1. Catheter Ablation for an Incessant Form of Antiarrhythmic Drug-Resistant Ventricular Fibrillation After Acute Coronary Syndrome
- Author
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Yoshihisa Enjoji, Atsushi Funatsu, Daisuke Kanbayashi, Masahiro Mizobuchi, Shigeru Nakamura, Tomoko Kobayashi, Itaru Yokouchi, and Kensaku Shibata
- Subjects
Male ,medicine.medical_specialty ,Acute coronary syndrome ,Purkinje fibers ,medicine.medical_treatment ,Drug Resistance ,Catheter ablation ,Drug resistance ,Purkinje Fibers ,Electrocardiography ,Internal medicine ,medicine ,Humans ,cardiovascular diseases ,Aged ,Premature ventricular complexes ,business.industry ,General Medicine ,Ablation ,medicine.disease ,Ventricular Premature Complexes ,medicine.anatomical_structure ,Anesthesia ,Heart failure ,Ventricular Fibrillation ,Ventricular fibrillation ,Catheter Ablation ,cardiovascular system ,Cardiology ,Cardiology and Cardiovascular Medicine ,business ,Anti-Arrhythmia Agents - Abstract
A 77-year-old man was admitted with an acute coronary syndrome (ACS), severe heart failure (HF), and repeated ventricular fibrillation (VF) episodes. A single premature ventricular complex (PVC) induced ventricular tachycardia (VT), which degenerated to VF reproducibly. This PVC was eliminated by catheter ablation at the left ventricular posteroseptal region where double Purkinje potentials preceding the ventricular wave had been recorded. The electrical storm disappeared, and programmable stimulation failed to induce any tachyarrhythmias after the ablation. A Purkinje fiber network-related PVC served as a trigger and as a substrate for VT and VF in a case of ACS with HF.
- Published
- 2006
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