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10 results on '"Jacobs, Muazzam"'

6. Relative contribution of IL‐1α, IL‐1β and TNF to the host response to Mycobacterium tuberculosis and attenuated M. bovis BCG

Author

Bourigault, Marie‐Laure, primary, Segueni, Noria, additional, Rose, Stéphanie, additional, Court, Nathalie, additional, Vacher, Rachel, additional, Vasseur, Virginie, additional, Erard, François, additional, Le Bert, Marc, additional, Garcia, Irene, additional, Iwakura, Yoichiro, additional, Jacobs, Muazzam, additional, Ryffel, Bernhard, additional, and Quesniaux, Valerie F. J., additional

Published
2013
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7. Type I interferons contribute to experimental cerebral malaria development in response to sporozoite or blood-stagePlasmodium bergheiANKA

Author

Palomo, Jennifer, primary, Fauconnier, Mathilde, additional, Coquard, Laurie, additional, Gilles, Maïlys, additional, Meme, Sandra, additional, Szeremeta, Frederic, additional, Fick, Lizette, additional, Franetich, Jean-François, additional, Jacobs, Muazzam, additional, Togbe, Dieudonnée, additional, Beloeil, Jean-Claude, additional, Mazier, Dominique, additional, Ryffel, Bernhard, additional, and Quesniaux, Valerie F.J., additional

Published
2013
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9. T cell-derived TNF down-regulates acute airway response to endotoxin

Author

Togbe, Dieudonnée, primary, Grivennikov, Sergei I., additional, Noulin, Nicolas, additional, Couillin, Isabelle, additional, Maillet, Isabelle, additional, Jacobs, Muazzam, additional, Maret, Marielle, additional, Fick, Lizette, additional, Nedospasov, Sergei A., additional, Quesniaux, Valérie F. J., additional, Schnyder, Bruno, additional, and Schnyder-Candrian, Silvia, additional

Published
2007
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10. Toll-like receptors and control of mycobacterial infection in mice.

Author

Ryffel B, Jacobs M, Parida S, Botha T, Togbe D, and Quesniaux V

Subjects
Animals, Ligands, Mice, Mycobacterium Infections microbiology, Mycobacterium bovis physiology, Receptors, Pattern Recognition immunology, Signal Transduction, Mycobacterium Infections prevention & control, Toll-Like Receptors immunology
Abstract

Microbial products including mycobacterial antigens are recognized by distinct Toll-like receptors (TLRs) resulting in activation of cells of the innate immune system. Ablation of most of the TLR signalling in mice deficient for the common adaptor protein MyD88 revealed that TLRs are crucial for the activation of an innate immune response as MyD88-deficient mice are highly sensitive to infection with Mycobacterium tuberculosis. Despite the profound defect of the innate immune response, MyD88 deficiency allows the emergence of an adaptive immunity. These data demonstrate that activation of multiple TLRs contributes to an efficient innate response to mycobacteria, while MyD88-dependent signalling is dispensable to generate adaptive immunity.

Published
2006

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