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42 results on '"Anderson, Judy"'

1. Age‐related nitration/dysfunction of myogenic stem cell activator HGF

Author

Elgaabari, Alaa, primary, Imatomi, Nana, additional, Kido, Hirochika, additional, Nakashima, Takashi, additional, Okuda, Shoko, additional, Manabe, Yoshitaka, additional, Sawano, Shoko, additional, Mizunoya, Wataru, additional, Kaneko, Ryuki, additional, Tanaka, Sakiho, additional, Maeno, Takahiro, additional, Matsuyoshi, Yuji, additional, Seki, Miyumi, additional, Kuwakado, So, additional, Zushi, Kahona, additional, Daneshvar, Nasibeh, additional, Nakamura, Mako, additional, Suzuki, Takahiro, additional, Sunagawa, Kenji, additional, Anderson, Judy E., additional, Allen, Ronald E., additional, and Tatsumi, Ryuichi, additional

Published
2023
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7. Fast‐to‐slow shift of muscle fiber‐type composition by dietary apple polyphenols in rats: Impact of the low‐dose supplementation

Author

Mizunoya, Wataru, primary, Okamoto, Shinpei, additional, Miyahara, Hideo, additional, Akahoshi, Mariko, additional, Suzuki, Takahiro, additional, Do, Mai‐Khoi Q., additional, Ohtsubo, Hideaki, additional, Komiya, Yusuke, additional, Qahar, Mulan, additional, Waga, Toshiaki, additional, Nakazato, Koichi, additional, Ikeuchi, Yoshihide, additional, Anderson, Judy E., additional, and Tatsumi, Ryuichi, additional

Published
2016
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9. Transmembrane proteoglycans syndecan-2, 4, receptor candidates for the impact of HGF and FGF2 on semaphorin 3A expression in early-differentiated myoblasts

Author

Do, Mai-Khoi Q., primary, Shimizu, Naomi, additional, Suzuki, Takahiro, additional, Ohtsubo, Hideaki, additional, Mizunoya, Wataru, additional, Nakamura, Mako, additional, Sawano, Shoko, additional, Furuse, Mitsuhiro, additional, Ikeuchi, Yoshihide, additional, Anderson, Judy E., additional, and Tatsumi, Ryuichi, additional

Published
2015
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12. Muscle‐specific AMPK β1β2‐null mice display a myopathy due to loss of capillary density in nonpostural muscles

Author

Thomas, Melissa M., primary, Wang, David C., additional, D'Souza, Donna M., additional, Krause, Matthew P., additional, Layne, Andrew S., additional, Criswell, David S., additional, O'Neill, Hayley M., additional, Connor, Michael K., additional, Anderson, Judy E., additional, Kemp, Bruce E., additional, Steinberg, Gregory R., additional, and Hawke, Thomas J., additional

Published
2014
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18. Utilization of micelles formed from poly(ethylene glycol)-block-poly(ϵ-caprolactone) block copolymers as nanocarriers to enable hydrophobic red two-photon absorbing emitters for cells imaging

Author

Tian, Yanqing, primary, Wu, Wen-Chung, additional, Chen, Ching-Yi, additional, Jang, Sei-Hum, additional, Zhang, Meng, additional, Strovas, Tim, additional, Anderson, Judy, additional, Cookson, Brad, additional, Li, Yongzhong, additional, Meldrum, Deirdre, additional, Chen, Wen-Chang, additional, and Jen, Alex K.-Y., additional

Published
2009
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40. Fast-to-slow shift of muscle fiber-type composition by dietary apple polyphenols in rats: Impact of the low-dose supplementation.

Author

Mizunoya W, Okamoto S, Miyahara H, Akahoshi M, Suzuki T, Do MQ, Ohtsubo H, Komiya Y, Qahar M, Waga T, Nakazato K, Ikeuchi Y, Anderson JE, and Tatsumi R

Subjects
Animals, Dose-Response Relationship, Drug, Humans, Male, Muscle Fatigue drug effects, Myoglobin metabolism, Myosin Heavy Chains metabolism, Protein Isoforms, Rats, Sprague-Dawley, Up-Regulation drug effects, Dietary Supplements, Malus, Muscle Fibers, Fast-Twitch metabolism, Polyphenols administration & dosage, Polyphenols pharmacology
Abstract

Our previous studies demonstrated that an 8-week intake of 5% (w/w) apple polyphenol (APP) in the diet improves muscle endurance of young-adult rats. In order to identify a lower limit of the dietary contribution of APP to the effect, the experiments were designed for lower-dose supplementation (8-week feeding of 0.5% APP in AIN-93G diet) to 12-week-old male Sprague-Dawley rats. Results clearly showed that the 0.5% APP diet significantly up-regulates slower myosin-heavy-chain (MyHC) isoform ratios (IIx and IIa relative to total MyHC) and myoglobin expression in lower hind-limb muscles examined (P < 0.05). There was a trend to increased fatigue resistance detected from measurements of relative isometric plantar-flexion force torque generated by a stimulus train delivered to the tibial nerve (F(98, 1372) = 1.246, P = 0.0574). Importantly, there was no significant difference in the animal body-phenotypes or locomotor activity shown as total moving distance in light and dark periods. Therefore, the present study encourages the notion that even low APP-intake may increase the proportions of fatigue-resistant myofibers, and has promise as a strategy for modifying performance in human sports and improving function in age-related muscle atrophy., (© 2016 Japanese Society of Animal Science.)

