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Your search keyword '"LIU Xiao-Hong"' showing total 31 results
Start Over Author "LIU Xiao-Hong" Publisher wiley
31 results on '"LIU Xiao-Hong"'

1. A complete oral regimen for induction therapy of patients with high‐risk APL: An oral etoposide instead of intravenous infusion for cytoreductive chemotherapy

Author

Tang, Fei‐Fei, primary, Duan, Wen‐Bing, additional, Liu, Xiao‐Hong, additional, Lu, Sheng‐Ye, additional, Zhao, Xiao‐Su, additional, Qin, Ya‐Zhen, additional, Jia, Jin‐Song, additional, Wang, Jing, additional, Gong, Li‐Zhong, additional, Jiang, Qian, additional, Zhao, Ting, additional, Shi, Hong‐Xia, additional, Chang, Ying‐Jun, additional, Huang, Xiao‐Jun, additional, and Jiang, Hao, additional

Published
2024
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6. PML‐RARA transcript levels at the end of induction therapy are associated with prognosis in non‐high‐risk acute promyelocytic leukaemia with all‐ trans retinoic acid plus arsenic in front‐line therapy: long‐term follow‐up of a single‐centre cohort study

Author

Tang, Fei‐Fei, primary, Lu, Sheng‐Ye, additional, Zhao, Xiao‐Su, additional, Qin, Ya‐Zhen, additional, Liu, Xiao‐Hong, additional, Jia, Jin‐Song, additional, Wang, Jing, additional, Gong, Li‐Zhong, additional, Jiang, Qian, additional, Zhao, Ting, additional, Shi, Hong‐Xia, additional, Chang, Ying-Jun, additional, Huang, Xiao-Jun, additional, and Jiang, Hao, additional

Published
2021
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7. Systemic lupus erythematosus complicated with gastric cancer in an old man: A case report and literature review

Author

Ge Nan, Qu Xuan, Liu Xiao Hong, Zhang Ning, and Li Xiao Yi

Subjects
030203 arthritis & rheumatology, medicine.medical_specialty, business.industry, MEDLINE, Cancer, Case Report, General Medicine, 030204 cardiovascular system & hematology, medicine.disease, Dermatology, 03 medical and health sciences, 0302 clinical medicine, medicine, business
Published
2018
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12. Synthesis and Crystal Structure of a Novel Supramolecular Compound [Mg(H2O)6](C16H11O4SO3)2·10H2O

Author

Gao Zi-Wd, Zhang Zun-ting, Liu Qianguang, Yang Bo-lun, Liu Xiao-Hong, and Yu Kaibei

Subjects
Crystal, Crystallography, chemistry, Magnesium, Stereochemistry, Hydrogen bond, Supramolecular chemistry, Stacking, Proton NMR, chemistry.chemical_element, General Chemistry, Crystal structure, Triclinic crystal system
Abstract

A hydrolyte of magnesium 7-methoxyl-4′-hydroxylisoflavone-3′-sulfonate ([Mg(H2O)6](C16H11O4SO3)2·10H2O) was synthesized and its crystal structure was determined by X-ray diffraction analyses. The crystal belongs to triclinic crystal system, space group P-1 with cell constants a = 0.7359(1) nm, b = 0.9935(1) nm, c = 1.6335(2) nm, α = 91.16(1)°, β = 92.07(1)°, γ = 106.53(1)°, V = 1.1436(2) nm5 and Z = 1. The results show that the title compound consists of [Mg(H2O)6]2+, C16H11O4SO3- and H2O. Mg(II) is located at the symmetry center of the title compound. [Mg(H2O)6]2+, C16H11SO3− and H2O form fourteen kinds of hydrogen bonds. The distance of 0.351 nm between parallel isoflavone rings shows a #-# stacking interaction. The hydrogen bonds and #-# stacking interaction lead to supramolecular formation with three-dimensional network structure. The title compound was also characterized by IR and 1H NMR spectra.

Published
2010
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14. CD38 promotes angiotensin II-induced cardiac hypertrophy

Author

Guan, Xiao-Hui, primary, Hong, Xuan, additional, Zhao, Ning, additional, Liu, Xiao-Hong, additional, Xiao, Yun-Fei, additional, Chen, Ting-Tao, additional, Deng, Li-Bin, additional, Wang, Xiao-Lei, additional, Wang, Jian-Bin, additional, Ji, Guang-Ju, additional, Fu, Mingui, additional, Deng, Ke-Yu, additional, and Xin, Hong-Bo, additional

Published
2017
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30. CD38 Deficiency Protects the Heart from Ischemia/Reperfusion Injury through Activating SIRT1/FOXOs-Mediated Antioxidative Stress Pathway.

