S DISEASE PATHOLOGIC CHANGE

40. P3‐064: COGNITIVE DECLINE TRAJECTORIES IN AUTOPSIED DEMENTIA PARTICIPANTS OF THE NUN STUDY (NS) AND THE HONOLULU‐ASIA AGING STUDY (HAAS): RELATIONSHIPS TO AGE, PREMORBID COGNITIVE TEST SCORES, EDUCATION, APOE4, AND NEUROPATHOLOGIC BURDEN

41. P2‐067: WHAT WOULD BE THE LIKELY IMPACT OF AN INTERVENTION THAT SUCCESSFULLY PREVENTED NEOCORTICAL ACCUMULATION OF NEURITIC AMYLOID PLAQUES AND A HIGH BRAAK STAGE? THOUGHT EXPERIMENTS USING DATA FROM THE NUN STUDY (NS) AND THE HONOLULU‐ASIA AGING STUDY

42. The National Institute on Aging and the Alzheimer's Association Research Framework for Alzheimer's disease: Perspectives from the Research Roundtable

43. Vasodilator dysfunction and oligodendrocyte dysmaturation in aging white matter

44. Application of the condensed protocol for the NIA‐AA guidelines for the neuropathological assessment of Alzheimer's disease in an academic clinical practice

47. [P2-383]: COMPARISON OF REGIONAL FLORTAUCIPIR PET TO QUANTITATIVE TAU AND AMYLOID IMMUNOASSAY IN PATIENTS WITH ALZHEIMER's DISEASE PATHOLOGY: A PILOT CLINICO-PATHOLOGICAL STUDY

48. IDENTIFICATION OF AN ITGA7 VARIANT ASSOCIATED WITH ALZHEIMER’S DISEASE AND MULTIPLE OTHER NEURODEGENERATIVE DISEASES

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