Published
2017
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41. Nitric oxide-dependence of satellite stem cell activation and quiescence on normal skeletal muscle fibers.

Author

Wozniak AC and Anderson JE

Subjects
Animals, Cycloheximide pharmacology, Hepatocyte Growth Factor metabolism, Mice, Mice, Inbred mdx, Mice, Transgenic, Nitric Oxide Synthase antagonists & inhibitors, PAX7 Transcription Factor metabolism, Proto-Oncogene Proteins c-met metabolism, Muscle Fibers, Skeletal metabolism, Muscle, Skeletal metabolism, Nitric Oxide metabolism, Satellite Cells, Skeletal Muscle metabolism, Stem Cells metabolism
Abstract

Satellite cells (quiescent precursors in normal adult skeletal muscle) are activated for growth and regeneration. Signaling by nitric oxide (NO) and hepatocyte growth factor (HGF) during activation has not been examined in a model that can distinguish quiescent from activated satellite cells. We tested the hypothesis that NO and HGF are required to regulate activation using the single-fiber culture model. In normal fibers, HGF and inhibition of NO synthase (NOS) each increased activation without stretching, and NOS inhibition reduced stretch-activation. Activation in unstretched mdx and NOS-I(-/-) fibers was three- to fourfold higher than normal, and was reduced by stretching. Distinctions were not due to different pax7-expressing populations on normal and mdx fibers. The population of c-met-expressing satellite cells on normal fibers was increased by stretch, demonstrating functional heterogeneity among normal satellite cells. Cycloheximide did not prevent the stretch-related increase in c-met expression, suggesting c-met may be an immediate-early gene in satellite cell activation. Results have important implications for designing new therapies that target the role of exercise in health, aging, and disease.

Published
2007
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42. Satellite cells from dystrophic (mdx) mice display accelerated differentiation in primary cultures and in isolated myofibers.

Author

Yablonka-Reuveni Z and Anderson JE

Subjects
Animals, Cells, Cultured, Mice, Mice, Inbred C57BL, Mice, Inbred mdx, Muscular Dystrophies pathology, Cell Differentiation physiology, Myofibrils physiology, Satellite Cells, Skeletal Muscle cytology
Abstract

In the dystrophic (mdx) mouse, skeletal muscle undergoes cycles of degeneration and regeneration, and myogenic progenitors (satellite cells) show ongoing proliferation and differentiation at a time when counterpart cells in normal healthy muscle enter quiescence. However, it remains unclear whether this enhanced satellite cell activity is triggered solely by the muscle environment or is also governed by factors inherent in satellite cells. To obtain a better picture of myogenesis in dystrophic muscle, a direct cell-by-cell analysis was performed to compare satellite cell dynamics from mdx and normal (C57Bl/10) mice in two cell culture models. In one model, the kinetics of satellite cell differentiation was quantified in primary cell cultures from diaphragm and limb muscles by immunodetection of MyoD, myogenin, and MEF2. In mdx cell cultures, myogenin protein was expressed earlier than normal and was followed more rapidly by dual myogenin/MEF2A expression and myotube formation. In the second model, the dynamics of satellite cell myogenesis were investigated in cultured myofibers isolated from flexor digitorum brevis (FDB) muscle, which retain satellite cells in the native position. Consistent with primary cultures, satellite cells in mdx myofibers displayed earlier myogenin expression, as well as an enhanced number of myogenin-expressing satellite cells per myofiber compared to normal. The addition of fibroblast growth factor 2 (FGF2) led to an increase in the number of satellite cells expressing myogenin in normal and mdx myofibers. However, the extent of the FGF effect was more robust in mdx myofibers. Notably, many myonuclei in mdx myofibers were centralized, evidence of segmental regeneration; all central nuclei and many peripheral nuclei in mdx myofibers were positive for MEF2A. Results indicated that myogenic cells in dystrophic muscle display accelerated differentiation. Furthermore, the study demonstrated that FDB myofibers are an excellent model of the in vivo state of muscle, as they accurately represented the dystrophic phenotype., (2005 Wiley-Liss, Inc.)

Published
2006
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