Author

Guan XH, Liu XH, Hong X, Zhao N, Xiao YF, Wang LF, Tang L, Jiang K, Qian YS, Deng KY, Ji G, Fu M, and Xin HB

Subjects
ADP-ribosyl Cyclase genetics, ADP-ribosyl Cyclase 1 genetics, Animals, Apoptosis, Calcium metabolism, Cell Line, Disease Models, Animal, Genotype, Male, Membrane Glycoproteins genetics, Mice, Inbred C57BL, Mice, Knockout, Myocardial Infarction enzymology, Myocardial Infarction genetics, Myocardial Infarction pathology, Myocardial Reperfusion Injury enzymology, Myocardial Reperfusion Injury genetics, Myocardial Reperfusion Injury pathology, Myocardium pathology, NADPH Oxidase 4, NADPH Oxidases metabolism, Phenotype, RNA Interference, Rats, Reactive Oxygen Species metabolism, Signal Transduction, Transfection, ADP-ribosyl Cyclase deficiency, ADP-ribosyl Cyclase 1 deficiency, Forkhead Box Protein O3 metabolism, Membrane Glycoproteins deficiency, Myocardial Infarction prevention & control, Myocardial Reperfusion Injury prevention & control, Myocardium enzymology, Nerve Tissue Proteins metabolism, Oxidative Stress, Sirtuin 1 metabolism
Abstract

Ischemia/reperfusion (I/R) injury induces irreversible oxidative stress damage to the cardiac muscle. We previously observed that CD38 deficiency remarkably protects mouse embryonic fibroblasts (MEFs) from oxidative stress-induced injury. However, whether CD38 deficiency protects from I/R injury in the heart is not explored. Here, we showed that the hearts of CD38 deficient mice or wild type mice supplied with exogenous NAD were significantly protected from ischemia/reperfusion injury, seen as reduction of the myocardial infarct sizes when the mice were subjected to 30 min ischemia followed by 24 hours of reperfusion. Consistently, the protection of CD38 deficiency on hypoxia/reoxygenation (H/R) injury was confirmed with a CD38 knockdown H9c2 stable cell line. Furthermore, we observed that knockdown of CD38 remarkably inhibited ROS generation and intracellular Ca(2+) overloading induced by H/R in H9c2 cells. The FOXO1 and FOXO3 expressions were significantly elevated by H/R injury in CD38 knockdown cells compared with normal H9c2 cells. The cell immunofluorescence assay showed that FOXO1 nuclear translocation was significantly increased in CD38 knockdown H9c2 cells. In addition, we demonstrated that the increase of FOXO1 nuclear translocation was associated with the increased expressions of antioxidant catalase and SOD2 and the attenuated expression of the ROS generation enzyme NOX4. In conclusion, our results provide new evidence that CD38 deficiency protects the heart from I/R injury through activating SIRT1/FOXOs-mediated antioxidative stress pathway.

Published
2016
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31. Attitudes towards Advance Care Planning and Healthcare Autonomy among Community-Dwelling Older Adults in Beijing, China.

Author

Zhang N, Ning XH, Zhu ML, Liu XH, Li JB, and Liu Q

Subjects
Adult, Aged, Aged, 80 and over, China, Cross-Sectional Studies, Delivery of Health Care trends, Female, Health Planning Support trends, Humans, Male, Middle Aged, Socioeconomic Factors, Delivery of Health Care organization & administration, Health Planning Support organization & administration, Personal Autonomy
Abstract

Objectives: To investigate the preferences of ACP and healthcare autonomy in community-dwelling older Chinese adults., Methods: A community-based cross-sectional study was conducted with older adults living in the residential estate of Chaoyang District, Beijing., Results: 900 residents were enrolled. 80.9% of them wanted to hear the truth regarding their own condition from the physician; 52.4% preferred to make their own healthcare decisions. Only 8.9% of them preferred to endure life-prolonging interventions when faced with irreversible conditions. 78.3% of the respondents had not heard of an ACP; only 39.4% preferred to document in an ACP. Respondents with higher education had significantly higher proportion of having heard of an ACP, as well as preferring to document in an ACP, compared to those with lower education. Those aged <70 years had higher proportion of having heard of an ACP, as well as refusing life-prolonging interventions when faced with irreversible conditions, compared to those aged ≥ 70 years., Conclusions: Although the majority of community-dwelling older Chinese adults appeared to have healthcare autonomy and refuse life-prolonging interventions in terms of end-of-life care, a low level of "Planning ahead" awareness and preference was apparent. Age and education level may be the influential factors.

Published
2015
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